--- _id: '2655' abstract: - lang: eng text: Input-dependent left-right asymmetry of NMDA receptor ε2 (NR2B) subunit allocation was discovered in hippocampal Schaffer collateral (Sch) and commissural fiber pyramidal cell synapses (Kawakami et al., 2003). To investigate whether this asymmetrical ε2 allocation is also related to the types of the postsynaptic cells, we compared postembedding immunogold labeling for ε2 in left and right Sch synapses on pyramidal cells and interneurons. To facilitate the detection of ε2 density difference, we used ε1 (NR2A) knock-out (KO) mice, which have a simplified NMDA receptor subunit composition. The labeling density for ε2 but not ζ1 (NR1) and subtype 2/3 glutamate receptor (GluR2/3) in Sch-CA1 pyramidal cell synapses was significantly different between the left and right hippocampus with opposite directions in strata oriens and radiatum; the left to right ratio of ε2 labeling density was 1:1.50 in stratum oriens and 1.44:1 in stratum radiatum. No significant difference, however, was detected in CA1 stratum radiatum between the left and right Sch-GluR4-positive (mostly parvalbumin-positive) and Sch-GluR4-negative interneuron synapses. Consistent with the anatomical asymmetry, the amplitude ratio of NMDA EPSCs to non-NMDA EPSCs in pyramidal cells was approximately two times larger in right than left stratum radiatum and vice versa in stratum oriens of ε1 KO mice. Moreover, the amplitude of long-term potentiation in the Sch-CA1 synapses of left stratum radiatum was significantly larger than that in the right corresponding synapses. These results indicate that the asymmetry of ε2 distribution is target cell specific, resulting in the left-right difference in NMDA receptor content and plasticity in Sch-CA1 pyramidal cell synapses in ε1 KO mice. author: - first_name: Yue full_name: Wu, Yue last_name: Wu - first_name: Ryosuke full_name: Kawakami, Ryosuke last_name: Kawakami - first_name: Yoshiaki full_name: Shinohara, Yoshiaki last_name: Shinohara - first_name: Masahiro full_name: Fukaya, Masahiro last_name: Fukaya - first_name: Kenji full_name: Sakimura, Kenji last_name: Sakimura - first_name: Masayoshi full_name: Mishina, Masayoshi last_name: Mishina - first_name: Masahiko full_name: Watanabe, Masahiko last_name: Watanabe - first_name: Isao full_name: Ito, Isao last_name: Ito - first_name: Ryuichi full_name: Ryuichi Shigemoto id: 499F3ABC-F248-11E8-B48F-1D18A9856A87 last_name: Shigemoto orcid: 0000-0001-8761-9444 citation: ama: Wu Y, Kawakami R, Shinohara Y, et al. Target-cell-specific left-right asymmetry of NMDA receptor content in Schaffer collateral synapses in ε1/NR2A knock-out mice. Journal of Neuroscience. 2005;25(40):9213-9226. doi:10.1523/JNEUROSCI.2134-05.2005 apa: Wu, Y., Kawakami, R., Shinohara, Y., Fukaya, M., Sakimura, K., Mishina, M., … Shigemoto, R. (2005). Target-cell-specific left-right asymmetry of NMDA receptor content in Schaffer collateral synapses in ε1/NR2A knock-out mice. Journal of Neuroscience. Society for Neuroscience. https://doi.org/10.1523/JNEUROSCI.2134-05.2005 chicago: Wu, Yue, Ryosuke Kawakami, Yoshiaki Shinohara, Masahiro Fukaya, Kenji Sakimura, Masayoshi Mishina, Masahiko Watanabe, Isao Ito, and Ryuichi Shigemoto. “Target-Cell-Specific Left-Right Asymmetry of NMDA Receptor Content in Schaffer Collateral Synapses in Ε1/NR2A Knock-out Mice.” Journal of Neuroscience. Society for Neuroscience, 2005. https://doi.org/10.1523/JNEUROSCI.2134-05.2005. ieee: Y. Wu et al., “Target-cell-specific left-right asymmetry of NMDA receptor content in Schaffer collateral synapses in ε1/NR2A knock-out mice,” Journal of Neuroscience, vol. 25, no. 40. Society for Neuroscience, pp. 9213–9226, 2005. ista: Wu Y, Kawakami R, Shinohara Y, Fukaya M, Sakimura K, Mishina M, Watanabe M, Ito I, Shigemoto R. 2005. Target-cell-specific left-right asymmetry of NMDA receptor content in Schaffer collateral synapses in ε1/NR2A knock-out mice. Journal of Neuroscience. 25(40), 9213–9226. mla: Wu, Yue, et al. “Target-Cell-Specific Left-Right Asymmetry of NMDA Receptor Content in Schaffer Collateral Synapses in Ε1/NR2A Knock-out Mice.” Journal of Neuroscience, vol. 25, no. 40, Society for Neuroscience, 2005, pp. 9213–26, doi:10.1523/JNEUROSCI.2134-05.2005. short: Y. Wu, R. Kawakami, Y. Shinohara, M. Fukaya, K. Sakimura, M. Mishina, M. Watanabe, I. Ito, R. Shigemoto, Journal of Neuroscience 25 (2005) 9213–9226. date_created: 2018-12-11T11:58:54Z date_published: 2005-10-05T00:00:00Z date_updated: 2021-01-12T06:58:51Z day: '05' doi: 10.1523/JNEUROSCI.2134-05.2005 extern: 1 intvolume: ' 25' issue: '40' month: '10' page: 9213 - 9226 publication: Journal of Neuroscience publication_status: published publisher: Society for Neuroscience publist_id: '4243' quality_controlled: 0 status: public title: Target-cell-specific left-right asymmetry of NMDA receptor content in Schaffer collateral synapses in ε1/NR2A knock-out mice type: journal_article volume: 25 year: '2005' ... --- _id: '2653' abstract: - lang: eng text: Synaptic vesicle release occurs at a specialized membrane domain known as the presynaptic active zone (AZ). Several membrane proteins are involved in the vesicle release processes such as docking, priming, and exocytotic fusion. Cytomatrix at the active zone (CAZ) proteins are structural components of the AZ and are highly concentrated in it. Localization of other release-related proteins including target soluble N-ethylmaleimide-sensitive-factor attachment protein receptor (t-SNARE) proteins, however, has not been well demonstrated in the AZ. Here, we used sodium dodecyl sulfate-digested freeze-fracture replica labeling (SDS-FRL) to analyze quantitatively the distribution of CAZ and t-SNARE proteins in the hippocampal CA3 area. The AZ in replicated membrane was identified by immunolabeling for CAZ proteins (CAZ-associated structural protein [CAST] and Bassoon). Clusters of immunogold particles for these proteins were found on the P-face of presynaptic terminals of the mossy fiber and associational/commissural (AJC) fiber. Co-labeling with CAST revealed distribution of the t-SNARE proteins syntaxin and synaptosomal-associated protein of 25 kDa (SNAP-25) in the AZ as well as in the extrasynaptic membrane surrounding the AZ (SZ). Quantitative analysis demonstrated that the density of immunoparticles for CAST in the AZ was more than 100 times higher than in the SZ, whereas that for syntaxin and SNAP-25 was not significantly different between the AZ and SZ in both the A/C and mossy fiber terminals. These results support the involvement of the t-SNARE proteins in exocytotic fusion in the AZ and the role of CAST in specialization of the membrane domain for the AZ. author: - first_name: Akari full_name: Hagiwara, Akari last_name: Hagiwara - first_name: Yugo full_name: Fukazawa, Yugo last_name: Fukazawa - first_name: Maki full_name: Deguchi-Tawarada, Maki last_name: Deguchi Tawarada - first_name: Toshihisa full_name: Ohtsuka, Toshihisa last_name: Ohtsuka - first_name: Ryuichi full_name: Ryuichi Shigemoto id: 499F3ABC-F248-11E8-B48F-1D18A9856A87 last_name: Shigemoto orcid: 0000-0001-8761-9444 citation: ama: Hagiwara A, Fukazawa Y, Deguchi Tawarada M, Ohtsuka T, Shigemoto R. Differential distribution of release-related proteins in the hippocampal CA3 area as revealed by freeze-fracture replica labeling. Journal of Comparative Neurology. 