@inbook{2368,
abstract = {The recent experimental success in creating Bose-Einstein condensates of alkali atoms, honored by the Nobel prize awards in 2001 [1,5], led to renewed interest in the mathematical description of interacting Bose gases.},
author = {Robert Seiringer},
booktitle = {Large Coulomb Systems},
editor = {Dereziński, Jan and Siedentop, Heinz},
pages = {249 -- 274},
publisher = {Springer},
title = {{Dilute, trapped Bose gases and Bose-Einstein condensation}},
doi = {10.1007/3-540-32579-4_6},
volume = {695},
year = {2006},
}
@inbook{2369,
abstract = {One of the most remarkable recent developments in the study of ultracold Bose gases is the observation of a reversible transition from a Bose Einstein condensate to a state composed of localized atoms as the strength of a periodic, optical trapping potential is varied. In [1] a model of this phenomenon has been analyzed rigorously. The gas is a hard core lattice gas and the optical lattice is modeled by a periodic potential of strength λ. For small λ and temperature Bose- Einstein condensation (BEC) is proved to occur, while at large λ BEC disappears, even in the ground state, which is a Mott-insulator state with a characteristic gap. The inter-particle interaction is essential for this effect. This contribution gives a pedagogical survey of these results.},
author = {Aizenman, Michael and Lieb, Élliott H and Robert Seiringer and Solovej, Jan P and Yngvason, Jakob},
booktitle = {Mathematical Physics of Quantum Mechanics},
editor = {Asch, Joachim and Joye, Alain},
pages = {199 -- 215},
publisher = {Springer},
title = {{Bose-Einstein condensation as a quantum phase transition in an optical lattice}},
doi = {10.1007/b11573432},
volume = {690},
year = {2006},
}
@inbook{2416,
author = {Bang-Jensen, Jørgen and Reed, Bruce and Schacht, Bruce and Šámal, Robert and Toft, Bjarne and Uli Wagner},
booktitle = {Topics in Discrete Mathematics},
pages = {613 -- 627},
publisher = {Springer},
title = {{On six problems posed by Jarik Nešetřil}},
doi = {10.1007/3-540-33700-8_30},
volume = {26},
year = {2006},
}
@article{2429,
abstract = {We show, with an elementary proof, that the number of halving simplices in a set of n points in 4 in general position is O(n4-2/45). This improves the previous bound of O(n4-1/134). Our main new ingredient is a bound on the maximum number of halving simplices intersecting a fixed 2-plane. },
author = {Matoušek, Jiří and Sharir, Micha and Smorodinsky, Shakhar and Uli Wagner},
journal = {Discrete & Computational Geometry},
number = {2},
pages = {177 -- 191},
publisher = {Springer},
title = {{K-sets in four dimensions}},
doi = {10.1007/s00454-005-1200-4},
volume = {35},
year = {2006},
}
@article{2430,
abstract = {We consider an online version of the conflict-free coloring of a set of points on the line, where each newly inserted point must be assigned a color upon insertion, and at all times the coloring has to be conflict-free, in the sense that in every interval I there is a color that appears exactly once in I. We present deterministic and randomized algorithms for achieving this goal, and analyze their performance, that is, the maximum number of colors that they need to use, as a function of the number n of inserted points. We first show that a natural and simple (deterministic) approach may perform rather poorly, requiring Ω(√̃) colors in the worst case. We then derive two efficient variants of this simple algorithm. The first is deterministic and uses O(log 2 n) colors, and the second is randomized and uses O(log n) colors with high probability. We also show that the O(log 2 n) bound on the number of colors used by our deterministic algorithm is tight on the worst case. We also analyze the performance of the simplest proposed algorithm when the points are inserted in a random order and present an incomplete analysis that indicates that, with high probability, it uses only O(log n) colors. Finally, we show that in the extension of this problem to two dimensions, where the relevant ranges are disks, n colors may be required in the worst case.},
author = {Chent, Ke and Fiat, Amos and Kaplan, Haim and Levy, Meital B and Matoušek, Jiří and Mossel, Elchanan and Pach, János and Sharir, Micha and Smorodinsky, Shakhar and Uli Wagner and Welzl, Emo},
journal = {SIAM Journal on Computing},
number = {5},
pages = {1342 -- 1359},
publisher = {SIAM},
title = {{Online conflict-free coloring for intervals}},
doi = {10.1137/S0097539704446682},
