---
_id: '2655'
abstract:
- lang: eng
text: Input-dependent left-right asymmetry of NMDA receptor ε2 (NR2B) subunit allocation
was discovered in hippocampal Schaffer collateral (Sch) and commissural fiber
pyramidal cell synapses (Kawakami et al., 2003). To investigate whether this asymmetrical
ε2 allocation is also related to the types of the postsynaptic cells, we compared
postembedding immunogold labeling for ε2 in left and right Sch synapses on pyramidal
cells and interneurons. To facilitate the detection of ε2 density difference,
we used ε1 (NR2A) knock-out (KO) mice, which have a simplified NMDA receptor subunit
composition. The labeling density for ε2 but not ζ1 (NR1) and subtype 2/3 glutamate
receptor (GluR2/3) in Sch-CA1 pyramidal cell synapses was significantly different
between the left and right hippocampus with opposite directions in strata oriens
and radiatum; the left to right ratio of ε2 labeling density was 1:1.50 in stratum
oriens and 1.44:1 in stratum radiatum. No significant difference, however, was
detected in CA1 stratum radiatum between the left and right Sch-GluR4-positive
(mostly parvalbumin-positive) and Sch-GluR4-negative interneuron synapses. Consistent
with the anatomical asymmetry, the amplitude ratio of NMDA EPSCs to non-NMDA EPSCs
in pyramidal cells was approximately two times larger in right than left stratum
radiatum and vice versa in stratum oriens of ε1 KO mice. Moreover, the amplitude
of long-term potentiation in the Sch-CA1 synapses of left stratum radiatum was
significantly larger than that in the right corresponding synapses. These results
indicate that the asymmetry of ε2 distribution is target cell specific, resulting
in the left-right difference in NMDA receptor content and plasticity in Sch-CA1
pyramidal cell synapses in ε1 KO mice.
author:
- first_name: Yue
full_name: Wu, Yue
last_name: Wu
- first_name: Ryosuke
full_name: Kawakami, Ryosuke
last_name: Kawakami
- first_name: Yoshiaki
full_name: Shinohara, Yoshiaki
last_name: Shinohara
- first_name: Masahiro
full_name: Fukaya, Masahiro
last_name: Fukaya
- first_name: Kenji
full_name: Sakimura, Kenji
last_name: Sakimura
- first_name: Masayoshi
full_name: Mishina, Masayoshi
last_name: Mishina
- first_name: Masahiko
full_name: Watanabe, Masahiko
last_name: Watanabe
- first_name: Isao
full_name: Ito, Isao
last_name: Ito
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
citation:
ama: Wu Y, Kawakami R, Shinohara Y, et al. Target-cell-specific left-right asymmetry
of NMDA receptor content in Schaffer collateral synapses in ε1/NR2A knock-out
mice. Journal of Neuroscience. 2005;25(40):9213-9226. doi:10.1523/JNEUROSCI.2134-05.2005
apa: Wu, Y., Kawakami, R., Shinohara, Y., Fukaya, M., Sakimura, K., Mishina, M.,
… Shigemoto, R. (2005). Target-cell-specific left-right asymmetry of NMDA receptor
content in Schaffer collateral synapses in ε1/NR2A knock-out mice. Journal
of Neuroscience. Society for Neuroscience. https://doi.org/10.1523/JNEUROSCI.2134-05.2005
chicago: Wu, Yue, Ryosuke Kawakami, Yoshiaki Shinohara, Masahiro Fukaya, Kenji Sakimura,
Masayoshi Mishina, Masahiko Watanabe, Isao Ito, and Ryuichi Shigemoto. “Target-Cell-Specific
Left-Right Asymmetry of NMDA Receptor Content in Schaffer Collateral Synapses
in Ε1/NR2A Knock-out Mice.” Journal of Neuroscience. Society for Neuroscience,
2005. https://doi.org/10.1523/JNEUROSCI.2134-05.2005.
ieee: Y. Wu et al., “Target-cell-specific left-right asymmetry of NMDA receptor
content in Schaffer collateral synapses in ε1/NR2A knock-out mice,” Journal
of Neuroscience, vol. 25, no. 40. Society for Neuroscience, pp. 9213–9226,
2005.
ista: Wu Y, Kawakami R, Shinohara Y, Fukaya M, Sakimura K, Mishina M, Watanabe M,
Ito I, Shigemoto R. 2005. Target-cell-specific left-right asymmetry of NMDA receptor
content in Schaffer collateral synapses in ε1/NR2A knock-out mice. Journal of
Neuroscience. 25(40), 9213–9226.
mla: Wu, Yue, et al. “Target-Cell-Specific Left-Right Asymmetry of NMDA Receptor
Content in Schaffer Collateral Synapses in Ε1/NR2A Knock-out Mice.” Journal
of Neuroscience, vol. 25, no. 40, Society for Neuroscience, 2005, pp. 9213–26,
doi:10.1523/JNEUROSCI.2134-05.2005.
short: Y. Wu, R. Kawakami, Y. Shinohara, M. Fukaya, K. Sakimura, M. Mishina, M.
Watanabe, I. Ito, R. Shigemoto, Journal of Neuroscience 25 (2005) 9213–9226.
date_created: 2018-12-11T11:58:54Z
date_published: 2005-10-05T00:00:00Z
date_updated: 2021-01-12T06:58:51Z
day: '05'
doi: 10.1523/JNEUROSCI.2134-05.2005
extern: 1
intvolume: ' 25'
issue: '40'
month: '10'
page: 9213 - 9226
publication: Journal of Neuroscience
publication_status: published
publisher: Society for Neuroscience
publist_id: '4243'
quality_controlled: 0
status: public
title: Target-cell-specific left-right asymmetry of NMDA receptor content in Schaffer
collateral synapses in ε1/NR2A knock-out mice
type: journal_article
volume: 25
year: '2005'
...
---
_id: '2653'
abstract:
- lang: eng
text: Synaptic vesicle release occurs at a specialized membrane domain known as
the presynaptic active zone (AZ). Several membrane proteins are involved in the
vesicle release processes such as docking, priming, and exocytotic fusion. Cytomatrix
at the active zone (CAZ) proteins are structural components of the AZ and are
highly concentrated in it. Localization of other release-related proteins including
target soluble N-ethylmaleimide-sensitive-factor attachment protein receptor (t-SNARE)
proteins, however, has not been well demonstrated in the AZ. Here, we used sodium
dodecyl sulfate-digested freeze-fracture replica labeling (SDS-FRL) to analyze
quantitatively the distribution of CAZ and t-SNARE proteins in the hippocampal
CA3 area. The AZ in replicated membrane was identified by immunolabeling for CAZ
proteins (CAZ-associated structural protein [CAST] and Bassoon). Clusters of immunogold
particles for these proteins were found on the P-face of presynaptic terminals
of the mossy fiber and associational/commissural (AJC) fiber. Co-labeling with
CAST revealed distribution of the t-SNARE proteins syntaxin and synaptosomal-associated
protein of 25 kDa (SNAP-25) in the AZ as well as in the extrasynaptic membrane
surrounding the AZ (SZ). Quantitative analysis demonstrated that the density of
immunoparticles for CAST in the AZ was more than 100 times higher than in the
SZ, whereas that for syntaxin and SNAP-25 was not significantly different between
the AZ and SZ in both the A/C and mossy fiber terminals. These results support
the involvement of the t-SNARE proteins in exocytotic fusion in the AZ and the
role of CAST in specialization of the membrane domain for the AZ.
author:
- first_name: Akari
full_name: Hagiwara, Akari
last_name: Hagiwara
- first_name: Yugo
full_name: Fukazawa, Yugo
last_name: Fukazawa
- first_name: Maki
full_name: Deguchi-Tawarada, Maki
last_name: Deguchi Tawarada
- first_name: Toshihisa
full_name: Ohtsuka, Toshihisa
last_name: Ohtsuka
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
citation:
ama: Hagiwara A, Fukazawa Y, Deguchi Tawarada M, Ohtsuka T, Shigemoto R. Differential
distribution of release-related proteins in the hippocampal CA3 area as revealed
by freeze-fracture replica labeling. Journal of Comparative Neurology.
2005;489(2):195-216. doi:10.1002/cne.20633
apa: Hagiwara, A., Fukazawa, Y., Deguchi Tawarada, M., Ohtsuka, T., & Shigemoto,
R. (2005). Differential distribution of release-related proteins in the hippocampal
CA3 area as revealed by freeze-fracture replica labeling. Journal of Comparative
Neurology. Wiley-Blackwell. https://doi.org/10.1002/cne.20633
chicago: Hagiwara, Akari, Yugo Fukazawa, Maki Deguchi Tawarada, Toshihisa Ohtsuka,
and Ryuichi Shigemoto. “Differential Distribution of Release-Related Proteins
in the Hippocampal CA3 Area as Revealed by Freeze-Fracture Replica Labeling.”
Journal of Comparative Neurology. Wiley-Blackwell, 2005. https://doi.org/10.1002/cne.20633.
ieee: A. Hagiwara, Y. Fukazawa, M. Deguchi Tawarada, T. Ohtsuka, and R. Shigemoto,
“Differential distribution of release-related proteins in the hippocampal CA3
area as revealed by freeze-fracture replica labeling,” Journal of Comparative
Neurology, vol. 489, no. 2. Wiley-Blackwell, pp. 195–216, 2005.
ista: Hagiwara A, Fukazawa Y, Deguchi Tawarada M, Ohtsuka T, Shigemoto R. 2005.
Differential distribution of release-related proteins in the hippocampal CA3 area
as revealed by freeze-fracture replica labeling. Journal of Comparative Neurology.
489(2), 195–216.
mla: Hagiwara, Akari, et al. “Differential Distribution of Release-Related Proteins
in the Hippocampal CA3 Area as Revealed by Freeze-Fracture Replica Labeling.”