2005;489(2):195-216. doi:10.1002/cne.20633 apa: Hagiwara, A., Fukazawa, Y., Deguchi Tawarada, M., Ohtsuka, T., & Shigemoto, R. (2005). Differential distribution of release-related proteins in the hippocampal CA3 area as revealed by freeze-fracture replica labeling. Journal of Comparative Neurology. Wiley-Blackwell. https://doi.org/10.1002/cne.20633 chicago: Hagiwara, Akari, Yugo Fukazawa, Maki Deguchi Tawarada, Toshihisa Ohtsuka, and Ryuichi Shigemoto. “Differential Distribution of Release-Related Proteins in the Hippocampal CA3 Area as Revealed by Freeze-Fracture Replica Labeling.” Journal of Comparative Neurology. Wiley-Blackwell, 2005. https://doi.org/10.1002/cne.20633. ieee: A. Hagiwara, Y. Fukazawa, M. Deguchi Tawarada, T. Ohtsuka, and R. Shigemoto, “Differential distribution of release-related proteins in the hippocampal CA3 area as revealed by freeze-fracture replica labeling,” Journal of Comparative Neurology, vol. 489, no. 2. Wiley-Blackwell, pp. 195–216, 2005. ista: Hagiwara A, Fukazawa Y, Deguchi Tawarada M, Ohtsuka T, Shigemoto R. 2005. Differential distribution of release-related proteins in the hippocampal CA3 area as revealed by freeze-fracture replica labeling. Journal of Comparative Neurology. 489(2), 195–216. mla: Hagiwara, Akari, et al. “Differential Distribution of Release-Related Proteins in the Hippocampal CA3 Area as Revealed by Freeze-Fracture Replica Labeling.” Journal of Comparative Neurology, vol. 489, no. 2, Wiley-Blackwell, 2005, pp. 195–216, doi:10.1002/cne.20633. short: A. Hagiwara, Y. Fukazawa, M. Deguchi Tawarada, T. Ohtsuka, R. Shigemoto, Journal of Comparative Neurology 489 (2005) 195–216. date_created: 2018-12-11T11:58:53Z date_published: 2005-08-22T00:00:00Z date_updated: 2021-01-12T06:58:50Z day: '22' doi: 10.1002/cne.20633 extern: 1 intvolume: ' 489' issue: '2' month: '08' page: 195 - 216 publication: Journal of Comparative Neurology publication_status: published publisher: Wiley-Blackwell publist_id: '4244' quality_controlled: 0 status: public title: Differential distribution of release-related proteins in the hippocampal CA3 area as revealed by freeze-fracture replica labeling type: journal_article volume: 489 year: '2005' ... --- _id: '2656' abstract: - lang: eng text: Previous studies have shown that neurons in the sacral dorsal commissural nucleus (SDCN) express neurokinin-1 receptor (NK1R) and can be modulated by the co-release of GABA and glycine (Gly) from single presynaptic terminal. These results raise the possibility that GABA/Gly-cocontaining terminals might make synaptic contacts with NK1R-expressing neurons in the SDCN. In order to provide morphological evidence for this hypothesis, the triple-immunohistochemical studies were performed in the SDCN. Triple-immunofluorescence histochemical study showed that some axon terminals in close association with NK1R-immunopositive (NK1R-ip) neurons in the SDCN were immunopositive for both glutamic acid decarboxylase (GAD) and glycine transporter 2 (GlyT2). In electron microscopic dual- and triple-immunohistochemistry for GAD/GlyT2, GAD/NK1R, GlyT2/NK1R, or GAD/GlyT2/NK1R also revealed dually labeled (GAD/GlyT2-ip) synaptic terminals upon SDCN neurons, as well as GAD- and/or GlyT2-ip axon terminals in synaptic contact with NK1R-ip SDCN neurons. These results suggested that some synaptic terminals upon NK1R-expressing SDCN neurons co-released both GABA and Gly. author: - first_name: Yu full_name: Feng, Yu-Peng last_name: Feng - first_name: Yun full_name: Li, Yun-Qing last_name: Li - first_name: Wen full_name: Wang, Wen last_name: Wang - first_name: Sheng full_name: Wu, Sheng-Xi last_name: Wu - first_name: Tao full_name: Chen, Tao last_name: Chen - first_name: Ryuichi full_name: Ryuichi Shigemoto id: 499F3ABC-F248-11E8-B48F-1D18A9856A87 last_name: Shigemoto orcid: 0000-0001-8761-9444 - first_name: Noboru full_name: Mizuno, Noboru last_name: Mizuno citation: ama: Feng Y, Li Y, Wang W, et al. Morphological evidence for GABA/glycine-cocontaining terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural nucleus of the rat. Neuroscience Letters. 2005;388(3):144-148. doi:10.1016/j.neulet.2005.06.068 apa: Feng, Y., Li, Y., Wang, W., Wu, S., Chen, T., Shigemoto, R., & Mizuno, N. (2005). Morphological evidence for GABA/glycine-cocontaining terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural nucleus of the rat. Neuroscience Letters. Elsevier. https://doi.org/10.1016/j.neulet.2005.06.068 chicago: Feng, Yu, Yun Li, Wen Wang, Sheng Wu, Tao Chen, Ryuichi Shigemoto, and Noboru Mizuno. “Morphological Evidence for GABA/Glycine-Cocontaining Terminals in Synaptic Contact with Neurokinin-1 Receptor-Expressing Neurons in the Sacral Dorsal Commissural Nucleus of the Rat.” Neuroscience Letters. Elsevier, 2005. https://doi.org/10.1016/j.neulet.2005.06.068. ieee: Y. Feng et al., “Morphological evidence for GABA/glycine-cocontaining terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural nucleus of the rat,” Neuroscience Letters, vol. 388, no. 3. Elsevier, pp. 144–148, 2005. ista: Feng Y, Li Y, Wang W, Wu S, Chen T, Shigemoto R, Mizuno N. 2005. Morphological evidence for GABA/glycine-cocontaining terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural nucleus of the rat. Neuroscience Letters. 