---
_id: '1660'
abstract:
- lang: eng
text: We study the pattern frequency vector for runs in probabilistic Vector Addition
Systems with States (pVASS). Intuitively, each configuration of a given pVASS
is assigned one of finitely many patterns, and every run can thus be seen as an
infinite sequence of these patterns. The pattern frequency vector assigns to each
run the limit of pattern frequencies computed for longer and longer prefixes of
the run. If the limit does not exist, then the vector is undefined. We show that
for one-counter pVASS, the pattern frequency vector is defined and takes one of
finitely many values for almost all runs. Further, these values and their associated
probabilities can be approximated up to an arbitrarily small relative error in
polynomial time. For stable two-counter pVASS, we show the same result, but we
do not provide any upper complexity bound. As a byproduct of our study, we discover
counterexamples falsifying some classical results about stochastic Petri nets
published in the 80s.
alternative_title:
- LICS
author:
- first_name: Tomáš
full_name: Brázdil, Tomáš
last_name: Brázdil
- first_name: Stefan
full_name: Kiefer, Stefan
last_name: Kiefer
- first_name: Antonín
full_name: Kučera, Antonín
last_name: Kučera
- first_name: Petr
full_name: Novotny, Petr
id: 3CC3B868-F248-11E8-B48F-1D18A9856A87
last_name: Novotny
citation:
ama: 'Brázdil T, Kiefer S, Kučera A, Novotný P. Long-run average behaviour of probabilistic
vector addition systems. In: IEEE; 2015:44-55. doi:10.1109/LICS.2015.15'
apa: 'Brázdil, T., Kiefer, S., Kučera, A., & Novotný, P. (2015). Long-run average
behaviour of probabilistic vector addition systems (pp. 44–55). Presented at the
LICS: Logic in Computer Science, Kyoto, Japan: IEEE. https://doi.org/10.1109/LICS.2015.15'
chicago: Brázdil, Tomáš, Stefan Kiefer, Antonín Kučera, and Petr Novotný. “Long-Run
Average Behaviour of Probabilistic Vector Addition Systems,” 44–55. IEEE, 2015.
https://doi.org/10.1109/LICS.2015.15.
ieee: 'T. Brázdil, S. Kiefer, A. Kučera, and P. Novotný, “Long-run average behaviour
of probabilistic vector addition systems,” presented at the LICS: Logic in Computer
Science, Kyoto, Japan, 2015, pp. 44–55.'
ista: 'Brázdil T, Kiefer S, Kučera A, Novotný P. 2015. Long-run average behaviour
of probabilistic vector addition systems. LICS: Logic in Computer Science, LICS,
, 44–55.'
mla: Brázdil, Tomáš, et al. Long-Run Average Behaviour of Probabilistic Vector
Addition Systems. IEEE, 2015, pp. 44–55, doi:10.1109/LICS.2015.15.
short: T. Brázdil, S. Kiefer, A. Kučera, P. Novotný, in:, IEEE, 2015, pp. 44–55.
conference:
end_date: 2015-07-10
location: Kyoto, Japan
name: 'LICS: Logic in Computer Science'
start_date: 2015-07-06
date_created: 2018-12-11T11:53:19Z
date_published: 2015-07-01T00:00:00Z
date_updated: 2021-01-12T06:52:20Z
day: '01'
department:
- _id: KrCh
doi: 10.1109/LICS.2015.15
ec_funded: 1
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://arxiv.org/abs/1505.02655
month: '07'
oa: 1
oa_version: Preprint
page: 44 - 55
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
publication_status: published
publisher: IEEE
publist_id: '5490'
quality_controlled: '1'
scopus_import: 1
status: public
title: Long-run average behaviour of probabilistic vector addition systems
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
year: '2015'
...
---
_id: '1665'
abstract:
- lang: eng
text: Which genetic alterations drive tumorigenesis and how they evolve over the
course of disease and therapy are central questions in cancer biology. Here we
identify 44 recurrently mutated genes and 11 recurrent somatic copy number variations
through whole-exome sequencing of 538 chronic lymphocytic leukaemia (CLL) and
matched germline DNA samples, 278 of which were collected in a prospective clinical
trial. These include previously unrecognized putative cancer drivers (RPS15, IKZF3),
and collectively identify RNA processing and export, MYC activity, and MAPK signalling
as central pathways involved in CLL. Clonality analysis of this large data set
further enabled reconstruction of temporal relationships between driver events.