Journal of Comparative Neurology, vol. 489, no. 2, Wiley-Blackwell, 2005,
pp. 195–216, doi:10.1002/cne.20633.
short: A. Hagiwara, Y. Fukazawa, M. Deguchi Tawarada, T. Ohtsuka, R. Shigemoto,
Journal of Comparative Neurology 489 (2005) 195–216.
date_created: 2018-12-11T11:58:53Z
date_published: 2005-08-22T00:00:00Z
date_updated: 2021-01-12T06:58:50Z
day: '22'
doi: 10.1002/cne.20633
extern: 1
intvolume: ' 489'
issue: '2'
month: '08'
page: 195 - 216
publication: Journal of Comparative Neurology
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4244'
quality_controlled: 0
status: public
title: Differential distribution of release-related proteins in the hippocampal CA3
area as revealed by freeze-fracture replica labeling
type: journal_article
volume: 489
year: '2005'
...
---
_id: '2656'
abstract:
- lang: eng
text: Previous studies have shown that neurons in the sacral dorsal commissural
nucleus (SDCN) express neurokinin-1 receptor (NK1R) and can be modulated by the
co-release of GABA and glycine (Gly) from single presynaptic terminal. These results
raise the possibility that GABA/Gly-cocontaining terminals might make synaptic
contacts with NK1R-expressing neurons in the SDCN. In order to provide morphological
evidence for this hypothesis, the triple-immunohistochemical studies were performed
in the SDCN. Triple-immunofluorescence histochemical study showed that some axon
terminals in close association with NK1R-immunopositive (NK1R-ip) neurons in the
SDCN were immunopositive for both glutamic acid decarboxylase (GAD) and glycine
transporter 2 (GlyT2). In electron microscopic dual- and triple-immunohistochemistry
for GAD/GlyT2, GAD/NK1R, GlyT2/NK1R, or GAD/GlyT2/NK1R also revealed dually labeled
(GAD/GlyT2-ip) synaptic terminals upon SDCN neurons, as well as GAD- and/or GlyT2-ip
axon terminals in synaptic contact with NK1R-ip SDCN neurons. These results suggested
that some synaptic terminals upon NK1R-expressing SDCN neurons co-released both
GABA and Gly.
author:
- first_name: Yu
full_name: Feng, Yu-Peng
last_name: Feng
- first_name: Yun
full_name: Li, Yun-Qing
last_name: Li
- first_name: Wen
full_name: Wang, Wen
last_name: Wang
- first_name: Sheng
full_name: Wu, Sheng-Xi
last_name: Wu
- first_name: Tao
full_name: Chen, Tao
last_name: Chen
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Noboru
full_name: Mizuno, Noboru
last_name: Mizuno
citation:
ama: Feng Y, Li Y, Wang W, et al. Morphological evidence for GABA/glycine-cocontaining
terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in
the sacral dorsal commissural nucleus of the rat. Neuroscience Letters.
2005;388(3):144-148. doi:10.1016/j.neulet.2005.06.068
apa: Feng, Y., Li, Y., Wang, W., Wu, S., Chen, T., Shigemoto, R., & Mizuno,
N. (2005). Morphological evidence for GABA/glycine-cocontaining terminals in synaptic
contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural
nucleus of the rat. Neuroscience Letters. Elsevier. https://doi.org/10.1016/j.neulet.2005.06.068
chicago: Feng, Yu, Yun Li, Wen Wang, Sheng Wu, Tao Chen, Ryuichi Shigemoto, and
Noboru Mizuno. “Morphological Evidence for GABA/Glycine-Cocontaining Terminals
in Synaptic Contact with Neurokinin-1 Receptor-Expressing Neurons in the Sacral
Dorsal Commissural Nucleus of the Rat.” Neuroscience Letters. Elsevier,
2005. https://doi.org/10.1016/j.neulet.2005.06.068.
ieee: Y. Feng et al., “Morphological evidence for GABA/glycine-cocontaining
terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in
the sacral dorsal commissural nucleus of the rat,” Neuroscience Letters,
vol. 388, no. 3. Elsevier, pp. 144–148, 2005.
ista: Feng Y, Li Y, Wang W, Wu S, Chen T, Shigemoto R, Mizuno N. 2005. Morphological
evidence for GABA/glycine-cocontaining terminals in synaptic contact with neurokinin-1
receptor-expressing neurons in the sacral dorsal commissural nucleus of the rat.
Neuroscience Letters. 388(3), 144–148.
mla: Feng, Yu, et al. “Morphological Evidence for GABA/Glycine-Cocontaining Terminals
in Synaptic Contact with Neurokinin-1 Receptor-Expressing Neurons in the Sacral
Dorsal Commissural Nucleus of the Rat.” Neuroscience Letters, vol. 388,
no. 3, Elsevier, 2005, pp. 144–48, doi:10.1016/j.neulet.2005.06.068.
short: Y. Feng, Y. Li, W. Wang, S. Wu, T. Chen, R. Shigemoto, N. Mizuno, Neuroscience
Letters 388 (2005) 144–148.
date_created: 2018-12-11T11:58:54Z
date_published: 2005-11-18T00:00:00Z
date_updated: 2021-01-12T06:58:51Z
day: '18'
doi: 10.1016/j.neulet.2005.06.068
extern: 1
intvolume: ' 388'
issue: '3'
month: '11'
page: 144 - 148
publication: Neuroscience Letters
publication_status: published
publisher: Elsevier
publist_id: '4241'
quality_controlled: 0
status: public
title: Morphological evidence for GABA/glycine-cocontaining terminals in synaptic
contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural
nucleus of the rat
type: journal_article
volume: 388
year: '2005'
...
---
_id: '2744'
abstract:
- lang: eng
text: We study the long time evolution of a quantum particle interacting with a
random potential in the Boltzmann-Grad low density limit. We prove that the phase
space density of the quantum evolution defined through the Husimi function converges
weakly to a linear Boltzmann equation. The Boltzmann collision kernel is given
by the full quantum scattering cross-section of the obstacle potential.
author:
- first_name: David
full_name: Eng, David
last_name: Eng
- first_name: László
full_name: László Erdös
id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
last_name: Erdös
orcid: 0000-0001-5366-9603
citation:
ama: Eng D, Erdös L. The linear Boltzmann equation as the low density limit of a
random Schrödinger equation. Reviews in Mathematical Physics. 2005;17(6):669-743.
doi:10.1142/S0129055X0500242X
apa: Eng, D., & Erdös, L. (2005). The linear Boltzmann equation as the low density
limit of a random Schrödinger equation. Reviews in Mathematical Physics.
World Scientific Publishing. https://doi.org/10.1142/S0129055X0500242X
chicago: Eng, David, and László Erdös. “The Linear Boltzmann Equation as the Low
Density Limit of a Random Schrödinger Equation.” Reviews in Mathematical Physics.
World Scientific Publishing, 2005. https://doi.org/10.1142/S0129055X0500242X.
ieee: D. Eng and L. Erdös, “The linear Boltzmann equation as the low density limit
of a random Schrödinger equation,” Reviews in Mathematical Physics, vol.
17, no. 6. World Scientific Publishing, pp. 669–743, 2005.
ista: Eng D, Erdös L. 2005. The linear Boltzmann equation as the low density limit
of a random Schrödinger equation. Reviews in Mathematical Physics. 17(6), 669–743.
mla: Eng, David, and László Erdös. “The Linear Boltzmann Equation as the Low Density
Limit of a Random Schrödinger Equation.” Reviews in Mathematical Physics,
vol. 17, no. 6, World Scientific Publishing, 2005, pp. 669–743, doi:10.1142/S0129055X0500242X.
short: D. Eng, L. Erdös, Reviews in Mathematical Physics 17 (2005) 669–743.
date_created: 2018-12-11T11:59:22Z
date_published: 2005-07-01T00:00:00Z
date_updated: 2021-01-12T06:59:25Z
day: '01'
doi: 10.1142/S0129055X0500242X
extern: 1
intvolume: ' 17'
issue: '6'
month: '07'
page: 669 - 743
publication: Reviews in Mathematical Physics
publication_status: published
publisher: World Scientific Publishing
publist_id: '4148'
quality_controlled: 0
status: public
title: The linear Boltzmann equation as the low density limit of a random Schrödinger
equation
type: journal_article
volume: 17
year: '2005'
...
---
_id: '2743'
abstract:
- lang: eng
text: We consider the supersymmetric quantum mechanical system which is obtained
by dimensionally reducing d = 6, N = 1 supersymmetric gauge theory with gauge
group U(1) and a single charged hypermultiplet. Using the deformation method and
ideas introduced by Porrati and Rozenberg [1], we present a detailed proof of
the existence of a normalizable ground state for this system.
author:
- first_name: László
full_name: László Erdös
id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
last_name: Erdös
orcid: 0000-0001-5366-9603
- first_name: David
full_name: Hasler, David G
last_name: Hasler
- first_name: Jan
full_name: Solovej, Jan P
last_name: Solovej
citation:
ama: 'Erdös L, Hasler D, Solovej J. Existence of the D0-D4 bound state: A detailed
proof. Annales Henri Poincare. 2005;6(2):247-267. doi:10.1007/s00023-005-0205-0'
apa: 'Erdös, L., Hasler, D., & Solovej, J. (2005). Existence of the D0-D4 bound
state: A detailed proof. Annales Henri Poincare. Birkhäuser. https://doi.org/10.1007/s00023-005-0205-0'
chicago: 'Erdös, László, David Hasler, and Jan Solovej. “Existence of the D0-D4
Bound State: A Detailed Proof.” Annales Henri Poincare. Birkhäuser, 2005.
https://doi.org/10.1007/s00023-005-0205-0.'
ieee: 'L. Erdös, D. Hasler, and J. Solovej, “Existence of the D0-D4 bound state:
A detailed proof,” Annales Henri Poincare, vol. 6, no. 2. Birkhäuser, pp.