volume = {36},
year = {2006},
}
@inproceedings{2431,
abstract = {We prove an upper bound, tight up to a factor of 2, for the number of vertices of level at most t in an arrangement of n halfspaces in R , for arbitrary n and d (in particular, the dimension d is not considered constant). This partially settles a conjecture of Eckhoff, Linhart, and Welzl. Up to the factor of 2, the result generalizes McMullen's Upper Bound Theorem for convex polytopes (the case ℓ = O) and extends a theorem of Linhart for the case d ≤ 4. Moreover, the bound sharpens asymptotic estimates obtained by Clarkson and Shor. The proof is based on the h-matrix of the arrangement (a generalization, introduced by Mulmuley, of the h-vector of a convex polytope). We show that bounding appropriate sums of entries of this matrix reduces to a lemma about quadrupels of sets with certain intersection properties, and we prove this lemma, up to a factor of 2, using tools from multilinear algebra. This extends an approach of Alon and Kalai, who used linear algebra methods for an alternative proof of the classical Upper Bound Theorem. The bounds for the entries of the h-matrix also imply bounds for the number of i-dimensional faces, i > 0, at level at most ℓ. Furthermore, we discuss a connection with crossing numbers of graphs that was one of the main motivations for investigating exact bounds that are valid for arbitrary dimensions.},
author = {Uli Wagner},
pages = {635 -- 645},
publisher = {IEEE},
title = {{On a geometric generalization of the Upper Bound Theorem}},
doi = {10.1109/FOCS.2006.53},
year = {2006},
}
@article{2657,
abstract = {The highest densities of the two metabotropic GABA subunits, GABA B1 and GABAB2, have been reported as occurring around the glutamatergic synapses between Purkinje cell spines and parallel fibre varicosities. In order to determine how this distribution is achieved during development, we investigated the expression pattern and the cellular and subcellular localization of the GABAB1 and GABAB2 subunits in the rat cerebellum during postnatal development. At the light microscopic level, immunoreactivity for the GABAB1 and GABAB2 subunits was very prominent in the developing molecular layer, especially in Purkinje cells. Using double immunofluorescence, we demonstrated that GABAB1 was transiently expressed in glial cells. At the electron microscopic level, immunoreactivity for GABAB receptors was always detected both pre- and postsynaptically. Presynaptically, GABAB1 and GABAB2 were localized in the extrasynaptic membrane of parallel fibres at all ages, and only rarely in GABAergic axons. Postsynaptically, GABAB receptors were localized to the extrasynaptic and perisynaptic plasma membrane of Purkinje cell dendrites and spines throughout development. Quantitative analysis and three-dimensional reconstructions further revealed a progressive developmental movement of the GABAB1 subunit on the surface of Purkinje cells from dendritic shafts to its final destination, the dendritic spines. Together, these results indicate that GABAB receptors undergo dynamic regulation during cerebellar development in association with the establishment and maturation of glutamatergic synapses to Purkinje cells.},
author = {Luján, Rafael and Ryuichi Shigemoto},
journal = {European Journal of Neuroscience},
number = {6},
pages = {1479 -- 1490},
publisher = {Wiley-Blackwell},
title = {{Localization of metabotropic GABA receptor subunits GABAB1 and GABAB2 relative to synaptic sites in the rat developing cerebellum}},
doi = {10.1111/j.1460-9568.2006.04669.x},
volume = {23},
year = {2006},
}
@article{2659,
abstract = {Transmembrane AMPA receptor regulatory proteins (TARPs), including stargazin/γ-2, are associated with AMPA receptors and participate in their surface delivery and anchoring at the postsynaptic membrane. TARPs may also act as a positive modulator of the AMPA receptor ion channel function; however, little is known about other TARP members except for stargazin/γ-2. We examined the synaptic localization of stargazin/γ-2 and γ-8 by immunoelectron microscopy and biochemical analysis. The analysis of sodium dodecyl sulfate-digested freeze-fracture replica labeling revealed that stargazin/γ-2 was concentrated in the postsynaptic area, whereas γ-8 was distributed both in synaptic and extra-synaptic plasma membranes of the hippocampal neuron. When a synaptic plasma membrane-enriched brain fraction was treated with Triton X-100 and separated by sucrose density gradient ultracentrifugation, a large proportion of NMDA receptor and stargazin/γ-2 was accumulated in raft-enriched fractions, whereas AMPA receptor and γ-8 were distributed in both the raft-enriched fractions and other Triton-insoluble fractions. Phosphorylation of stargazin/γ-2 and γ-8 was regulated by different sets of kinases and phosphatases in cultured cortical neurons. These results suggested that stargazin/γ-2 and γ-8 have distinct roles in postsynaptic membranes under the regulation of different intracellular signaling pathways.},