Direct comparison between matched pre-treatment and relapse samples from 59 patients
demonstrated highly frequent clonal evolution. Thus, large sequencing data sets
of clinically informative samples enable the discovery of novel genes associated
with cancer, the network of relationships between the driver events, and their
impact on disease relapse and clinical outcome.
article_processing_charge: No
article_type: original
author:
- first_name: Dan
full_name: Landau, Dan
last_name: Landau
- first_name: Eugen
full_name: Tausch, Eugen
last_name: Tausch
- first_name: Amaro
full_name: Taylor Weiner, Amaro
last_name: Taylor Weiner
- first_name: Chip
full_name: Stewart, Chip
last_name: Stewart
- first_name: Johannes
full_name: Reiter, Johannes
id: 4A918E98-F248-11E8-B48F-1D18A9856A87
last_name: Reiter
orcid: 0000-0002-0170-7353
- first_name: Jasmin
full_name: Bahlo, Jasmin
last_name: Bahlo
- first_name: Sandra
full_name: Kluth, Sandra
last_name: Kluth
- first_name: Ivana
full_name: Božić, Ivana
last_name: Božić
- first_name: Michael
full_name: Lawrence, Michael
last_name: Lawrence
- first_name: Sebastian
full_name: Böttcher, Sebastian
last_name: Böttcher
- first_name: Scott
full_name: Carter, Scott
last_name: Carter
- first_name: Kristian
full_name: Cibulskis, Kristian
last_name: Cibulskis
- first_name: Daniel
full_name: Mertens, Daniel
last_name: Mertens
- first_name: Carrie
full_name: Sougnez, Carrie
last_name: Sougnez
- first_name: Mara
full_name: Rosenberg, Mara
last_name: Rosenberg
- first_name: Julian
full_name: Hess, Julian
last_name: Hess
- first_name: Jennifer
full_name: Edelmann, Jennifer
last_name: Edelmann
- first_name: Sabrina
full_name: Kless, Sabrina
last_name: Kless
- first_name: Michael
full_name: Kneba, Michael
last_name: Kneba
- first_name: Matthias
full_name: Ritgen, Matthias
last_name: Ritgen
- first_name: Anna
full_name: Fink, Anna
last_name: Fink
- first_name: Kirsten
full_name: Fischer, Kirsten
last_name: Fischer
- first_name: Stacey
full_name: Gabriel, Stacey
last_name: Gabriel
- first_name: Eric
full_name: Lander, Eric
last_name: Lander
- first_name: Martin
full_name: Nowak, Martin
last_name: Nowak
- first_name: Hartmut
full_name: Döhner, Hartmut
last_name: Döhner
- first_name: Michael
full_name: Hallek, Michael
last_name: Hallek
- first_name: Donna
full_name: Neuberg, Donna
last_name: Neuberg
- first_name: Gad
full_name: Getz, Gad
last_name: Getz
- first_name: Stephan
full_name: Stilgenbauer, Stephan
last_name: Stilgenbauer
- first_name: Catherine
full_name: Wu, Catherine
last_name: Wu
citation:
ama: Landau D, Tausch E, Taylor Weiner A, et al. Mutations driving CLL and their
evolution in progression and relapse. Nature. 2015;526(7574):525-530. doi:10.1038/nature15395
apa: Landau, D., Tausch, E., Taylor Weiner, A., Stewart, C., Reiter, J., Bahlo,
J., … Wu, C. (2015). Mutations driving CLL and their evolution in progression
and relapse. Nature. Nature Publishing Group. https://doi.org/10.1038/nature15395
chicago: Landau, Dan, Eugen Tausch, Amaro Taylor Weiner, Chip Stewart, Johannes
Reiter, Jasmin Bahlo, Sandra Kluth, et al. “Mutations Driving CLL and Their Evolution
in Progression and Relapse.” Nature. Nature Publishing Group, 2015. https://doi.org/10.1038/nature15395.
ieee: D. Landau et al., “Mutations driving CLL and their evolution in progression
and relapse,” Nature, vol. 526, no. 7574. Nature Publishing Group, pp.