247–267, 2005.'
ista: 'Erdös L, Hasler D, Solovej J. 2005. Existence of the D0-D4 bound state: A
detailed proof. Annales Henri Poincare. 6(2), 247–267.'
mla: 'Erdös, László, et al. “Existence of the D0-D4 Bound State: A Detailed Proof.”
Annales Henri Poincare, vol. 6, no. 2, Birkhäuser, 2005, pp. 247–67, doi:10.1007/s00023-005-0205-0.'
short: L. Erdös, D. Hasler, J. Solovej, Annales Henri Poincare 6 (2005) 247–267.
date_created: 2018-12-11T11:59:22Z
date_published: 2005-04-01T00:00:00Z
date_updated: 2021-01-12T06:59:24Z
day: '01'
doi: 10.1007/s00023-005-0205-0
extern: 1
intvolume: ' 6'
issue: '2'
month: '04'
page: 247 - 267
publication: Annales Henri Poincare
publication_status: published
publisher: Birkhäuser
publist_id: '4149'
quality_controlled: 0
status: public
title: 'Existence of the D0-D4 bound state: A detailed proof'
type: journal_article
volume: 6
year: '2005'
...
---
_id: '2788'
abstract:
- lang: eng
text: We present the results of an experimental investigation into the nature and
structure of turbulent pipe flow at moderate Reynolds numbers. A turbulence regeneration
mechanism is identified which sustains a symmetric traveling wave within the flow.
The periodicity of the mechanism allows comparison to the wavelength of numerically
observed exact traveling wave solutions and close agreement is found. The advection
speed of the upstream turbulence laminar interface in the experimental flow is
observed to form a lower bound on the phase velocities of the exact traveling
wave solutions. Overall our observations suggest that the dynamics of the turbulent
flow at moderate Reynolds numbers are governed by unstable nonlinear traveling
waves.
author:
- first_name: Björn
full_name: Björn Hof
id: 3A374330-F248-11E8-B48F-1D18A9856A87
last_name: Hof
orcid: 0000-0003-2057-2754
- first_name: Casimir
full_name: van Doorne, Casimir W
last_name: Van Doorne
- first_name: Jerry
full_name: Westerweel, Jerry
last_name: Westerweel
- first_name: Frans
full_name: Nieuwstadt, Frans T
last_name: Nieuwstadt
citation:
ama: Hof B, Van Doorne C, Westerweel J, Nieuwstadt F. Turbulence regeneration in
pipe flow at moderate reynolds numbers. Physical Review Letters. 2005;95(21).
doi:10.1103/PhysRevLett.95.214502
apa: Hof, B., Van Doorne, C., Westerweel, J., & Nieuwstadt, F. (2005). Turbulence
regeneration in pipe flow at moderate reynolds numbers. Physical Review Letters.
American Physical Society. https://doi.org/10.1103/PhysRevLett.95.214502
chicago: Hof, Björn, Casimir Van Doorne, Jerry Westerweel, and Frans Nieuwstadt.
“Turbulence Regeneration in Pipe Flow at Moderate Reynolds Numbers.” Physical
Review Letters. American Physical Society, 2005. https://doi.org/10.1103/PhysRevLett.95.214502.
ieee: B. Hof, C. Van Doorne, J. Westerweel, and F. Nieuwstadt, “Turbulence regeneration
in pipe flow at moderate reynolds numbers,” Physical Review Letters, vol.
95, no. 21. American Physical Society, 2005.
ista: Hof B, Van Doorne C, Westerweel J, Nieuwstadt F. 2005. Turbulence regeneration
in pipe flow at moderate reynolds numbers. Physical Review Letters. 95(21).
mla: Hof, Björn, et al. “Turbulence Regeneration in Pipe Flow at Moderate Reynolds
Numbers.” Physical Review Letters, vol. 95, no. 21, American Physical Society,
2005, doi:10.1103/PhysRevLett.95.214502.
short: B. Hof, C. Van Doorne, J. Westerweel, F. Nieuwstadt, Physical Review Letters
95 (2005).
date_created: 2018-12-11T11:59:36Z
date_published: 2005-11-17T00:00:00Z
date_updated: 2021-01-12T06:59:43Z
day: '17'
doi: 10.1103/PhysRevLett.95.214502
extern: 1
intvolume: ' 95'
issue: '21'
month: '11'
publication: Physical Review Letters
publication_status: published
publisher: American Physical Society
publist_id: '4101'
quality_controlled: 0
status: public
title: Turbulence regeneration in pipe flow at moderate reynolds numbers
type: journal_article
volume: 95
year: '2005'
...
---
_id: '2790'
abstract:
- lang: eng
text: We present the results of an experimental investigation of the effect of a
magnetic field on the stability of convection in a liquid metal. A rectangular
container of gallium is subjected to a horizontal temperature gradient and a uniform
magnetic field is applied separately in three directions. The magnetic field suppresses
the oscillation most effectively when it is applied in the vertical direction
and is least efficient when applied in the direction of the temperature gradient.
The critical temperature difference required for the onset of oscillations is
found to scale exponentially with the magnitude of the magnetic field for all
three orientations. Comparisons are made with available theory and qualitative
differences are discussed.
author:
- first_name: Björn
full_name: Björn Hof
id: 3A374330-F248-11E8-B48F-1D18A9856A87
last_name: Hof
orcid: 0000-0003-2057-2754
- first_name: Anne
full_name: Juel, Anne
last_name: Juel
- first_name: Tom
full_name: Mullin, Tom P
last_name: Mullin
citation:
ama: Hof B, Juel A, Mullin T. Magnetohydrodynamic damping of oscillations in low-Prandtl-number
convection. Journal of Fluid Mechanics. 2005;545:193-201. doi:10.1017/S0022112005006762
apa: Hof, B., Juel, A., & Mullin, T. (2005). Magnetohydrodynamic damping of
oscillations in low-Prandtl-number convection. Journal of Fluid Mechanics.
Cambridge University Press. https://doi.org/10.1017/S0022112005006762
chicago: Hof, Björn, Anne Juel, and Tom Mullin. “Magnetohydrodynamic Damping of
Oscillations in Low-Prandtl-Number Convection.” Journal of Fluid Mechanics.
Cambridge University Press, 2005. https://doi.org/10.1017/S0022112005006762.
ieee: B. Hof, A. Juel, and T. Mullin, “Magnetohydrodynamic damping of oscillations
in low-Prandtl-number convection,” Journal of Fluid Mechanics, vol. 545.
Cambridge University Press, pp. 193–201, 2005.
ista: Hof B, Juel A, Mullin T. 2005. Magnetohydrodynamic damping of oscillations
in low-Prandtl-number convection. Journal of Fluid Mechanics. 545, 193–201.
mla: Hof, Björn, et al. “Magnetohydrodynamic Damping of Oscillations in Low-Prandtl-Number
Convection.” Journal of Fluid Mechanics, vol. 545, Cambridge University
Press, 2005, pp. 193–201, doi:10.1017/S0022112005006762.
short: B. Hof, A. Juel, T. Mullin, Journal of Fluid Mechanics 545 (2005) 193–201.
date_created: 2018-12-11T11:59:37Z
date_published: 2005-12-25T00:00:00Z
date_updated: 2021-01-12T06:59:44Z
day: '25'
doi: 10.1017/S0022112005006762
extern: 1
intvolume: ' 545'
month: '12'
page: 193 - 201
publication: Journal of Fluid Mechanics
publication_status: published
publisher: Cambridge University Press
publist_id: '4099'
quality_controlled: 0
status: public
title: Magnetohydrodynamic damping of oscillations in low-Prandtl-number convection
type: journal_article
volume: 545
year: '2005'
...
---
_id: '2789'
abstract:
- lang: eng
text: Transitional pipe flow is investigated in two different experimental set-ups.
In the first the stability threshold and the initial growth of localized perturbations
are studied. Good agreement is found with an earlier investigation of the transition
threshold. The measurement technique applied in the last part of this study allows
the reconstruction of the streamwise vorticity in a turbulent puff.
author:
- first_name: Björn
full_name: Björn Hof
id: 3A374330-F248-11E8-B48F-1D18A9856A87
last_name: Hof
orcid: 0000-0003-2057-2754
citation:
ama: Hof B. Transition to turbulence in pipe flow. Fluid Mechanics and its Applications.
2005;77:221-231. doi:10.1007/1-4020-4049-0_12
apa: Hof, B. (2005). Transition to turbulence in pipe flow. Fluid Mechanics and
Its Applications. Springer. https://doi.org/10.1007/1-4020-4049-0_12
chicago: Hof, Björn. “Transition to Turbulence in Pipe Flow.” Fluid Mechanics
and Its Applications. Springer, 2005. https://doi.org/10.1007/1-4020-4049-0_12.
ieee: B. Hof, “Transition to turbulence in pipe flow,” Fluid Mechanics and its
Applications, vol. 77. Springer, pp. 221–231, 2005.
ista: Hof B. 2005. Transition to turbulence in pipe flow. Fluid Mechanics and its
Applications. 77, 221–231.
mla: Hof, Björn. “Transition to Turbulence in Pipe Flow.” Fluid Mechanics and
Its Applications, vol. 77, Springer, 2005, pp. 221–31, doi:10.1007/1-4020-4049-0_12.
short: B. Hof, Fluid Mechanics and Its Applications 77 (2005) 221–231.
date_created: 2018-12-11T11:59:36Z
date_published: 2005-09-19T00:00:00Z
date_updated: 2021-01-12T06:59:43Z
day: '19'
doi: 10.1007/1-4020-4049-0_12
extern: 1
intvolume: ' 77'
month: '09'
page: 221 - 231
publication: Fluid Mechanics and its Applications
publication_status: published
publisher: Springer
publist_id: '4100'
quality_controlled: 0
status: public
title: Transition to turbulence in pipe flow
type: journal_article
volume: 77
year: '2005'
...