388(3), 144–148. mla: Feng, Yu, et al. “Morphological Evidence for GABA/Glycine-Cocontaining Terminals in Synaptic Contact with Neurokinin-1 Receptor-Expressing Neurons in the Sacral Dorsal Commissural Nucleus of the Rat.” Neuroscience Letters, vol. 388, no. 3, Elsevier, 2005, pp. 144–48, doi:10.1016/j.neulet.2005.06.068. short: Y. Feng, Y. Li, W. Wang, S. Wu, T. Chen, R. Shigemoto, N. Mizuno, Neuroscience Letters 388 (2005) 144–148. date_created: 2018-12-11T11:58:54Z date_published: 2005-11-18T00:00:00Z date_updated: 2021-01-12T06:58:51Z day: '18' doi: 10.1016/j.neulet.2005.06.068 extern: 1 intvolume: ' 388' issue: '3' month: '11' page: 144 - 148 publication: Neuroscience Letters publication_status: published publisher: Elsevier publist_id: '4241' quality_controlled: 0 status: public title: Morphological evidence for GABA/glycine-cocontaining terminals in synaptic contact with neurokinin-1 receptor-expressing neurons in the sacral dorsal commissural nucleus of the rat type: journal_article volume: 388 year: '2005' ... --- _id: '2744' abstract: - lang: eng text: We study the long time evolution of a quantum particle interacting with a random potential in the Boltzmann-Grad low density limit. We prove that the phase space density of the quantum evolution defined through the Husimi function converges weakly to a linear Boltzmann equation. The Boltzmann collision kernel is given by the full quantum scattering cross-section of the obstacle potential. author: - first_name: David full_name: Eng, David last_name: Eng - first_name: László full_name: László Erdös id: 4DBD5372-F248-11E8-B48F-1D18A9856A87 last_name: Erdös orcid: 0000-0001-5366-9603 citation: ama: Eng D, Erdös L. The linear Boltzmann equation as the low density limit of a random Schrödinger equation. Reviews in Mathematical Physics. 2005;17(6):669-743. doi:10.1142/S0129055X0500242X apa: Eng, D., & Erdös, L. (2005). The linear Boltzmann equation as the low density limit of a random Schrödinger equation. Reviews in Mathematical Physics. World Scientific Publishing. https://doi.org/10.1142/S0129055X0500242X chicago: Eng, David, and László Erdös. “The Linear Boltzmann Equation as the Low Density Limit of a Random Schrödinger Equation.” Reviews in Mathematical Physics. World Scientific Publishing, 2005. https://doi.org/10.1142/S0129055X0500242X. ieee: D. Eng and L. Erdös, “The linear Boltzmann equation as the low density limit of a random Schrödinger equation,” Reviews in Mathematical Physics, vol. 17, no. 6. World Scientific Publishing, pp. 669–743, 2005. ista: Eng D, Erdös L. 2005. The linear Boltzmann equation as the low density limit of a random Schrödinger equation. Reviews in Mathematical Physics. 17(6), 669–743. mla: Eng, David, and László Erdös. “The Linear Boltzmann Equation as the Low Density Limit of a Random Schrödinger Equation.” Reviews in Mathematical Physics, vol. 17, no. 6, World Scientific Publishing, 2005, pp. 669–743, doi:10.1142/S0129055X0500242X. short: D. Eng, L. Erdös, Reviews in Mathematical Physics 17 (2005) 669–743. date_created: 2018-12-11T11:59:22Z date_published: 2005-07-01T00:00:00Z date_updated: 2021-01-12T06:59:25Z day: '01' doi: 10.1142/S0129055X0500242X extern: 1 intvolume: ' 17' issue: '6' month: '07' page: 669 - 743 publication: Reviews in Mathematical Physics publication_status: published publisher: World Scientific Publishing publist_id: '4148' quality_controlled: 0 status: public title: The linear Boltzmann equation as the low density limit of a random Schrödinger equation type: journal_article volume: 17 year: '2005' ... --- _id: '2743' abstract: - lang: eng text: We consider the supersymmetric quantum mechanical system which is obtained by dimensionally reducing d = 6, N = 1 supersymmetric gauge theory with gauge group U(1) and a single charged hypermultiplet. Using the deformation method and ideas introduced by Porrati and Rozenberg [1], we present a detailed proof of the existence of a normalizable ground state for this system. author: - first_name: László full_name: László Erdös id: 4DBD5372-F248-11E8-B48F-1D18A9856A87 last_name: Erdös orcid: 0000-0001-5366-9603 - first_name: David full_name: Hasler, David G last_name: Hasler - first_name: Jan full_name: Solovej, Jan P last_name: Solovej citation: ama: 'Erdös L, Hasler D, Solovej J. Existence of the D0-D4 bound state: A detailed proof. Annales Henri Poincare. 2005;6(2):247-267. doi:10.1007/s00023-005-0205-0' apa: 'Erdös, L., Hasler, D., & Solovej, J. (2005). Existence of the D0-D4 bound state: A detailed proof. Annales Henri Poincare. Birkhäuser. https://doi.org/10.1007/s00023-005-0205-0' chicago: 'Erdös, László, David Hasler, and Jan Solovej. “Existence of the D0-D4 Bound State: A Detailed Proof.” Annales Henri Poincare. Birkhäuser, 2005. https://doi.org/10.1007/s00023-005-0205-0.' ieee: 'L. Erdös, D. Hasler, and J. Solovej, “Existence of the D0-D4 bound state: A detailed proof,” Annales Henri Poincare, vol. 6, no. 2. Birkhäuser, pp. 247–267, 2005.' ista: 'Erdös L, Hasler D, Solovej J. 2005. Existence of the D0-D4 bound state: A detailed proof. Annales Henri Poincare. 6(2), 247–267.' mla: 'Erdös, László, et al. “Existence of the D0-D4 Bound State: A Detailed Proof.” Annales Henri Poincare, vol. 6, no. 2, Birkhäuser, 2005, pp. 247–67, doi:10.1007/s00023-005-0205-0.' short: L. Erdös, D. Hasler, J. Solovej, Annales Henri Poincare 6 (2005) 247–267. date_created: 2018-12-11T11:59:22Z date_published: 2005-04-01T00:00:00Z date_updated: 2021-01-12T06:59:24Z day: '01' doi: 10.1007/s00023-005-0205-0 extern: 1 intvolume: ' 6' issue: '2' month: '04' page: 247 - 267 publication: Annales Henri Poincare publication_status: published publisher: Birkhäuser publist_id: '4149' quality_controlled: 0 status: public title: 'Existence of the D0-D4 bound state: A detailed proof' type: journal_article volume: 6 year: '2005' ... --- _id: '2788' abstract: - lang: eng text: We present the results of an experimental investigation into the nature and structure of turbulent pipe flow at moderate Reynolds numbers. A turbulence regeneration mechanism is identified which sustains a symmetric traveling wave within the flow. The periodicity of the mechanism allows comparison to the wavelength of numerically observed exact traveling wave solutions and close agreement is found. The advection speed of the upstream turbulence laminar interface in the experimental flow is observed to form a lower bound on the phase velocities of the exact traveling wave solutions. Overall our observations suggest that the dynamics of the turbulent flow at moderate Reynolds numbers are governed by unstable nonlinear traveling waves. author: - first_name: Björn full_name: Björn Hof id: 3A374330-F248-11E8-B48F-1D18A9856A87 last_name: Hof orcid: 0000-0003-2057-2754 - first_name: Casimir full_name: van Doorne, Casimir W last_name: Van Doorne - first_name: Jerry full_name: Westerweel, Jerry last_name: Westerweel - first_name: Frans full_name: Nieuwstadt, Frans T last_name: Nieuwstadt citation: ama: Hof B, Van Doorne C, Westerweel J, Nieuwstadt F. Turbulence regeneration in pipe flow at moderate reynolds numbers. Physical Review Letters. 