525–530, 2015.
ista: Landau D, Tausch E, Taylor Weiner A, Stewart C, Reiter J, Bahlo J, Kluth S,
Božić I, Lawrence M, Böttcher S, Carter S, Cibulskis K, Mertens D, Sougnez C,
Rosenberg M, Hess J, Edelmann J, Kless S, Kneba M, Ritgen M, Fink A, Fischer K,
Gabriel S, Lander E, Nowak M, Döhner H, Hallek M, Neuberg D, Getz G, Stilgenbauer
S, Wu C. 2015. Mutations driving CLL and their evolution in progression and relapse.
Nature. 526(7574), 525–530.
mla: Landau, Dan, et al. “Mutations Driving CLL and Their Evolution in Progression
and Relapse.” Nature, vol. 526, no. 7574, Nature Publishing Group, 2015,
pp. 525–30, doi:10.1038/nature15395.
short: D. Landau, E. Tausch, A. Taylor Weiner, C. Stewart, J. Reiter, J. Bahlo,
S. Kluth, I. Božić, M. Lawrence, S. Böttcher, S. Carter, K. Cibulskis, D. Mertens,
C. Sougnez, M. Rosenberg, J. Hess, J. Edelmann, S. Kless, M. Kneba, M. Ritgen,
A. Fink, K. Fischer, S. Gabriel, E. Lander, M. Nowak, H. Döhner, M. Hallek, D.
Neuberg, G. Getz, S. Stilgenbauer, C. Wu, Nature 526 (2015) 525–530.
date_created: 2018-12-11T11:53:21Z
date_published: 2015-10-22T00:00:00Z
date_updated: 2021-01-12T06:52:23Z
day: '22'
department:
- _id: KrCh
doi: 10.1038/nature15395
ec_funded: 1
external_id:
pmid:
- '26466571'
intvolume: ' 526'
issue: '7574'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4815041/
month: '10'
oa: 1
oa_version: Submitted Version
page: 525 - 530
pmid: 1
project:
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '279307'
name: 'Quantitative Graph Games: Theory and Applications'
- _id: 2584A770-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: P 23499-N23
name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25832EC2-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: S 11407_N23
name: Rigorous Systems Engineering
publication: Nature
publication_status: published
publisher: Nature Publishing Group
publist_id: '5484'
quality_controlled: '1'
scopus_import: 1
status: public
title: Mutations driving CLL and their evolution in progression and relapse
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 526
year: '2015'
...
---
_id: '1663'
abstract:
- lang: eng
text: CREB-binding protein (CBP) and p300 are transcriptional coactivators involved
in numerous biological processes that affect cell growth, transformation, differentiation,
and development. In this study, we provide evidence of the involvement of homeodomain-interacting
protein kinase 2 (HIPK2) in the regulation of CBP activity. We show that HIPK2
interacts with and phosphorylates several regions of CBP. We demonstrate that
serines 2361, 2363, 2371, 2376, and 2381 are responsible for the HIPK2-induced
mobility shift of CBP C-terminal activation domain. Moreover, we show that HIPK2
strongly potentiates the transcriptional activity of CBP. However, our data suggest
that HIPK2 activates CBP mainly by counteracting the repressive action of cell
cycle regulatory domain 1 (CRD1), located between amino acids 977 and 1076, independently
of CBP phosphorylation. Our findings thus highlight a complex regulation of CBP
activity by HIPK2, which might be relevant for the control of specific sets of
target genes involved in cellular proliferation, differentiation and apoptosis.
author:
- first_name: Krisztián
full_name: Kovács, Krisztián
id: 2AB5821E-F248-11E8-B48F-1D18A9856A87
last_name: Kovács
- first_name: Myriam
full_name: Steinmann, Myriam
last_name: Steinmann
- first_name: Olivier
full_name: Halfon, Olivier
last_name: Halfon
- first_name: Pierre
full_name: Magistretti, Pierre
last_name: Magistretti
- first_name: Jean
full_name: Cardinaux, Jean
last_name: Cardinaux
citation:
ama: Kovács K, Steinmann M, Halfon O, Magistretti P, Cardinaux J. Complex regulation
of CREB-binding protein by homeodomain-interacting protein kinase 2. Cellular
Signalling. 2015;27(11):2252-2260. doi:10.1016/j.cellsig.2015.08.001
apa: Kovács, K., Steinmann, M., Halfon, O., Magistretti, P., & Cardinaux, J.