---
_id: '2867'
abstract:
- lang: eng
text: The plant hormone auxin elicits many specific context-dependent developmental
responses. Auxin promotes degradation of Aux/IAA proteins that prevent transcription
factors of the auxin response factor (ARF) family from regulating auxin-responsive
target genes. Aux/IAAs and ARFs are represented by large gene families in Arabidopsis.
Here we show that stabilization of BDL/IAA12 or its sister protein IAA13 prevents
MP/ARF5-dependent embryonic root formation whereas stabilized SHY2/IAA3 interferes
with seedling growth. Although both bdl and shy2-2 proteins inhibited MP/ARF5-dependent
reporter gene activation, shy2-2 was much less efficient than bdl to interfere
with embryonic root initiation when expressed from the BDL promoter. Similarly,
MP was much more efficient than ARF16 in this process. When expressed from the
SHY2 promoter, both shy2-2 and bdl inhibited cell elongation and auxin-induced
gene expression in the seedling hypocotyl. By contrast, gravitropism and auxin-induced
gene expression in the root, which were promoted by functionally redundant NPH4/ARF7
and ARF19 proteins, were inhibited by shy2-2, but not by bdl protein. Our results
suggest that auxin signals are converted into specific responses by matching pairs
of coexpressed ARF and Aux/IAA proteins.
author:
- first_name: Dolf
full_name: Weijers, Dolf
last_name: Weijers
- first_name: Eva
full_name: Eva Benková
id: 38F4F166-F248-11E8-B48F-1D18A9856A87
last_name: Benková
orcid: 0000-0002-8510-9739
- first_name: Katja
full_name: Jäger, Katja E
last_name: Jäger
- first_name: Alexandra
full_name: Schlereth, Alexandra
last_name: Schlereth
- first_name: Thorsten
full_name: Hamann, Thorsten
last_name: Hamann
- first_name: Marika
full_name: Kientz, Marika
last_name: Kientz
- first_name: Jill
full_name: Wilmoth, Jill C
last_name: Wilmoth
- first_name: Jason
full_name: Reed, Jason W
last_name: Reed
- first_name: Gerd
full_name: Jürgens, Gerd
last_name: Jürgens
citation:
ama: Weijers D, Benková E, Jäger K, et al. Developmental specificity of auxin response
by pairs of ARF and Aux/IAA transcriptional regulators. EMBO Journal. 2005;24(10):1874-1885.
doi:10.1038/sj.emboj.7600659
apa: Weijers, D., Benková, E., Jäger, K., Schlereth, A., Hamann, T., Kientz, M.,
… Jürgens, G. (2005). Developmental specificity of auxin response by pairs of
ARF and Aux/IAA transcriptional regulators. EMBO Journal. Wiley-Blackwell.
https://doi.org/10.1038/sj.emboj.7600659
chicago: Weijers, Dolf, Eva Benková, Katja Jäger, Alexandra Schlereth, Thorsten
Hamann, Marika Kientz, Jill Wilmoth, Jason Reed, and Gerd Jürgens. “Developmental
Specificity of Auxin Response by Pairs of ARF and Aux/IAA Transcriptional Regulators.”
EMBO Journal. Wiley-Blackwell, 2005. https://doi.org/10.1038/sj.emboj.7600659.
ieee: D. Weijers et al., “Developmental specificity of auxin response by
pairs of ARF and Aux/IAA transcriptional regulators,” EMBO Journal, vol.
24, no. 10. Wiley-Blackwell, pp. 1874–1885, 2005.
ista: Weijers D, Benková E, Jäger K, Schlereth A, Hamann T, Kientz M, Wilmoth J,
Reed J, Jürgens G. 2005. Developmental specificity of auxin response by pairs
of ARF and Aux/IAA transcriptional regulators. EMBO Journal. 24(10), 1874–1885.
mla: Weijers, Dolf, et al. “Developmental Specificity of Auxin Response by Pairs
of ARF and Aux/IAA Transcriptional Regulators.” EMBO Journal, vol. 24,
no. 10, Wiley-Blackwell, 2005, pp. 1874–85, doi:10.1038/sj.emboj.7600659.
short: D. Weijers, E. Benková, K. Jäger, A. Schlereth, T. Hamann, M. Kientz, J.
Wilmoth, J. Reed, G. Jürgens, EMBO Journal 24 (2005) 1874–1885.
date_created: 2018-12-11T12:00:01Z
date_published: 2005-05-18T00:00:00Z
date_updated: 2021-01-12T07:00:22Z
day: '18'
doi: 10.1038/sj.emboj.7600659
extern: 1
intvolume: ' 24'
issue: '10'
main_file_link:
- open_access: '1'
url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1142592/
month: '05'
oa: 1
page: 1874 - 1885
publication: EMBO Journal
publication_status: published
publisher: Wiley-Blackwell
publist_id: '3918'
quality_controlled: 0
status: public
title: Developmental specificity of auxin response by pairs of ARF and Aux/IAA transcriptional
regulators
type: journal_article
volume: 24
year: '2005'
...
---
_id: '2895'
abstract:
- lang: eng
text: One of the fundamental properties of the immune system is its capacity to
avoid autoimmune diseases. The mechanism underlying this process, known as self-tolerance,
is hitherto unresolved but seems to involve the control of clonal expansion of
autoreactive lymphocytes. This article reviews mathematical modeling of self-tolerance,
addressing two specific hypotheses. The first hypothesis posits that self-tolerance
is mediated by tuning of activation thresholds, which makes autoreactive T lymphocytes
reversibly "anergic" and unable to proliferate. The second hypothesis
posits that the proliferation of autoreactive T lymphocytes is instead controlled
by specific regulatory T lymphocytes. Models representing the population dynamics
of autoreactive T lymphocytes according to these two hypotheses were derived.
For each model we identified how cell density affects tolerance, and predicted
the corresponding phase spaces and bifurcations. We show that the simple induction
of proliferative anergy, as modeled here, has a density dependence that is only
partially compatible with adoptive transfers of tolerance, and that the models
of tolerance mediated by specific regulatory T cells are closer to the observations.
acknowledgement: 'The work was financially supported by Fundação para a Ciência e
Tecnologia: grants P/BIA/10094/1998, POCTI/36413/99, and POCTI/MGI/46477/2002;
and fellowships to JF (Praxis/BCC/18972/98), JS (BD/13546/97), KL (SFRH/BPD+/1157/2002),
DM (SFRH/BD/2960/2000) and TP (SFRH/BD/10550/2002).'
author:
- first_name: Jorge
full_name: Carneiro, Jorge
last_name: Carneiro
- first_name: Tiago
full_name: Tiago Paixao
id: 2C5658E6-F248-11E8-B48F-1D18A9856A87
last_name: Paixao
orcid: 0000-0003-2361-3953
- first_name: Dejan
full_name: Milutinovic, Dejan
last_name: Milutinovic
- first_name: João
full_name: Sousa, João
last_name: Sousa
- first_name: Kalet
full_name: Leon, Kalet
last_name: Leon
- first_name: Rui
full_name: Gardner, Rui
last_name: Gardner
- first_name: Jose
full_name: Faro, Jose
last_name: Faro
citation:
ama: 'Carneiro J, Paixao T, Milutinovic D, et al. Immunological self tolerance:
Lessons from mathematical modeling. Journal of Computational and Applied Mathematics.
2005;184(1):77-100. doi:10.1016/j.cam.2004.10.025'
apa: 'Carneiro, J., Paixao, T., Milutinovic, D., Sousa, J., Leon, K., Gardner, R.,
& Faro, J. (2005). Immunological self tolerance: Lessons from mathematical
modeling. Journal of Computational and Applied Mathematics. Elsevier. https://doi.org/10.1016/j.cam.2004.10.025'
chicago: 'Carneiro, Jorge, Tiago Paixao, Dejan Milutinovic, João Sousa, Kalet Leon,
Rui Gardner, and Jose Faro. “Immunological Self Tolerance: Lessons from Mathematical
Modeling.” Journal of Computational and Applied Mathematics. Elsevier,
2005. https://doi.org/10.1016/j.cam.2004.10.025.'
ieee: 'J. Carneiro et al., “Immunological self tolerance: Lessons from mathematical
modeling,” Journal of Computational and Applied Mathematics, vol. 184,
no. 1. Elsevier, pp. 77–100, 2005.'
ista: 'Carneiro J, Paixao T, Milutinovic D, Sousa J, Leon K, Gardner R, Faro J.
2005. Immunological self tolerance: Lessons from mathematical modeling. Journal
of Computational and Applied Mathematics. 184(1), 77–100.'
mla: 'Carneiro, Jorge, et al. “Immunological Self Tolerance: Lessons from Mathematical
Modeling.” Journal of Computational and Applied Mathematics, vol. 184,
no. 1, Elsevier, 2005, pp. 77–100, doi:10.1016/j.cam.2004.10.025.'
short: J. Carneiro, T. Paixao, D. Milutinovic, J. Sousa, K. Leon, R. Gardner, J.
Faro, Journal of Computational and Applied Mathematics 184 (2005) 77–100.
date_created: 2018-12-11T12:00:12Z
date_published: 2005-12-01T00:00:00Z
date_updated: 2021-01-12T07:00:32Z
day: '01'
doi: 10.1016/j.cam.2004.10.025
extern: 1
intvolume: ' 184'
issue: '1'
month: '12'
page: 77 - 100
publication: Journal of Computational and Applied Mathematics
publication_status: published
publisher: Elsevier
publist_id: '3863'
quality_controlled: 0
status: public
title: 'Immunological self tolerance: Lessons from mathematical modeling'
type: journal_article
volume: 184
year: '2005'
...