2005;95(21). doi:10.1103/PhysRevLett.95.214502 apa: Hof, B., Van Doorne, C., Westerweel, J., & Nieuwstadt, F. (2005). Turbulence regeneration in pipe flow at moderate reynolds numbers. Physical Review Letters. American Physical Society. https://doi.org/10.1103/PhysRevLett.95.214502 chicago: Hof, Björn, Casimir Van Doorne, Jerry Westerweel, and Frans Nieuwstadt. “Turbulence Regeneration in Pipe Flow at Moderate Reynolds Numbers.” Physical Review Letters. American Physical Society, 2005. https://doi.org/10.1103/PhysRevLett.95.214502. ieee: B. Hof, C. Van Doorne, J. Westerweel, and F. Nieuwstadt, “Turbulence regeneration in pipe flow at moderate reynolds numbers,” Physical Review Letters, vol. 95, no. 21. American Physical Society, 2005. ista: Hof B, Van Doorne C, Westerweel J, Nieuwstadt F. 2005. Turbulence regeneration in pipe flow at moderate reynolds numbers. Physical Review Letters. 95(21). mla: Hof, Björn, et al. “Turbulence Regeneration in Pipe Flow at Moderate Reynolds Numbers.” Physical Review Letters, vol. 95, no. 21, American Physical Society, 2005, doi:10.1103/PhysRevLett.95.214502. short: B. Hof, C. Van Doorne, J. Westerweel, F. Nieuwstadt, Physical Review Letters 95 (2005). date_created: 2018-12-11T11:59:36Z date_published: 2005-11-17T00:00:00Z date_updated: 2021-01-12T06:59:43Z day: '17' doi: 10.1103/PhysRevLett.95.214502 extern: 1 intvolume: ' 95' issue: '21' month: '11' publication: Physical Review Letters publication_status: published publisher: American Physical Society publist_id: '4101' quality_controlled: 0 status: public title: Turbulence regeneration in pipe flow at moderate reynolds numbers type: journal_article volume: 95 year: '2005' ... --- _id: '2790' abstract: - lang: eng text: We present the results of an experimental investigation of the effect of a magnetic field on the stability of convection in a liquid metal. A rectangular container of gallium is subjected to a horizontal temperature gradient and a uniform magnetic field is applied separately in three directions. The magnetic field suppresses the oscillation most effectively when it is applied in the vertical direction and is least efficient when applied in the direction of the temperature gradient. The critical temperature difference required for the onset of oscillations is found to scale exponentially with the magnitude of the magnetic field for all three orientations. Comparisons are made with available theory and qualitative differences are discussed. author: - first_name: Björn full_name: Björn Hof id: 3A374330-F248-11E8-B48F-1D18A9856A87 last_name: Hof orcid: 0000-0003-2057-2754 - first_name: Anne full_name: Juel, Anne last_name: Juel - first_name: Tom full_name: Mullin, Tom P last_name: Mullin citation: ama: Hof B, Juel A, Mullin T. Magnetohydrodynamic damping of oscillations in low-Prandtl-number convection. Journal of Fluid Mechanics. 2005;545:193-201. doi:10.1017/S0022112005006762 apa: Hof, B., Juel, A., & Mullin, T. (2005). Magnetohydrodynamic damping of oscillations in low-Prandtl-number convection. Journal of Fluid Mechanics. Cambridge University Press. https://doi.org/10.1017/S0022112005006762 chicago: Hof, Björn, Anne Juel, and Tom Mullin. “Magnetohydrodynamic Damping of Oscillations in Low-Prandtl-Number Convection.” Journal of Fluid Mechanics. Cambridge University Press, 2005. https://doi.org/10.1017/S0022112005006762. ieee: B. Hof, A. Juel, and T. Mullin, “Magnetohydrodynamic damping of oscillations in low-Prandtl-number convection,” Journal of Fluid Mechanics, vol. 545. Cambridge University Press, pp. 193–201, 2005. ista: Hof B, Juel A, Mullin T. 2005. Magnetohydrodynamic damping of oscillations in low-Prandtl-number convection. Journal of Fluid Mechanics. 545, 193–201. mla: Hof, Björn, et al. “Magnetohydrodynamic Damping of Oscillations in Low-Prandtl-Number Convection.” Journal of Fluid Mechanics, vol. 545, Cambridge University Press, 2005, pp. 193–201, doi:10.1017/S0022112005006762. short: B. Hof, A. Juel, T. Mullin, Journal of Fluid Mechanics 545 (2005) 193–201. date_created: 2018-12-11T11:59:37Z date_published: 2005-12-25T00:00:00Z date_updated: 2021-01-12T06:59:44Z day: '25' doi: 10.1017/S0022112005006762 extern: 1 intvolume: ' 545' month: '12' page: 193 - 201 publication: Journal of Fluid Mechanics publication_status: published publisher: Cambridge University Press publist_id: '4099' quality_controlled: 0 status: public title: Magnetohydrodynamic damping of oscillations in low-Prandtl-number convection type: journal_article volume: 545 year: '2005' ... --- _id: '2789' abstract: - lang: eng text: Transitional pipe flow is investigated in two different experimental set-ups. In the first the stability threshold and the initial growth of localized perturbations are studied. Good agreement is found with an earlier investigation of the transition threshold. The measurement technique applied in the last part of this study allows the reconstruction of the streamwise vorticity in a turbulent puff. author: - first_name: Björn full_name: Björn Hof id: 3A374330-F248-11E8-B48F-1D18A9856A87 last_name: Hof orcid: 0000-0003-2057-2754 citation: ama: Hof B. Transition to turbulence in pipe flow. Fluid Mechanics and its Applications. 2005;77:221-231. doi:10.1007/1-4020-4049-0_12 apa: Hof, B. (2005). Transition to turbulence in pipe flow. Fluid Mechanics and Its Applications. Springer. https://doi.org/10.1007/1-4020-4049-0_12 chicago: Hof, Björn. “Transition to Turbulence in Pipe Flow.” Fluid Mechanics and Its Applications. Springer, 2005. https://doi.org/10.1007/1-4020-4049-0_12. ieee: B. Hof, “Transition to turbulence in pipe flow,” Fluid Mechanics and its Applications, vol. 77. Springer, pp. 221–231, 2005. ista: Hof B. 2005. Transition to turbulence in pipe flow. Fluid Mechanics and its Applications. 