(2015). Complex regulation of CREB-binding protein by homeodomain-interacting
protein kinase 2. Cellular Signalling. Elsevier. https://doi.org/10.1016/j.cellsig.2015.08.001
chicago: Kovács, Krisztián, Myriam Steinmann, Olivier Halfon, Pierre Magistretti,
and Jean Cardinaux. “Complex Regulation of CREB-Binding Protein by Homeodomain-Interacting
Protein Kinase 2.” Cellular Signalling. Elsevier, 2015. https://doi.org/10.1016/j.cellsig.2015.08.001.
ieee: K. Kovács, M. Steinmann, O. Halfon, P. Magistretti, and J. Cardinaux, “Complex
regulation of CREB-binding protein by homeodomain-interacting protein kinase 2,”
Cellular Signalling, vol. 27, no. 11. Elsevier, pp. 2252–2260, 2015.
ista: Kovács K, Steinmann M, Halfon O, Magistretti P, Cardinaux J. 2015. Complex
regulation of CREB-binding protein by homeodomain-interacting protein kinase 2.
Cellular Signalling. 27(11), 2252–2260.
mla: Kovács, Krisztián, et al. “Complex Regulation of CREB-Binding Protein by Homeodomain-Interacting
Protein Kinase 2.” Cellular Signalling, vol. 27, no. 11, Elsevier, 2015,
pp. 2252–60, doi:10.1016/j.cellsig.2015.08.001.
short: K. Kovács, M. Steinmann, O. Halfon, P. Magistretti, J. Cardinaux, Cellular
Signalling 27 (2015) 2252–2260.
date_created: 2018-12-11T11:53:20Z
date_published: 2015-11-01T00:00:00Z
date_updated: 2021-01-12T06:52:22Z
day: '01'
ddc:
- '570'
department:
- _id: JoCs
doi: 10.1016/j.cellsig.2015.08.001
ec_funded: 1
file:
- access_level: local
checksum: 4ee690b6444b7a43523237f0941457d1
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:18:03Z
date_updated: 2020-07-14T12:45:10Z
file_id: '5321'
file_name: IST-2016-578-v1+1_CLS-D-15-00072R1_.pdf
file_size: 1735337
relation: main_file
file_date_updated: 2020-07-14T12:45:10Z
has_accepted_license: '1'
intvolume: ' 27'
issue: '11'
language:
- iso: eng
month: '11'
oa_version: Published Version
page: 2252 - 2260
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
publication: Cellular Signalling
publication_status: published
publisher: Elsevier
publist_id: '5487'
pubrep_id: '578'
quality_controlled: '1'
scopus_import: 1
status: public
title: Complex regulation of CREB-binding protein by homeodomain-interacting protein
kinase 2
tmp:
image: /images/cc_by_nc_nd.png
legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
(CC BY-NC-ND 4.0)
short: CC BY-NC-ND (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 27
year: '2015'
...
---
_id: '1667'
abstract:
- lang: eng
text: We consider parametric version of fixed-delay continuoustime Markov chains
(or equivalently deterministic and stochastic Petri nets, DSPN) where fixed-delay
transitions are specified by parameters, rather than concrete values. Our goal
is to synthesize values of these parameters that, for a given cost function, minimise
expected total cost incurred before reaching a given set of target states. We
show that under mild assumptions, optimal values of parameters can be effectively
approximated using translation to a Markov decision process (MDP) whose actions
correspond to discretized values of these parameters. To this end we identify
and overcome several interesting phenomena arising in systems with fixed delays.