---
_id: '3004'
abstract:
- lang: eng
text: Molecular mechanisms of pattern formation in the plant embryo are not well
understood. Recent molecular and cellular studies, in conjunction with earlier
microsurgical, physiological, and genetic work, are now starting to define the
outlines of a model where gradients of the signaling molecule auxin play a central
role in embryo patterning. It is relatively clear how these gradients are established
and interpreted, but how they are maintained is still unresolved. Here, we have
studied the contributions of auxin biosynthesis, conjugation, and transport pathways
to the maintenance of embryonic auxin gradients. Auxin homeostasis in the embryo
was manipulated by region-specific conditional expression of indoleacetic acid-tryptophan
monooxygenase or indoleacetic acid-lysine synthetase, bacterial enzymes for auxin
biosynthesis or conjugation. Neither manipulation of auxin biosynthesis nor of
auxin conjugation interfered with auxin gradients and patterning in the embryo.
This result suggests a compensatory mechanism for buffering auxin gradients in
the embryo. Chemical and genetic inhibition revealed that auxin transport activity,
in particular that of the PIN-FORMED1 (PIN1) and PIN4 proteins, is a major factor
in the maintenance of these gradients.
author:
- first_name: Dolf
full_name: Weijers, Dolf
last_name: Weijers
- first_name: Michael
full_name: Sauer, Michael
last_name: Sauer
- first_name: Olivier
full_name: Meurette, Olivier
last_name: Meurette
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
- first_name: Karin
full_name: Ljung, Karin
last_name: Ljung
- first_name: Göran
full_name: Sandberg, Göran
last_name: Sandberg
- first_name: Paul
full_name: Hooykaas, Paul
last_name: Hooykaas
- first_name: Remko
full_name: Offringa, Remko
last_name: Offringa
citation:
ama: Weijers D, Sauer M, Meurette O, et al. Maintenance of embryonic auxin distribution
for apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis.
Plant Cell. 2005;17(9):2517-2526. doi:10.1105/tpc.105.034637
apa: Weijers, D., Sauer, M., Meurette, O., Friml, J., Ljung, K., Sandberg, G., …
Offringa, R. (2005). Maintenance of embryonic auxin distribution for apical basal
patterning by PIN FORMED dependent auxin transport in Arabidopsis. Plant Cell.
American Society of Plant Biologists. https://doi.org/10.1105/tpc.105.034637
chicago: Weijers, Dolf, Michael Sauer, Olivier Meurette, Jiří Friml, Karin Ljung,
Göran Sandberg, Paul Hooykaas, and Remko Offringa. “Maintenance of Embryonic Auxin
Distribution for Apical Basal Patterning by PIN FORMED Dependent Auxin Transport
in Arabidopsis.” Plant Cell. American Society of Plant Biologists, 2005.
https://doi.org/10.1105/tpc.105.034637.
ieee: D. Weijers et al., “Maintenance of embryonic auxin distribution for
apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis,”
Plant Cell, vol. 17, no. 9. American Society of Plant Biologists, pp. 2517–2526,
2005.
ista: Weijers D, Sauer M, Meurette O, Friml J, Ljung K, Sandberg G, Hooykaas P,
Offringa R. 2005. Maintenance of embryonic auxin distribution for apical basal
patterning by PIN FORMED dependent auxin transport in Arabidopsis. Plant Cell.
17(9), 2517–2526.
mla: Weijers, Dolf, et al. “Maintenance of Embryonic Auxin Distribution for Apical
Basal Patterning by PIN FORMED Dependent Auxin Transport in Arabidopsis.” Plant
Cell, vol. 17, no. 9, American Society of Plant Biologists, 2005, pp. 2517–26,
doi:10.1105/tpc.105.034637.
short: D. Weijers, M. Sauer, O. Meurette, J. Friml, K. Ljung, G. Sandberg, P. Hooykaas,
R. Offringa, Plant Cell 17 (2005) 2517–2526.
date_created: 2018-12-11T12:00:48Z
date_published: 2005-07-01T00:00:00Z
date_updated: 2021-01-12T07:40:24Z
day: '01'
doi: 10.1105/tpc.105.034637
extern: 1
intvolume: ' 17'
issue: '9'
month: '07'
page: 2517 - 2526
publication: Plant Cell
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '3698'
quality_controlled: 0
status: public
title: Maintenance of embryonic auxin distribution for apical basal patterning by
PIN FORMED dependent auxin transport in Arabidopsis
type: journal_article
volume: 17
year: '2005'
...
---
_id: '3000'
abstract:
- lang: eng
text: In plants, cell polarity is an issue more recurring than in other systems,
because plants, due to their adaptive and flexible development, often change cell
polarity postembryonically according to intrinsic cues and demands of the environment.
Recent findings on the directional movement of the plant signalling molecule auxin
provide a unique connection between individual cell polarity and the establishment
of polarity at the tissue, organ, and whole-plant levels. Decisions about the
subcellular polar targeting of PIN auxin transport components determine the direction
of auxin flow between cells and consequently mediate multiple developmental events.
In addition, mutations or chemical interference with PIN-based auxin transport
result in abnormal cell divisions. Thus, the complicated links between cell polarity
establishment, auxin transport, cytoskeleton, and oriented cell divisions now
begin to emerge. Here we review the available literature on the issues of cell
polarity in both plants and animals to extend our understanding on the generation,
maintenance, and transmission of cell polarity in plants.
author:
- first_name: Pankaj
full_name: Dhonukshe, Pankaj
last_name: Dhonukshe
- first_name: Jürgen
full_name: Kleine Vehn, Jürgen
last_name: Kleine Vehn
- first_name: Jirí
full_name: Friml, Jirí
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
citation:
ama: 'Dhonukshe P, Kleine Vehn J, Friml J. Cell polarity, auxin transport and cytoskeleton
mediated division planes: Who comes first? Protoplasma. 2005;226(1-2):67-73.
doi:10.1007/s00709-005-0104-8'
apa: 'Dhonukshe, P., Kleine Vehn, J., & Friml, J. (2005). Cell polarity, auxin
transport and cytoskeleton mediated division planes: Who comes first? Protoplasma.
Springer. https://doi.org/10.1007/s00709-005-0104-8'
chicago: 'Dhonukshe, Pankaj, Jürgen Kleine Vehn, and Jiří Friml. “Cell Polarity,
Auxin Transport and Cytoskeleton Mediated Division Planes: Who Comes First?” Protoplasma.
Springer, 2005. https://doi.org/10.1007/s00709-005-0104-8.'
ieee: 'P. Dhonukshe, J. Kleine Vehn, and J. Friml, “Cell polarity, auxin transport
and cytoskeleton mediated division planes: Who comes first?,” Protoplasma,
vol. 226, no. 1–2. Springer, pp. 67–73, 2005.'
ista: 'Dhonukshe P, Kleine Vehn J, Friml J. 2005. Cell polarity, auxin transport
and cytoskeleton mediated division planes: Who comes first? Protoplasma. 226(1–2),
67–73.'
mla: 'Dhonukshe, Pankaj, et al. “Cell Polarity, Auxin Transport and Cytoskeleton
Mediated Division Planes: Who Comes First?” Protoplasma, vol. 226, no.
1–2, Springer, 2005, pp. 67–73, doi:10.1007/s00709-005-0104-8.'
short: P. Dhonukshe, J. Kleine Vehn, J. Friml, Protoplasma 226 (2005) 67–73.
date_created: 2018-12-11T12:00:47Z
date_published: 2005-10-01T00:00:00Z
date_updated: 2021-01-12T07:40:22Z
day: '01'
doi: 10.1007/s00709-005-0104-8
extern: '1'
intvolume: ' 226'
issue: 1-2
language:
- iso: eng
month: '10'
oa_version: None
page: 67 - 73
publication: Protoplasma
publication_status: published
publisher: Springer
publist_id: '3701'
quality_controlled: '1'
status: public
title: 'Cell polarity, auxin transport and cytoskeleton mediated division planes:
Who comes first?'
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 226
year: '2005'
...
---
_id: '3001'
abstract:
- lang: eng
text: 'One of the mechanisms by which signalling molecules regulate cellular behaviour
is modulating subcellular protein translocation. This mode of regulation is often
based on specialized vesicle trafficking, termed constitutive cycling, which consists
of repeated internalization and recycling of proteins to and from the plasma membrane.
No such mechanism of hormone action has been shown in plants although several
proteins, including the PIN auxin efflux facilitators, exhibit constitutive cycling.
Here we show that a major regulator of plant development, auxin, inhibits endocytosis.
This effect is specific to biologically active auxins and requires activity of
the Calossin-like protein BIG. By inhibiting the internalization step of PIN constitutive
cycling, auxin increases levels of PINs at the plasma membrane. Concomitantly,
auxin promotes its own efflux from cells by a vesicle-trafficking-dependent mechanism.