77, 221–231. mla: Hof, Björn. “Transition to Turbulence in Pipe Flow.” Fluid Mechanics and Its Applications, vol. 77, Springer, 2005, pp. 221–31, doi:10.1007/1-4020-4049-0_12. short: B. Hof, Fluid Mechanics and Its Applications 77 (2005) 221–231. date_created: 2018-12-11T11:59:36Z date_published: 2005-09-19T00:00:00Z date_updated: 2021-01-12T06:59:43Z day: '19' doi: 10.1007/1-4020-4049-0_12 extern: 1 intvolume: ' 77' month: '09' page: 221 - 231 publication: Fluid Mechanics and its Applications publication_status: published publisher: Springer publist_id: '4100' quality_controlled: 0 status: public title: Transition to turbulence in pipe flow type: journal_article volume: 77 year: '2005' ... --- _id: '2867' abstract: - lang: eng text: The plant hormone auxin elicits many specific context-dependent developmental responses. Auxin promotes degradation of Aux/IAA proteins that prevent transcription factors of the auxin response factor (ARF) family from regulating auxin-responsive target genes. Aux/IAAs and ARFs are represented by large gene families in Arabidopsis. Here we show that stabilization of BDL/IAA12 or its sister protein IAA13 prevents MP/ARF5-dependent embryonic root formation whereas stabilized SHY2/IAA3 interferes with seedling growth. Although both bdl and shy2-2 proteins inhibited MP/ARF5-dependent reporter gene activation, shy2-2 was much less efficient than bdl to interfere with embryonic root initiation when expressed from the BDL promoter. Similarly, MP was much more efficient than ARF16 in this process. When expressed from the SHY2 promoter, both shy2-2 and bdl inhibited cell elongation and auxin-induced gene expression in the seedling hypocotyl. By contrast, gravitropism and auxin-induced gene expression in the root, which were promoted by functionally redundant NPH4/ARF7 and ARF19 proteins, were inhibited by shy2-2, but not by bdl protein. Our results suggest that auxin signals are converted into specific responses by matching pairs of coexpressed ARF and Aux/IAA proteins. author: - first_name: Dolf full_name: Weijers, Dolf last_name: Weijers - first_name: Eva full_name: Eva Benková id: 38F4F166-F248-11E8-B48F-1D18A9856A87 last_name: Benková orcid: 0000-0002-8510-9739 - first_name: Katja full_name: Jäger, Katja E last_name: Jäger - first_name: Alexandra full_name: Schlereth, Alexandra last_name: Schlereth - first_name: Thorsten full_name: Hamann, Thorsten last_name: Hamann - first_name: Marika full_name: Kientz, Marika last_name: Kientz - first_name: Jill full_name: Wilmoth, Jill C last_name: Wilmoth - first_name: Jason full_name: Reed, Jason W last_name: Reed - first_name: Gerd full_name: Jürgens, Gerd last_name: Jürgens citation: ama: Weijers D, Benková E, Jäger K, et al. Developmental specificity of auxin response by pairs of ARF and Aux/IAA transcriptional regulators. EMBO Journal. 2005;24(10):1874-1885. doi:10.1038/sj.emboj.7600659 apa: Weijers, D., Benková, E., Jäger, K., Schlereth, A., Hamann, T., Kientz, M., … Jürgens, G. (2005). Developmental specificity of auxin response by pairs of ARF and Aux/IAA transcriptional regulators. EMBO Journal. Wiley-Blackwell. https://doi.org/10.1038/sj.emboj.7600659 chicago: Weijers, Dolf, Eva Benková, Katja Jäger, Alexandra Schlereth, Thorsten Hamann, Marika Kientz, Jill Wilmoth, Jason Reed, and Gerd Jürgens. “Developmental Specificity of Auxin Response by Pairs of ARF and Aux/IAA Transcriptional Regulators.” EMBO Journal. Wiley-Blackwell, 2005. https://doi.org/10.1038/sj.emboj.7600659. ieee: D. Weijers et al., “Developmental specificity of auxin response by pairs of ARF and Aux/IAA transcriptional regulators,” EMBO Journal, vol. 24, no. 10. Wiley-Blackwell, pp. 1874–1885, 2005. ista: Weijers D, Benková E, Jäger K, Schlereth A, Hamann T, Kientz M, Wilmoth J, Reed J, Jürgens G. 2005. Developmental specificity of auxin response by pairs of ARF and Aux/IAA transcriptional regulators. EMBO Journal. 24(10), 1874–1885. mla: Weijers, Dolf, et al. “Developmental Specificity of Auxin Response by Pairs of ARF and Aux/IAA Transcriptional Regulators.” EMBO Journal, vol. 24, no. 10, Wiley-Blackwell, 2005, pp. 1874–85, doi:10.1038/sj.emboj.7600659. short: D. Weijers, E. Benková, K. Jäger, A. Schlereth, T. Hamann, M. Kientz, J. Wilmoth, J. Reed, G. Jürgens, EMBO Journal 24 (2005) 1874–1885. date_created: 2018-12-11T12:00:01Z date_published: 2005-05-18T00:00:00Z date_updated: 2021-01-12T07:00:22Z day: '18' doi: 10.1038/sj.emboj.7600659 extern: 1 intvolume: ' 24' issue: '10' main_file_link: - open_access: '1' url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1142592/ month: '05' oa: 1 page: 1874 - 1885 publication: EMBO Journal publication_status: published publisher: Wiley-Blackwell publist_id: '3918' quality_controlled: 0 status: public title: Developmental specificity of auxin response by pairs of ARF and Aux/IAA transcriptional regulators type: journal_article volume: 24 year: '2005' ... --- _id: '2895' abstract: - lang: eng text: One of the fundamental properties of the immune system is its capacity to avoid autoimmune diseases. The mechanism underlying this process, known as self-tolerance, is hitherto unresolved but seems to involve the control of clonal expansion of autoreactive lymphocytes. This article reviews mathematical modeling of self-tolerance, addressing two specific hypotheses. The first hypothesis posits that self-tolerance is mediated by tuning of activation thresholds, which makes autoreactive T lymphocytes reversibly "anergic" and unable to proliferate. The second hypothesis posits that the proliferation of autoreactive T lymphocytes is instead controlled by specific regulatory T lymphocytes. Models representing the population dynamics of autoreactive T lymphocytes according to these two hypotheses were derived. For each model we identified how cell density affects tolerance, and predicted the corresponding phase spaces and bifurcations. We show that the simple induction of proliferative anergy, as modeled here, has a density dependence that is only partially compatible with adoptive transfers of tolerance, and that the models of tolerance mediated by specific regulatory T cells are closer to the observations. acknowledgement: 'The work was financially supported by Fundação para a Ciência e Tecnologia: grants P/BIA/10094/1998, POCTI/36413/99, and POCTI/MGI/46477/2002; and fellowships to JF (Praxis/BCC/18972/98), JS (BD/13546/97), KL (SFRH/BPD+/1157/2002), DM (SFRH/BD/2960/2000) and TP (SFRH/BD/10550/2002).' author: - first_name: Jorge full_name: Carneiro, Jorge last_name: Carneiro - first_name: Tiago full_name: Tiago Paixao id: 2C5658E6-F248-11E8-B48F-1D18A9856A87 last_name: Paixao orcid: 0000-0003-2361-3953 - first_name: Dejan full_name: Milutinovic, Dejan last_name: Milutinovic - first_name: João full_name: Sousa, João last_name: Sousa - first_name: Kalet full_name: Leon, Kalet last_name: Leon - first_name: Rui full_name: Gardner, Rui last_name: Gardner - first_name: Jose full_name: Faro, Jose last_name: Faro citation: ama: 'Carneiro J, Paixao T, Milutinovic D, et al. Immunological self tolerance: Lessons from mathematical modeling. Journal of Computational and Applied Mathematics. 