author = {Inamura, Mihoko and Itakura, Makoto and Okamoto, Hirotsugu and Hoka, Sumio and Mizoguchi, Akira and Fukazawa, Yugo and Ryuichi Shigemoto and Yamamori, Saori and Takahashi, Masami},
journal = {Neuroscience Research},
number = {1},
pages = {45 -- 53},
publisher = {Elsevier},
title = {{ Differential localization and regulation of stargazin-like protein, γ-8 and stargazin in the plasma membrane of hippocampal and cortical neurons}},
doi = {10.1016/j.neures.2006.01.004},
volume = {55},
year = {2006},
}
@article{2660,
abstract = {Pavlovian fear conditioning, a simple form of associative learning, is thought to involve the induction of associative, NMDA receptor-dependent long-term potentiation (LTP) in the lateral amygdala. Using a combined genetic and electrophysiological approach, we show here that lack of a specific GABAB receptor subtype, GABAB(1a,2), unmasks a nonassociative, NMDA receptor-independent form of presynaptic LTP at cortico-amygdala afferents. Moreover, the level of presynaptic GABA B(1a,2) receptor activation, and hence the balance between associative and nonassociative forms of LTP, can be dynamically modulated by local inhibitory activity. At the behavioral level, genetic loss of GABA B(1a) results in a generalization of conditioned fear to nonconditioned stimuli. Our findings indicate that presynaptic inhibition through GABAB(1a,2) receptors serves as an activity-dependent constraint on the induction of homosynaptic plasticity, which may be important to prevent the generalization of conditioned fear.},
author = {Shaban, Hamdy and Humeau, Yann and Herry, Cyril and Cassasus, Guillaume and Ryuichi Shigemoto and Ciocchi, Stéphane and Barbieri, Samuel and Van Der Putten, Herman V and Kaupmann, Klemens and Bettler, Bernhard and Lüthi, Andreas},
journal = {Nature Neuroscience},
number = {8},
pages = {1028 -- 1035},
publisher = {Nature Publishing Group},
title = {{Generalization of amygdala LTP and conditioned fear in the absence of presynaptic inhibition}},
doi = {10.1038/nn1732},
volume = {9},
year = {2006},
}
@article{2661,
abstract = {GABAB receptors are the G protein-coupled receptors for the main inhibitory neurotransmitter in the brain, γ-aminobutyric acid (GABA). Molecular diversity in the GABAB system arises from the GABAB1a and GABAB1b subunit isoforms that solely differ in their ectodomains by a pair of sushi repeats that is unique to GABAB1a. Using a combined genetic, physiological, and morphological approach, we now demonstrate that GABAB1 isoforms localize to distinct synaptic sites and convey separate functions in vivo. At hippocampal CA3-to-CA1 synapses, GABAB1a assembles heteroreceptors inhibiting glutamate release, while predominantly GABAB1b mediates postsynaptic inhibition. Electron microscopy reveals a synaptic distribution of GABAB1 isoforms that agrees with the observed functional differences. Transfected CA3 neurons selectively express GABAB1a in distal axons, suggesting that the sushi repeats, a conserved protein interaction motif, specify heteroreceptor localization. The constitutive absence of GABAB1a but not GABAB1b results in impaired synaptic plasticity and hippocampus-dependent memory, emphasizing molecular differences in synaptic GABAB functions.},
author = {Vigot, Réjan and Barbieri, Samuel and Bräuner-Osborne, Hans and Tureček, Rostislav and Ryuichi Shigemoto and Zhang, Yan Ping and Luján, Rafael and Jacobson, Laura H and Biermann, Barbara and Fritschy, Jean-Marc and Vacher, Claire-Marie and Müller, Matthias P and Sansig, Gilles and Guetg, Nicole and Cryan, John F and Kaupmann, Klemens and Gassmann, Martin and Oertner, Thomas G and Bettler, Bernhard},
journal = {Neuron},
number = {4},
pages = {589 -- 601},
publisher = {Elsevier},
title = {{Differential Compartmentalization and Distinct Functions of GABAB Receptor Variants}},
doi = {10.1016/j.neuron.2006.04.014},
volume = {50},
year = {2006},
}