Furthermore, asymmetric auxin translocation during gravitropism is correlated
with decreased PIN internalization. Our data imply a previously undescribed mode
of plant hormone action: by modulating PIN protein trafficking, auxin regulates
PIN abundance and activity at the cell surface, providing a mechanism for the
feedback regulation of auxin transport.'
author:
- first_name: Tomasz
full_name: Paciorek, Tomasz
last_name: Paciorek
- first_name: Eva
full_name: Zažímalová, Eva
last_name: Zažímalová
- first_name: Nadia
full_name: Ruthardt, Nadia
last_name: Ruthardt
- first_name: Jan
full_name: Petrášek, Jan
last_name: Petrášek
- first_name: York
full_name: Stierhof, York-Dieter
last_name: Stierhof
- first_name: Jürgen
full_name: Kleine-Vehn, Jürgen
last_name: Kleine Vehn
- first_name: David
full_name: Morris, David A
last_name: Morris
- first_name: Neil
full_name: Emans, Neil
last_name: Emans
- first_name: Gerd
full_name: Jürgens, Gerd
last_name: Jürgens
- first_name: Niko
full_name: Geldner, Niko
last_name: Geldner
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
citation:
ama: Paciorek T, Zažímalová E, Ruthardt N, et al. Auxin inhibits endocytosis and
promotes its own efflux from cells. Nature. 2005;435(7046):1251-1256. doi:10.1038/nature03633
apa: Paciorek, T., Zažímalová, E., Ruthardt, N., Petrášek, J., Stierhof, Y., Kleine
Vehn, J., … Friml, J. (2005). Auxin inhibits endocytosis and promotes its own
efflux from cells. Nature. Nature Publishing Group. https://doi.org/10.1038/nature03633
chicago: Paciorek, Tomasz, Eva Zažímalová, Nadia Ruthardt, Jan Petrášek, York Stierhof,
Jürgen Kleine Vehn, David Morris, et al. “Auxin Inhibits Endocytosis and Promotes
Its Own Efflux from Cells.” Nature. Nature Publishing Group, 2005. https://doi.org/10.1038/nature03633.
ieee: T. Paciorek et al., “Auxin inhibits endocytosis and promotes its own
efflux from cells,” Nature, vol. 435, no. 7046. Nature Publishing Group,
pp. 1251–1256, 2005.
ista: Paciorek T, Zažímalová E, Ruthardt N, Petrášek J, Stierhof Y, Kleine Vehn
J, Morris D, Emans N, Jürgens G, Geldner N, Friml J. 2005. Auxin inhibits endocytosis
and promotes its own efflux from cells. Nature. 435(7046), 1251–1256.
mla: Paciorek, Tomasz, et al. “Auxin Inhibits Endocytosis and Promotes Its Own Efflux
from Cells.” Nature, vol. 435, no. 7046, Nature Publishing Group, 2005,
pp. 1251–56, doi:10.1038/nature03633.
short: T. Paciorek, E. Zažímalová, N. Ruthardt, J. Petrášek, Y. Stierhof, J. Kleine
Vehn, D. Morris, N. Emans, G. Jürgens, N. Geldner, J. Friml, Nature 435 (2005)
1251–1256.
date_created: 2018-12-11T12:00:47Z
date_published: 2005-06-30T00:00:00Z
date_updated: 2021-01-12T07:40:23Z
day: '30'
doi: 10.1038/nature03633
extern: 1
intvolume: ' 435'
issue: '7046'
month: '06'
page: 1251 - 1256
publication: Nature
publication_status: published
publisher: Nature Publishing Group
publist_id: '3702'
quality_controlled: 0
status: public
title: Auxin inhibits endocytosis and promotes its own efflux from cells
type: journal_article
volume: 435
year: '2005'
...
---
_id: '3003'
abstract:
- lang: eng
text: 'Plant development displays an exceptional plasticity and adaptability that
involves the dynamic, asymmetric distribution of the phytohormone auxin. Polar
auxin flow, which requires polarly localized transport facilitators of the PIN
family, largely contributes to the establishment and maintenance of the auxin
gradients. Functionally overlapping action of PIN proteins mediates multiple developmental
processes, including embryo formation, organ development and tropisms. Here we
show that PIN proteins exhibit synergistic interactions, which involve cross-regulation
of PIN gene expression in pin mutants or plants with inhibited auxin transport.
Auxin itself positively feeds back on PIN gene expression in a tissue-specific
manner through an AUX/IAA-dependent signalling pathway. This regulatory switch
is indicative of a mechanism by which the loss of a specific PIN protein is compensated
for by auxin-dependent ectopic: expression of its homologues. The compensatory
properties of the PIN-dependent transport network might enable the stabilization
of auxin gradients and potentially contribute to the robustness of plant adaptive
development.'
author:
- first_name: Anne
full_name: Vieten, Anne
last_name: Vieten
- first_name: Steffen
full_name: Vanneste, Steffen
last_name: Vanneste
- first_name: Justyna
full_name: Wiśniewska, Justyna
last_name: Wiśniewska
- first_name: Eva
full_name: Eva Benková
id: 38F4F166-F248-11E8-B48F-1D18A9856A87
last_name: Benková
orcid: 0000-0002-8510-9739
- first_name: René
full_name: Benjamins, René
last_name: Benjamins
- first_name: Tom
full_name: Beeckman, Tom
last_name: Beeckman
- first_name: Christian
full_name: Luschnig, Christian
last_name: Luschnig
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
citation:
ama: Vieten A, Vanneste S, Wiśniewska J, et al. Functional redundancy of PIN proteins
is accompanied by auxin-dependent cross-regulation of PIN expression. Development.
2005;132(20):4521-4531. doi:10.1242/dev.02027
apa: Vieten, A., Vanneste, S., Wiśniewska, J., Benková, E., Benjamins, R., Beeckman,
T., … Friml, J. (2005). Functional redundancy of PIN proteins is accompanied by
auxin-dependent cross-regulation of PIN expression. Development. Company
of Biologists. https://doi.org/10.1242/dev.02027
chicago: Vieten, Anne, Steffen Vanneste, Justyna Wiśniewska, Eva Benková, René Benjamins,
Tom Beeckman, Christian Luschnig, and Jiří Friml. “Functional Redundancy of PIN
Proteins Is Accompanied by Auxin-Dependent Cross-Regulation of PIN Expression.”
Development. Company of Biologists, 2005. https://doi.org/10.1242/dev.02027.
ieee: A. Vieten et al., “Functional redundancy of PIN proteins is accompanied
by auxin-dependent cross-regulation of PIN expression,” Development, vol.
132, no. 20. Company of Biologists, pp. 4521–4531, 2005.
ista: Vieten A, Vanneste S, Wiśniewska J, Benková E, Benjamins R, Beeckman T, Luschnig
C, Friml J. 2005. Functional redundancy of PIN proteins is accompanied by auxin-dependent
cross-regulation of PIN expression. Development. 132(20), 4521–4531.
mla: Vieten, Anne, et al. “Functional Redundancy of PIN Proteins Is Accompanied
by Auxin-Dependent Cross-Regulation of PIN Expression.” Development, vol.
132, no. 20, Company of Biologists, 2005, pp. 4521–31, doi:10.1242/dev.02027.
short: A. Vieten, S. Vanneste, J. Wiśniewska, E. Benková, R. Benjamins, T. Beeckman,
C. Luschnig, J. Friml, Development 132 (2005) 4521–4531.
date_created: 2018-12-11T12:00:48Z
date_published: 2005-10-01T00:00:00Z
date_updated: 2021-01-12T07:40:23Z
day: '01'
doi: 10.1242/dev.02027
extern: 1
intvolume: ' 132'
issue: '20'
month: '10'
page: 4521 - 4531
publication: Development
publication_status: published
publisher: Company of Biologists
publist_id: '3700'
quality_controlled: 0
status: public
title: Functional redundancy of PIN proteins is accompanied by auxin-dependent cross-regulation
of PIN expression
type: journal_article
volume: 132
year: '2005'
...
---
_id: '3212'
abstract:
- lang: eng
text: |-
The Full-Domain Hash (FDH) signature scheme [3] forms one the most basic usages of random oracles. It works with a family F of trapdoor permutations (TDP), where the signature of m is computed as f−1(h(m)) (here f ∈R F and h is modelled as a random oracle). It is known to be existentially unforgeable for any TDP family F [3], although a much tighter security reduction is known for a restrictive class of TDP’s [10,14] — namely, those induced by a family of claw-free permutations (CFP) pairs. The latter result was shown [11] to match the best possible “black-box” security reduction in the random oracle model, irrespective of the TDP family F (e.g., RSA) one might use.
In this work we investigate the question if it is possible to instantiate the random oracle h with a “real” family of hash functions H such that the corresponding schemes can be proven secure in the standard model, under some natural assumption on the family F. Our main result rules out the existence of such instantiations for any assumption on F which (1) is satisfied by a family of random permutations; and (2) does not allow the attacker to invert f ∈R F on an a-priori unbounded number of points. Moreover, this holds even if the choice of H can arbitrarily depend on f. As an immediate corollary, we rule out instantiating FDH based on general claw-free permutations, which shows that in order to prove the security of FDH in the standard model one must utilize significantly more structure on F than what is sufficient for the best proof of security in the random oracle model.
acknowledgement: Supported by NSF CAREER Award CCR-0133806 and TC Grant No.CCR-0311095.
Supported by the Swiss National Science Foundation, project No. 200020-103847/1
alternative_title:
- LNCS
author:
- first_name: Yevgeniy
full_name: Dodis, Yevgeniy
last_name: Dodis
- first_name: Roberto
full_name: Oliveira, Roberto
last_name: Oliveira
- first_name: Krzysztof Z
full_name: Krzysztof Pietrzak
id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87
last_name: Pietrzak
orcid: 0000-0002-9139-1654
citation:
ama: 'Dodis Y, Oliveira R, Pietrzak KZ. On the generic insecurity of the full domain
hash. In: Vol 3621. Springer; 2005:449-466. doi:10.1007/11535218_27'
apa: 'Dodis, Y., Oliveira, R., & Pietrzak, K. Z. (2005). On the generic insecurity
of the full domain hash (Vol. 3621, pp. 449–466). Presented at the CRYPTO: International
Cryptology Conference, Springer. https://doi.org/10.1007/11535218_27'
chicago: Dodis, Yevgeniy, Roberto Oliveira, and Krzysztof Z Pietrzak. “On the Generic
Insecurity of the Full Domain Hash,” 3621:449–66. Springer, 2005. https://doi.org/10.1007/11535218_27.
ieee: 'Y. Dodis, R. Oliveira, and K. Z. Pietrzak, “On the generic insecurity of
the full domain hash,” presented at the CRYPTO: International Cryptology Conference,
2005, vol. 3621, pp. 449–466.'
ista: 'Dodis Y, Oliveira R, Pietrzak KZ. 2005. On the generic insecurity of the
full domain hash. CRYPTO: International Cryptology Conference, LNCS, vol. 3621,
449–466.'
mla: Dodis, Yevgeniy, et al. On the Generic Insecurity of the Full Domain Hash.