2005;184(1):77-100. doi:10.1016/j.cam.2004.10.025' apa: 'Carneiro, J., Paixao, T., Milutinovic, D., Sousa, J., Leon, K., Gardner, R., & Faro, J. (2005). Immunological self tolerance: Lessons from mathematical modeling. Journal of Computational and Applied Mathematics. Elsevier. https://doi.org/10.1016/j.cam.2004.10.025' chicago: 'Carneiro, Jorge, Tiago Paixao, Dejan Milutinovic, João Sousa, Kalet Leon, Rui Gardner, and Jose Faro. “Immunological Self Tolerance: Lessons from Mathematical Modeling.” Journal of Computational and Applied Mathematics. Elsevier, 2005. https://doi.org/10.1016/j.cam.2004.10.025.' ieee: 'J. Carneiro et al., “Immunological self tolerance: Lessons from mathematical modeling,” Journal of Computational and Applied Mathematics, vol. 184, no. 1. Elsevier, pp. 77–100, 2005.' ista: 'Carneiro J, Paixao T, Milutinovic D, Sousa J, Leon K, Gardner R, Faro J. 2005. Immunological self tolerance: Lessons from mathematical modeling. Journal of Computational and Applied Mathematics. 184(1), 77–100.' mla: 'Carneiro, Jorge, et al. “Immunological Self Tolerance: Lessons from Mathematical Modeling.” Journal of Computational and Applied Mathematics, vol. 184, no. 1, Elsevier, 2005, pp. 77–100, doi:10.1016/j.cam.2004.10.025.' short: J. Carneiro, T. Paixao, D. Milutinovic, J. Sousa, K. Leon, R. Gardner, J. Faro, Journal of Computational and Applied Mathematics 184 (2005) 77–100. date_created: 2018-12-11T12:00:12Z date_published: 2005-12-01T00:00:00Z date_updated: 2021-01-12T07:00:32Z day: '01' doi: 10.1016/j.cam.2004.10.025 extern: 1 intvolume: ' 184' issue: '1' month: '12' page: 77 - 100 publication: Journal of Computational and Applied Mathematics publication_status: published publisher: Elsevier publist_id: '3863' quality_controlled: 0 status: public title: 'Immunological self tolerance: Lessons from mathematical modeling' type: journal_article volume: 184 year: '2005' ... --- _id: '3004' abstract: - lang: eng text: Molecular mechanisms of pattern formation in the plant embryo are not well understood. Recent molecular and cellular studies, in conjunction with earlier microsurgical, physiological, and genetic work, are now starting to define the outlines of a model where gradients of the signaling molecule auxin play a central role in embryo patterning. It is relatively clear how these gradients are established and interpreted, but how they are maintained is still unresolved. Here, we have studied the contributions of auxin biosynthesis, conjugation, and transport pathways to the maintenance of embryonic auxin gradients. Auxin homeostasis in the embryo was manipulated by region-specific conditional expression of indoleacetic acid-tryptophan monooxygenase or indoleacetic acid-lysine synthetase, bacterial enzymes for auxin biosynthesis or conjugation. Neither manipulation of auxin biosynthesis nor of auxin conjugation interfered with auxin gradients and patterning in the embryo. This result suggests a compensatory mechanism for buffering auxin gradients in the embryo. Chemical and genetic inhibition revealed that auxin transport activity, in particular that of the PIN-FORMED1 (PIN1) and PIN4 proteins, is a major factor in the maintenance of these gradients. author: - first_name: Dolf full_name: Weijers, Dolf last_name: Weijers - first_name: Michael full_name: Sauer, Michael last_name: Sauer - first_name: Olivier full_name: Meurette, Olivier last_name: Meurette - first_name: Jirí full_name: Jirí Friml id: 4159519E-F248-11E8-B48F-1D18A9856A87 last_name: Friml orcid: 0000-0002-8302-7596 - first_name: Karin full_name: Ljung, Karin last_name: Ljung - first_name: Göran full_name: Sandberg, Göran last_name: Sandberg - first_name: Paul full_name: Hooykaas, Paul last_name: Hooykaas - first_name: Remko full_name: Offringa, Remko last_name: Offringa citation: ama: Weijers D, Sauer M, Meurette O, et al. Maintenance of embryonic auxin distribution for apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis. Plant Cell. 2005;17(9):2517-2526. doi:10.1105/tpc.105.034637 apa: Weijers, D., Sauer, M., Meurette, O., Friml, J., Ljung, K., Sandberg, G., … Offringa, R. (2005). Maintenance of embryonic auxin distribution for apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis. Plant Cell. American Society of Plant Biologists. https://doi.org/10.1105/tpc.105.034637 chicago: Weijers, Dolf, Michael Sauer, Olivier Meurette, Jiří Friml, Karin Ljung, Göran Sandberg, Paul Hooykaas, and Remko Offringa. “Maintenance of Embryonic Auxin Distribution for Apical Basal Patterning by PIN FORMED Dependent Auxin Transport in Arabidopsis.” Plant Cell. American Society of Plant Biologists, 2005. https://doi.org/10.1105/tpc.105.034637. ieee: D. Weijers et al., “Maintenance of embryonic auxin distribution for apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis,” Plant Cell, vol. 17, no. 9. American Society of Plant Biologists, pp. 2517–2526, 2005. ista: Weijers D, Sauer M, Meurette O, Friml J, Ljung K, Sandberg G, Hooykaas P, Offringa R. 2005. Maintenance of embryonic auxin distribution for apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis. Plant Cell. 17(9), 2517–2526. mla: Weijers, Dolf, et al. “Maintenance of Embryonic Auxin Distribution for Apical Basal Patterning by PIN FORMED Dependent Auxin Transport in Arabidopsis.” Plant Cell, vol. 17, no. 9, American Society of Plant Biologists, 2005, pp. 2517–26, doi:10.1105/tpc.105.034637. short: D. Weijers, M. Sauer, O. Meurette, J. Friml, K. Ljung, G. Sandberg, P. Hooykaas, R. Offringa, Plant Cell 17 (2005) 2517–2526. date_created: 2018-12-11T12:00:48Z date_published: 2005-07-01T00:00:00Z date_updated: 2021-01-12T07:40:24Z day: '01' doi: 10.1105/tpc.105.034637 extern: 1 intvolume: ' 17' issue: '9' month: '07' page: 2517 - 2526 publication: Plant Cell publication_status: published publisher: American Society of Plant Biologists publist_id: '3698' quality_controlled: 0 status: public title: Maintenance of embryonic auxin distribution for apical basal patterning by PIN FORMED dependent auxin transport in Arabidopsis type: journal_article volume: 17 year: '2005' ... --- _id: '3000' abstract: - lang: eng text: In plants, cell polarity is an issue more recurring than in other systems, because plants, due to their adaptive and flexible development, often change cell polarity postembryonically according to intrinsic cues and demands of the environment. Recent findings on the directional movement of the plant signalling molecule auxin provide a unique connection between individual cell polarity and the establishment of polarity at the tissue, organ, and whole-plant levels. Decisions about the subcellular polar targeting of PIN auxin transport components determine the direction of auxin flow between cells and consequently mediate multiple developmental events. In addition, mutations or chemical interference with PIN-based auxin transport result in abnormal cell divisions. Thus, the complicated links between cell polarity establishment, auxin transport, cytoskeleton, and oriented cell divisions now begin to emerge. Here we review the available literature on the issues of cell polarity in both plants and animals to extend our understanding on the generation, maintenance, and transmission of cell polarity in plants. author: - first_name: Pankaj full_name: Dhonukshe, Pankaj last_name: Dhonukshe - first_name: Jürgen full_name: Kleine Vehn, Jürgen last_name: Kleine Vehn - first_name: Jirí full_name: Friml, Jirí id: 4159519E-F248-11E8-B48F-1D18A9856A87 last_name: Friml orcid: 0000-0002-8302-7596 citation: ama: 'Dhonukshe P, Kleine Vehn J, Friml J. Cell polarity, auxin transport and cytoskeleton mediated division planes: Who comes first? Protoplasma. 