acknowledgement: The research leading to these results has received funding from the
People Programme (Marie Curie Actions) of the European Union’s Seventh Framework
Programme (FP7/2007-2013) under REA grant agreement n∘ [291734]. This work is partly
supported by the German Research Council (DFG) as part of the Transregional Collaborative
Research Center AVACS (SFB/TR 14), by the EU 7th Framework Programme under grant
agreement no. 295261 (MEALS) and 318490 (SENSATION), by the Czech Science Foundation,
grant No. 15-17564S, and by the CAS/SAFEA International Partnership Program for
Creative Research Teams.
alternative_title:
- LNCS
author:
- first_name: Tomáš
full_name: Brázdil, Tomáš
last_name: Brázdil
- first_name: L'Uboš
full_name: Korenčiak, L'Uboš
last_name: Korenčiak
- first_name: Jan
full_name: Krčál, Jan
last_name: Krčál
- first_name: Petr
full_name: Novotny, Petr
id: 3CC3B868-F248-11E8-B48F-1D18A9856A87
last_name: Novotny
- first_name: Vojtěch
full_name: Řehák, Vojtěch
last_name: Řehák
citation:
ama: Brázdil T, Korenčiak L, Krčál J, Novotný P, Řehák V. Optimizing performance
of continuous-time stochastic systems using timeout synthesis. 2015;9259:141-159.
doi:10.1007/978-3-319-22264-6_10
apa: 'Brázdil, T., Korenčiak, L., Krčál, J., Novotný, P., & Řehák, V. (2015).
Optimizing performance of continuous-time stochastic systems using timeout synthesis.
Presented at the QEST: Quantitative Evaluation of Systems, Madrid, Spain: Springer.
https://doi.org/10.1007/978-3-319-22264-6_10'
chicago: Brázdil, Tomáš, L’Uboš Korenčiak, Jan Krčál, Petr Novotný, and Vojtěch
Řehák. “Optimizing Performance of Continuous-Time Stochastic Systems Using Timeout
Synthesis.” Lecture Notes in Computer Science. Springer, 2015. https://doi.org/10.1007/978-3-319-22264-6_10.
ieee: T. Brázdil, L. Korenčiak, J. Krčál, P. Novotný, and V. Řehák, “Optimizing
performance of continuous-time stochastic systems using timeout synthesis,” vol.
9259. Springer, pp. 141–159, 2015.
ista: Brázdil T, Korenčiak L, Krčál J, Novotný P, Řehák V. 2015. Optimizing performance
of continuous-time stochastic systems using timeout synthesis. 9259, 141–159.
mla: Brázdil, Tomáš, et al. Optimizing Performance of Continuous-Time Stochastic
Systems Using Timeout Synthesis. Vol. 9259, Springer, 2015, pp. 141–59, doi:10.1007/978-3-319-22264-6_10.
short: T. Brázdil, L. Korenčiak, J. Krčál, P. Novotný, V. Řehák, 9259 (2015) 141–159.
conference:
end_date: 2015-09-03
location: Madrid, Spain
name: 'QEST: Quantitative Evaluation of Systems'
start_date: 2015-09-01
date_created: 2018-12-11T11:53:22Z
date_published: 2015-08-22T00:00:00Z
date_updated: 2021-01-12T06:52:24Z
day: '22'
department:
- _id: KrCh
doi: 10.1007/978-3-319-22264-6_10
ec_funded: 1
intvolume: ' 9259'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://arxiv.org/abs/1407.4777
month: '08'
oa: 1
oa_version: Preprint
page: 141 - 159
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
publication_status: published
publisher: Springer
publist_id: '5482'
quality_controlled: '1'
scopus_import: 1
series_title: Lecture Notes in Computer Science
status: public
title: Optimizing performance of continuous-time stochastic systems using timeout
synthesis
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 9259
year: '2015'
...