Vol. 3621, Springer, 2005, pp. 449–66, doi:10.1007/11535218_27.
short: Y. Dodis, R. Oliveira, K.Z. Pietrzak, in:, Springer, 2005, pp. 449–466.
conference:
name: 'CRYPTO: International Cryptology Conference'
date_created: 2018-12-11T12:02:03Z
date_published: 2005-09-12T00:00:00Z
date_updated: 2021-01-12T07:41:50Z
day: '12'
doi: 10.1007/11535218_27
extern: 1
intvolume: ' 3621'
month: '09'
page: 449 - 466
publication_status: published
publisher: Springer
publist_id: '3470'
quality_controlled: 0
status: public
title: On the generic insecurity of the full domain hash
type: conference
volume: 3621
year: '2005'
...
---
_id: '3213'
abstract:
- lang: eng
text: |-
We study the question whether the sequential or parallel composition of two functions, each indistinguishable from a random function by non-adaptive distinguishers is secure against adaptive distinguishers. The sequential composition of F and G is the function G(F()), the parallel composition is F G where ⋆ is some group operation. It has been shown that composition indeed gives adaptive security in the information theoretic setting, but unfortunately the proof does not translate into the more interesting computational case.
In this work we show that in the computational setting composition does not imply adaptive security: If there is a prime order cyclic group where the decisional Diffie-Hellman assumption holds, then there are functions F and G which are indistinguishable by non-adaptive polynomially time-bounded adversaries, but whose parallel composition can be completely broken (i.e. we recover the key) with only three adaptive queries. We give a similar result for sequential composition. Interestingly, we need a standard assumption from the asymmetric (aka. public-key) world to prove a negative result for symmetric (aka. private-key) systems.
acknowledgement: Supported by the Swiss National Science Foundation, project No. 200020-103847/1.
alternative_title:
- LNCS
author:
- first_name: Krzysztof Z
full_name: Krzysztof Pietrzak
id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87
last_name: Pietrzak
orcid: 0000-0002-9139-1654
citation:
ama: 'Pietrzak KZ. Composition does not imply adaptive security. In: Vol 3621. Springer;
2005:55-65. doi:10.1007/11535218_4'
apa: 'Pietrzak, K. Z. (2005). Composition does not imply adaptive security (Vol.
3621, pp. 55–65). Presented at the CRYPTO: International Cryptology Conference,
Springer. https://doi.org/10.1007/11535218_4'
chicago: Pietrzak, Krzysztof Z. “Composition Does Not Imply Adaptive Security,”
3621:55–65. Springer, 2005. https://doi.org/10.1007/11535218_4.
ieee: 'K. Z. Pietrzak, “Composition does not imply adaptive security,” presented
at the CRYPTO: International Cryptology Conference, 2005, vol. 3621, pp. 55–65.'
ista: 'Pietrzak KZ. 2005. Composition does not imply adaptive security. CRYPTO:
International Cryptology Conference, LNCS, vol. 3621, 55–65.'
mla: Pietrzak, Krzysztof Z. Composition Does Not Imply Adaptive Security.
Vol. 3621, Springer, 2005, pp. 55–65, doi:10.1007/11535218_4.
short: K.Z. Pietrzak, in:, Springer, 2005, pp. 55–65.
conference:
name: 'CRYPTO: International Cryptology Conference'
date_created: 2018-12-11T12:02:03Z
date_published: 2005-09-12T00:00:00Z
date_updated: 2021-01-12T07:41:50Z
day: '12'
doi: 10.1007/11535218_4
extern: 1
intvolume: ' 3621'
month: '09'
page: 55 - 65
publication_status: published
publisher: Springer
publist_id: '3468'
quality_controlled: 0
status: public
title: Composition does not imply adaptive security
type: conference
volume: 3621
year: '2005'
...
---
_id: '3211'
abstract:
- lang: eng
text: We present an improved bound on the advantage of any q-query adversary at
distinguishing between the CBC MAC over a random n-bit permutation and a random
function outputting n bits. The result assumes that no message queried is a prefix
of any other, as is the case when all messages to be MACed have the same length.
We go on to give an improved analysis of the encrypted CBC MAC, where there is
no restriction on queried messages. Letting m be the block length of the longest
query, our bounds are about mq2/2n for the basic CBC MAC and mo(1)q2/2n for the
encrypted CBC MAC, improving prior bounds of m2q2/2n. The new bounds translate
into improved guarantees on the probability of forging these MACs.
acknowledgement: Pietrzak was supported by the Swiss National Science Foundation,
project No. 200020-103847/1.
alternative_title:
- LNCS
author:
- first_name: Mihir
full_name: Bellare, Mihir
last_name: Bellare
- first_name: Krzysztof Z
full_name: Krzysztof Pietrzak
id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87
last_name: Pietrzak
orcid: 0000-0002-9139-1654
- first_name: Phillip
full_name: Rogaway, Phillip
last_name: Rogaway
citation:
ama: 'Bellare M, Pietrzak KZ, Rogaway P. Improved security analyses for CBC MACs.
In: Vol 3621. Springer; 2005:527-545. doi:10.1007/11535218_32'
apa: 'Bellare, M., Pietrzak, K. Z., & Rogaway, P. (2005). Improved security
analyses for CBC MACs (Vol. 3621, pp. 527–545). Presented at the CRYPTO: International
Cryptology Conference, Springer. https://doi.org/10.1007/11535218_32'
chicago: Bellare, Mihir, Krzysztof Z Pietrzak, and Phillip Rogaway. “Improved Security
Analyses for CBC MACs,” 3621:527–45. Springer, 2005. https://doi.org/10.1007/11535218_32.
ieee: 'M. Bellare, K. Z. Pietrzak, and P. Rogaway, “Improved security analyses for
CBC MACs,” presented at the CRYPTO: International Cryptology Conference, 2005,
vol. 3621, pp. 527–545.'
ista: 'Bellare M, Pietrzak KZ, Rogaway P. 2005. Improved security analyses for CBC
MACs. CRYPTO: International Cryptology Conference, LNCS, vol. 3621, 527–545.'
mla: Bellare, Mihir, et al. Improved Security Analyses for CBC MACs. Vol.
3621, Springer, 2005, pp. 527–45, doi:10.1007/11535218_32.
short: M. Bellare, K.Z. Pietrzak, P. Rogaway, in:, Springer, 2005, pp. 527–545.
conference:
name: 'CRYPTO: International Cryptology Conference'
date_created: 2018-12-11T12:02:02Z
date_published: 2005-09-12T00:00:00Z
date_updated: 2021-01-12T07:41:50Z
day: '12'
doi: 10.1007/11535218_32
extern: 1
intvolume: ' 3621'
month: '09'
page: 527 - 545
publication_status: published
publisher: Springer
publist_id: '3469'
quality_controlled: 0
status: public
title: Improved security analyses for CBC MACs
type: conference
volume: 3621
year: '2005'
...
---
_id: '3426'
abstract:
- lang: eng
text: We discuss the formation of graded morphogen profiles in a cell layer by nonlinear
transport phenomena, important for patterning developing organisms. We focus on
a process termed transcytosis, where morphogen transport results from the binding
of ligands to receptors on the cell surface, incorporation into the cell, and
subsequent externalization. Starting from a microscopic model, we derive effective
transport equations. We show that, in contrast to morphogen transport by extracellular
diffusion, transcytosis leads to robust ligand profiles which are insensitive
to the rate of ligand production.
article_processing_charge: No
author:
- first_name: Mark Tobias
full_name: Bollenbach, Mark Tobias
id: 3E6DB97A-F248-11E8-B48F-1D18A9856A87
last_name: Bollenbach
orcid: 0000-0003-4398-476X
- first_name: Karsten
full_name: Kruse, Karsten
last_name: Kruse
- first_name: Periklis
full_name: Pantazis, Periklis
last_name: Pantazis
- first_name: Marcos
full_name: González Gaitán, Marcos
last_name: González Gaitán
- first_name: Frank
full_name: Jülicher, Frank
last_name: Jülicher
citation:
ama: Bollenbach MT, Kruse K, Pantazis P, González Gaitán M, Jülicher F. Robust formation
of morphogen gradients. Physical Review Letters. 2005;94(1). doi:10.1103/PhysRevLett.94.018103
apa: Bollenbach, M. T., Kruse, K., Pantazis, P., González Gaitán, M., & Jülicher,
F. (2005). Robust formation of morphogen gradients. Physical Review Letters.
American Physical Society. https://doi.org/10.1103/PhysRevLett.94.018103
chicago: Bollenbach, Mark Tobias, Karsten Kruse, Periklis Pantazis, Marcos González
Gaitán, and Frank Jülicher. “Robust Formation of Morphogen Gradients.” Physical
Review Letters. American Physical Society, 2005. https://doi.org/10.1103/PhysRevLett.94.018103.
ieee: M. T. Bollenbach, K. Kruse, P. Pantazis, M. González Gaitán, and F. Jülicher,
“Robust formation of morphogen gradients,” Physical Review Letters, vol.
94, no. 1. American Physical Society, 2005.
ista: Bollenbach MT, Kruse K, Pantazis P, González Gaitán M, Jülicher F. 2005. Robust
formation of morphogen gradients. Physical Review Letters. 94(1).
mla: Bollenbach, Mark Tobias, et al. “Robust Formation of Morphogen Gradients.”