2005;226(1-2):67-73. doi:10.1007/s00709-005-0104-8' apa: 'Dhonukshe, P., Kleine Vehn, J., & Friml, J. (2005). Cell polarity, auxin transport and cytoskeleton mediated division planes: Who comes first? Protoplasma. Springer. https://doi.org/10.1007/s00709-005-0104-8' chicago: 'Dhonukshe, Pankaj, Jürgen Kleine Vehn, and Jiří Friml. “Cell Polarity, Auxin Transport and Cytoskeleton Mediated Division Planes: Who Comes First?” Protoplasma. Springer, 2005. https://doi.org/10.1007/s00709-005-0104-8.' ieee: 'P. Dhonukshe, J. Kleine Vehn, and J. Friml, “Cell polarity, auxin transport and cytoskeleton mediated division planes: Who comes first?,” Protoplasma, vol. 226, no. 1–2. Springer, pp. 67–73, 2005.' ista: 'Dhonukshe P, Kleine Vehn J, Friml J. 2005. Cell polarity, auxin transport and cytoskeleton mediated division planes: Who comes first? Protoplasma. 226(1–2), 67–73.' mla: 'Dhonukshe, Pankaj, et al. “Cell Polarity, Auxin Transport and Cytoskeleton Mediated Division Planes: Who Comes First?” Protoplasma, vol. 226, no. 1–2, Springer, 2005, pp. 67–73, doi:10.1007/s00709-005-0104-8.' short: P. Dhonukshe, J. Kleine Vehn, J. Friml, Protoplasma 226 (2005) 67–73. date_created: 2018-12-11T12:00:47Z date_published: 2005-10-01T00:00:00Z date_updated: 2021-01-12T07:40:22Z day: '01' doi: 10.1007/s00709-005-0104-8 extern: '1' intvolume: ' 226' issue: 1-2 language: - iso: eng month: '10' oa_version: None page: 67 - 73 publication: Protoplasma publication_status: published publisher: Springer publist_id: '3701' quality_controlled: '1' status: public title: 'Cell polarity, auxin transport and cytoskeleton mediated division planes: Who comes first?' type: journal_article user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87 volume: 226 year: '2005' ... --- _id: '3001' abstract: - lang: eng text: 'One of the mechanisms by which signalling molecules regulate cellular behaviour is modulating subcellular protein translocation. This mode of regulation is often based on specialized vesicle trafficking, termed constitutive cycling, which consists of repeated internalization and recycling of proteins to and from the plasma membrane. No such mechanism of hormone action has been shown in plants although several proteins, including the PIN auxin efflux facilitators, exhibit constitutive cycling. Here we show that a major regulator of plant development, auxin, inhibits endocytosis. This effect is specific to biologically active auxins and requires activity of the Calossin-like protein BIG. By inhibiting the internalization step of PIN constitutive cycling, auxin increases levels of PINs at the plasma membrane. Concomitantly, auxin promotes its own efflux from cells by a vesicle-trafficking-dependent mechanism. Furthermore, asymmetric auxin translocation during gravitropism is correlated with decreased PIN internalization. Our data imply a previously undescribed mode of plant hormone action: by modulating PIN protein trafficking, auxin regulates PIN abundance and activity at the cell surface, providing a mechanism for the feedback regulation of auxin transport.' author: - first_name: Tomasz full_name: Paciorek, Tomasz last_name: Paciorek - first_name: Eva full_name: Zažímalová, Eva last_name: Zažímalová - first_name: Nadia full_name: Ruthardt, Nadia last_name: Ruthardt - first_name: Jan full_name: Petrášek, Jan last_name: Petrášek - first_name: York full_name: Stierhof, York-Dieter last_name: Stierhof - first_name: Jürgen full_name: Kleine-Vehn, Jürgen last_name: Kleine Vehn - first_name: David full_name: Morris, David A last_name: Morris - first_name: Neil full_name: Emans, Neil last_name: Emans - first_name: Gerd full_name: Jürgens, Gerd last_name: Jürgens - first_name: Niko full_name: Geldner, Niko last_name: Geldner - first_name: Jirí full_name: Jirí Friml id: 4159519E-F248-11E8-B48F-1D18A9856A87 last_name: Friml orcid: 0000-0002-8302-7596 citation: ama: Paciorek T, Zažímalová E, Ruthardt N, et al. Auxin inhibits endocytosis and promotes its own efflux from cells. Nature. 2005;435(7046):1251-1256. doi:10.1038/nature03633 apa: Paciorek, T., Zažímalová, E., Ruthardt, N., Petrášek, J., Stierhof, Y., Kleine Vehn, J., … Friml, J. (2005). Auxin inhibits endocytosis and promotes its own efflux from cells. Nature. Nature Publishing Group. https://doi.org/10.1038/nature03633 chicago: Paciorek, Tomasz, Eva Zažímalová, Nadia Ruthardt, Jan Petrášek, York Stierhof, Jürgen Kleine Vehn, David Morris, et al. “Auxin Inhibits Endocytosis and Promotes Its Own Efflux from Cells.” Nature. Nature Publishing Group, 2005. https://doi.org/10.1038/nature03633. ieee: T. Paciorek et al., “Auxin inhibits endocytosis and promotes its own efflux from cells,” Nature, vol. 435, no. 7046. Nature Publishing Group, pp. 1251–1256, 2005. ista: Paciorek T, Zažímalová E, Ruthardt N, Petrášek J, Stierhof Y, Kleine Vehn J, Morris D, Emans N, Jürgens G, Geldner N, Friml J. 2005. Auxin inhibits endocytosis and promotes its own efflux from cells. Nature. 