---
_id: '1664'
abstract:
- lang: eng
text: Over a century of research into the origin of turbulence in wall-bounded shear
flows has resulted in a puzzling picture in which turbulence appears in a variety
of different states competing with laminar background flow. At moderate flow speeds,
turbulence is confined to localized patches; it is only at higher speeds that
the entire flow becomes turbulent. The origin of the different states encountered
during this transition, the front dynamics of the turbulent regions and the transformation
to full turbulence have yet to be explained. By combining experiments, theory
and computer simulations, here we uncover a bifurcation scenario that explains
the transformation to fully turbulent pipe flow and describe the front dynamics
of the different states encountered in the process. Key to resolving this problem
is the interpretation of the flow as a bistable system with nonlinear propagation
(advection) of turbulent fronts. These findings bridge the gap between our understanding
of the onset of turbulence and fully turbulent flows.
acknowledgement: We acknowledge the Deutsche Forschungsgemeinschaft (Project No. FOR
1182), and the European Research Council under the European Union’s Seventh Framework
Programme (FP/2007-2013)/ERC Grant Agreement 306589 for financial support. B.S.
acknowledges financial support from the Chinese State Scholarship Fund under grant
number 2010629145. B.S. acknowledges support from the International Max Planck Research
School for the Physics of Biological and Complex Systems and the Göttingen Graduate
School for Neurosciences and Molecular Biosciences. We acknowledge computing resources
from GWDG (Gesellschaft für wissenschaftliche Datenverarbeitung Göttingen) and the
Jülich Supercomputing Centre (grant HGU16) where the simulations were performed.
author:
- first_name: Dwight
full_name: Barkley, Dwight
last_name: Barkley
- first_name: Baofang
full_name: Song, Baofang
last_name: Song
- first_name: Mukund
full_name: Vasudevan, Mukund
id: 3C5A959A-F248-11E8-B48F-1D18A9856A87
last_name: Vasudevan
- first_name: Grégoire M
full_name: Lemoult, Grégoire M
id: 4787FE80-F248-11E8-B48F-1D18A9856A87
last_name: Lemoult
- first_name: Marc
full_name: Avila, Marc
last_name: Avila
- first_name: Björn
full_name: Hof, Björn
id: 3A374330-F248-11E8-B48F-1D18A9856A87
last_name: Hof
orcid: 0000-0003-2057-2754
citation:
ama: Barkley D, Song B, Vasudevan M, Lemoult GM, Avila M, Hof B. The rise of fully
turbulent flow. Nature. 2015;526(7574):550-553. doi:10.1038/nature15701
apa: Barkley, D., Song, B., Vasudevan, M., Lemoult, G. M., Avila, M., & Hof,
B. (2015). The rise of fully turbulent flow. Nature. Nature Publishing
Group. https://doi.org/10.1038/nature15701
chicago: Barkley, Dwight, Baofang Song, Mukund Vasudevan, Grégoire M Lemoult, Marc
Avila, and Björn Hof. “The Rise of Fully Turbulent Flow.” Nature. Nature
Publishing Group, 2015. https://doi.org/10.1038/nature15701.
ieee: D. Barkley, B. Song, M. Vasudevan, G. M. Lemoult, M. Avila, and B. Hof, “The
rise of fully turbulent flow,” Nature, vol. 526, no. 7574. Nature Publishing
Group, pp. 550–553, 2015.
ista: Barkley D, Song B, Vasudevan M, Lemoult GM, Avila M, Hof B. 2015. The rise
of fully turbulent flow. Nature. 526(7574), 550–553.
mla: Barkley, Dwight, et al. “The Rise of Fully Turbulent Flow.” Nature,
vol. 526, no. 7574, Nature Publishing Group, 2015, pp. 550–53, doi:10.1038/nature15701.
short: D. Barkley, B. Song, M. Vasudevan, G.M. Lemoult, M. Avila, B. Hof, Nature
526 (2015) 550–553.
date_created: 2018-12-11T11:53:20Z
date_published: 2015-10-21T00:00:00Z
date_updated: 2021-01-12T06:52:22Z
day: '21'
department:
- _id: BjHo
doi: 10.1038/nature15701
ec_funded: 1
intvolume: ' 526'
issue: '7574'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://arxiv.org/abs/1510.09143
month: '10'
oa: 1
oa_version: Preprint
page: 550 - 553
project:
- _id: 25152F3A-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '306589'
name: Decoding the complexity of turbulence at its origin
publication: Nature
publication_status: published
publisher: Nature Publishing Group
publist_id: '5485'
quality_controlled: '1'
scopus_import: 1
status: public
title: The rise of fully turbulent flow
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 526
year: '2015'
...