Physical Review Letters, vol. 94, no. 1, American Physical Society, 2005,
doi:10.1103/PhysRevLett.94.018103.
short: M.T. Bollenbach, K. Kruse, P. Pantazis, M. González Gaitán, F. Jülicher,
Physical Review Letters 94 (2005).
date_created: 2018-12-11T12:03:16Z
date_published: 2005-01-01T00:00:00Z
date_updated: 2021-01-12T07:43:23Z
day: '01'
doi: 10.1103/PhysRevLett.94.018103
extern: '1'
external_id:
arxiv:
- q-bio/0412014
intvolume: ' 94'
issue: '1'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://arxiv.org/abs/q-bio/0412014
month: '01'
oa: 1
oa_version: Preprint
publication: Physical Review Letters
publication_status: published
publisher: American Physical Society
publist_id: '2975'
status: public
title: Robust formation of morphogen gradients
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 94
year: '2005'
...
---
_id: '3443'
abstract:
- lang: eng
text: In the hippocampal CA1 area, a relatively homogenous population of pyramidal
cells is accompanied by a diversity of GABAergic interneurons. Previously, we
found that parvalbumin-expressing basket, axo-axonic, bistratified, and oriens-lacunosum
moleculare cells, innervating different domains of pyramidal cells, have distinct
firing patterns during network oscillations in vivo. A second family of interneurons,
expressing cholecystokinin but not parvalbumin, is known to target the same domains
of pyramidal cells as do the parvalbumin cells. To test the temporal activity
of these independent and parallel GABAergic inputs, we recorded the precise spike
timing of identified cholecystokinin interneurons during hippocampal network oscillations
in anesthetized rats and determined their molecular expression profiles and synaptic
targets. The cells were cannabinoid receptor type 1 immunopositive. Contrary to
the stereotyped firing of parvalbumin interneurons, cholecystokinin-expressing
basket and dendrite-innervating cells discharge, on average, with 1.7 ± 2.0 Hz
during high-frequency ripple oscillations in an episode-dependent manner. During
theta oscillations, cholecystokinin- expressing interneurons fire with 8.8 ± 3.3
Hz at a characteristic time on the ascending phase of theta waves (155 ± 81°),
when place cells start firing in freely moving animals. The firing patterns of
some interneurons recorded in drug-free behaving rats were similar to cholecystokinin
cells in anesthetized animals. Our results demonstrate that cholecystokinin- and
parvalbumin-expressing interneurons make different contributions to network oscillations
and play distinct roles in different brain states. We suggest that the specific
spike timing of cholecystokinin interneurons and their sensitivity to endocannabinoids
might contribute to differentiate subgroups of pyramidal cells forming neuronal
assemblies, whereas parvalbumin interneurons contribute to synchronizing the entire
network. Copyright © 2005 Society for Neuroscience.
author:
- first_name: Thomas
full_name: Klausberger,Thomas
last_name: Klausberger
- first_name: Laszlo
full_name: Marton,Laszlo F
last_name: Marton
- first_name: Joseph
full_name: Joseph O'Neill
id: 426376DC-F248-11E8-B48F-1D18A9856A87
last_name: O'Neill
- first_name: Jojanneke
full_name: Huck, Jojanneke H
last_name: Huck
- first_name: Yannis
full_name: Dalezios, Yannis
last_name: Dalezios
- first_name: Pablo
full_name: Fuentealba,Pablo
last_name: Fuentealba
- first_name: Wai
full_name: Suen, Wai Yee
last_name: Suen
- first_name: Edit
full_name: Papp, Edit Cs
last_name: Papp
- first_name: Takeshi
full_name: Kaneko, Takeshi
last_name: Kaneko
- first_name: Masahiko
full_name: Watanabe, Masahiko
last_name: Watanabe
- first_name: Jozsef L
full_name: Jozsef Csicsvari
id: 3FA14672-F248-11E8-B48F-1D18A9856A87
last_name: Csicsvari
orcid: 0000-0002-5193-4036
- first_name: Péter
full_name: Somogyi, Péter
last_name: Somogyi
citation:
ama: Klausberger T, Marton L, O’Neill J, et al. Complementary roles of cholecystokinin-
and parvalbumin-expressing GABAergic neurons in hippocampal network oscillations.
Journal of Neuroscience. 2005;25(42):9782-9793. doi:10.1523/JNEUROSCI.3269-05.2005
apa: Klausberger, T., Marton, L., O’Neill, J., Huck, J., Dalezios, Y., Fuentealba,
P., … Somogyi, P. (2005). Complementary roles of cholecystokinin- and parvalbumin-expressing
GABAergic neurons in hippocampal network oscillations. Journal of Neuroscience.
Society for Neuroscience. https://doi.org/10.1523/JNEUROSCI.3269-05.2005
chicago: Klausberger, Thomas, Laszlo Marton, Joseph O’Neill, Jojanneke Huck, Yannis
Dalezios, Pablo Fuentealba, Wai Suen, et al. “Complementary Roles of Cholecystokinin-
and Parvalbumin-Expressing GABAergic Neurons in Hippocampal Network Oscillations.”
Journal of Neuroscience. Society for Neuroscience, 2005. https://doi.org/10.1523/JNEUROSCI.3269-05.2005.
ieee: T. Klausberger et al., “Complementary roles of cholecystokinin- and
parvalbumin-expressing GABAergic neurons in hippocampal network oscillations,”
Journal of Neuroscience, vol. 25, no. 42. Society for Neuroscience, pp.
9782–9793, 2005.
ista: Klausberger T, Marton L, O’Neill J, Huck J, Dalezios Y, Fuentealba P, Suen
W, Papp E, Kaneko T, Watanabe M, Csicsvari JL, Somogyi P. 2005. Complementary
roles of cholecystokinin- and parvalbumin-expressing GABAergic neurons in hippocampal
network oscillations. Journal of Neuroscience. 25(42), 9782–9793.
mla: Klausberger, Thomas, et al. “Complementary Roles of Cholecystokinin- and Parvalbumin-Expressing
GABAergic Neurons in Hippocampal Network Oscillations.” Journal of Neuroscience,
vol. 25, no. 42, Society for Neuroscience, 2005, pp. 9782–93, doi:10.1523/JNEUROSCI.3269-05.2005.
short: T. Klausberger, L. Marton, J. O’Neill, J. Huck, Y. Dalezios, P. Fuentealba,
W. Suen, E. Papp, T. Kaneko, M. Watanabe, J.L. Csicsvari, P. Somogyi, Journal
of Neuroscience 25 (2005) 9782–9793.
date_created: 2018-12-11T12:03:21Z
date_published: 2005-10-19T00:00:00Z
date_updated: 2021-01-12T07:43:30Z
day: '19'
doi: 10.1523/JNEUROSCI.3269-05.2005
extern: 1
intvolume: ' 25'
issue: '42'
month: '10'
page: 9782 - 9793
publication: Journal of Neuroscience
publication_status: published
publisher: Society for Neuroscience
publist_id: '2944'
quality_controlled: 0
status: public
title: Complementary roles of cholecystokinin- and parvalbumin-expressing GABAergic
neurons in hippocampal network oscillations
type: journal_article
volume: 25
year: '2005'
...
---
_id: '3557'
abstract:
- lang: eng
text: A challenging problem in computer-aided geometric design is the decomposition
of a surface into four-sided regions that are then represented by NURBS patches.
There are various approaches published in the literature and implemented as commercially
available software, but all fall short in either automation or quality of the
result. At Raindrop Geomagic, we have recently taken a fresh approach based on
concepts from Morse theory. This by itself is not a new idea, but we have some
novel ingredients that make this work, one being a rational notion of hierarchy
that guides the construction of a simplified decomposition sensitive to only the
major critical points.
author:
- first_name: Herbert
full_name: Herbert Edelsbrunner
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
citation:
ama: 'Edelsbrunner H. Surface tiling with differential topology. In: ACM; 2005:9-11.
doi:http://dx.doi.org/10.2312/SGP/SGP05/009-011'
apa: 'Edelsbrunner, H. (2005). Surface tiling with differential topology (pp. 9–11).
Presented at the SGP: Eurographics Symposium on Geometry processing, ACM. http://dx.doi.org/10.2312/SGP/SGP05/009-011'
chicago: Edelsbrunner, Herbert. “Surface Tiling with Differential Topology,” 9–11.
ACM, 2005. http://dx.doi.org/10.2312/SGP/SGP05/009-011.
ieee: 'H. Edelsbrunner, “Surface tiling with differential topology,” presented at
the SGP: Eurographics Symposium on Geometry processing, 2005, pp. 9–11.'
ista: 'Edelsbrunner H. 2005. Surface tiling with differential topology. SGP: Eurographics
Symposium on Geometry processing, 9–11.'
mla: Edelsbrunner, Herbert. Surface Tiling with Differential Topology. ACM,
2005, pp. 9–11, doi:http://dx.doi.org/10.2312/SGP/SGP05/009-011.
short: H. Edelsbrunner, in:, ACM, 2005, pp. 9–11.
conference:
name: 'SGP: Eurographics Symposium on Geometry processing'
date_created: 2018-12-11T12:03:57Z
date_published: 2005-07-01T00:00:00Z
date_updated: 2021-01-12T07:44:17Z
day: '01'
doi: http://dx.doi.org/10.2312/SGP/SGP05/009-011
extern: 1
main_file_link:
- open_access: '0'
url: http://www.cs.duke.edu/~edels/Papers/2005-P-03-SurfaceTiling.pdf
month: '07'
page: 9 - 11
publication_status: published
publisher: ACM
publist_id: '2828'
quality_controlled: 0
status: public
title: Surface tiling with differential topology
type: conference
year: '2005'
...