435(7046), 1251–1256. mla: Paciorek, Tomasz, et al. “Auxin Inhibits Endocytosis and Promotes Its Own Efflux from Cells.” Nature, vol. 435, no. 7046, Nature Publishing Group, 2005, pp. 1251–56, doi:10.1038/nature03633. short: T. Paciorek, E. Zažímalová, N. Ruthardt, J. Petrášek, Y. Stierhof, J. Kleine Vehn, D. Morris, N. Emans, G. Jürgens, N. Geldner, J. Friml, Nature 435 (2005) 1251–1256. date_created: 2018-12-11T12:00:47Z date_published: 2005-06-30T00:00:00Z date_updated: 2021-01-12T07:40:23Z day: '30' doi: 10.1038/nature03633 extern: 1 intvolume: ' 435' issue: '7046' month: '06' page: 1251 - 1256 publication: Nature publication_status: published publisher: Nature Publishing Group publist_id: '3702' quality_controlled: 0 status: public title: Auxin inhibits endocytosis and promotes its own efflux from cells type: journal_article volume: 435 year: '2005' ... --- _id: '3003' abstract: - lang: eng text: 'Plant development displays an exceptional plasticity and adaptability that involves the dynamic, asymmetric distribution of the phytohormone auxin. Polar auxin flow, which requires polarly localized transport facilitators of the PIN family, largely contributes to the establishment and maintenance of the auxin gradients. Functionally overlapping action of PIN proteins mediates multiple developmental processes, including embryo formation, organ development and tropisms. Here we show that PIN proteins exhibit synergistic interactions, which involve cross-regulation of PIN gene expression in pin mutants or plants with inhibited auxin transport. Auxin itself positively feeds back on PIN gene expression in a tissue-specific manner through an AUX/IAA-dependent signalling pathway. This regulatory switch is indicative of a mechanism by which the loss of a specific PIN protein is compensated for by auxin-dependent ectopic: expression of its homologues. The compensatory properties of the PIN-dependent transport network might enable the stabilization of auxin gradients and potentially contribute to the robustness of plant adaptive development.' author: - first_name: Anne full_name: Vieten, Anne last_name: Vieten - first_name: Steffen full_name: Vanneste, Steffen last_name: Vanneste - first_name: Justyna full_name: Wiśniewska, Justyna last_name: Wiśniewska - first_name: Eva full_name: Eva Benková id: 38F4F166-F248-11E8-B48F-1D18A9856A87 last_name: Benková orcid: 0000-0002-8510-9739 - first_name: René full_name: Benjamins, René last_name: Benjamins - first_name: Tom full_name: Beeckman, Tom last_name: Beeckman - first_name: Christian full_name: Luschnig, Christian last_name: Luschnig - first_name: Jirí full_name: Jirí Friml id: 4159519E-F248-11E8-B48F-1D18A9856A87 last_name: Friml orcid: 0000-0002-8302-7596 citation: ama: Vieten A, Vanneste S, Wiśniewska J, et al. Functional redundancy of PIN proteins is accompanied by auxin-dependent cross-regulation of PIN expression. Development. 2005;132(20):4521-4531. doi:10.1242/dev.02027 apa: Vieten, A., Vanneste, S., Wiśniewska, J., Benková, E., Benjamins, R., Beeckman, T., … Friml, J. (2005). Functional redundancy of PIN proteins is accompanied by auxin-dependent cross-regulation of PIN expression. Development. Company of Biologists. https://doi.org/10.1242/dev.02027 chicago: Vieten, Anne, Steffen Vanneste, Justyna Wiśniewska, Eva Benková, René Benjamins, Tom Beeckman, Christian Luschnig, and Jiří Friml. “Functional Redundancy of PIN Proteins Is Accompanied by Auxin-Dependent Cross-Regulation of PIN Expression.” Development. Company of Biologists, 2005. https://doi.org/10.1242/dev.02027. ieee: A. Vieten et al., “Functional redundancy of PIN proteins is accompanied by auxin-dependent cross-regulation of PIN expression,” Development, vol. 132, no. 20. Company of Biologists, pp. 4521–4531, 2005. ista: Vieten A, Vanneste S, Wiśniewska J, Benková E, Benjamins R, Beeckman T, Luschnig C, Friml J. 2005. Functional redundancy of PIN proteins is accompanied by auxin-dependent cross-regulation of PIN expression. Development. 132(20), 4521–4531. mla: Vieten, Anne, et al. “Functional Redundancy of PIN Proteins Is Accompanied by Auxin-Dependent Cross-Regulation of PIN Expression.” Development, vol. 132, no. 20, Company of Biologists, 2005, pp. 4521–31, doi:10.1242/dev.02027. short: A. Vieten, S. Vanneste, J. Wiśniewska, E. Benková, R. Benjamins, T. Beeckman, C. Luschnig, J. Friml, Development 132 (2005) 4521–4531. date_created: 2018-12-11T12:00:48Z date_published: 2005-10-01T00:00:00Z date_updated: 2021-01-12T07:40:23Z day: '01' doi: 10.1242/dev.02027 extern: 1 intvolume: ' 132' issue: '20' month: '10' page: 4521 - 4531 publication: Development publication_status: published publisher: Company of Biologists publist_id: '3700' quality_controlled: 0 status: public title: Functional redundancy of PIN proteins is accompanied by auxin-dependent cross-regulation of PIN expression type: journal_article volume: 132 year: '2005' ... --- _id: '3212' abstract: - lang: eng text: |- The Full-Domain Hash (FDH) signature scheme [3] forms one the most basic usages of random oracles. It works with a family F of trapdoor permutations (TDP), where the signature of m is computed as f−1(h(m)) (here f ∈R F and h is modelled as a random oracle). It is known to be existentially unforgeable for any TDP family F [3], although a much tighter security reduction is known for a restrictive class of TDP’s [10,14] — namely, those induced by a family of claw-free permutations (CFP) pairs. The latter result was shown [11] to match the best possible “black-box” security reduction in the random oracle model, irrespective of the TDP family F (e.g., RSA) one might use. In this work we investigate the question if it is possible to instantiate the random oracle h with a “real” family of hash functions H such that the corresponding schemes can be proven secure in the standard model, under some natural assumption on the family F. Our main result rules out the existence of such instantiations for any assumption on F which (1) is satisfied by a family of random permutations; and (2) does not allow the attacker to invert f ∈R F on an a-priori unbounded number of points. Moreover, this holds even if the choice of H can arbitrarily depend on f. As an immediate corollary, we rule out instantiating FDH based on general claw-free permutations, which shows that in order to prove the security of FDH in the standard model one must utilize significantly more structure on F than what is sufficient for the best proof of security in the random oracle model. acknowledgement: Supported by NSF CAREER Award CCR-0133806 and TC Grant No.CCR-0311095. Supported by the Swiss National Science Foundation, project No. 200020-103847/1 alternative_title: - LNCS author: - first_name: Yevgeniy full_name: Dodis, Yevgeniy last_name: Dodis - first_name: Roberto full_name: Oliveira, Roberto last_name: Oliveira - first_name: Krzysztof Z full_name: Krzysztof Pietrzak id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87 last_name: Pietrzak orcid: 0000-0002-9139-1654 citation: ama: 'Dodis Y, Oliveira R, Pietrzak KZ. On the generic insecurity of the full domain hash. In: Vol 3621. Springer; 2005:449-466. doi:10.1007/11535218_27' apa: 'Dodis, Y., Oliveira, R., & Pietrzak, K. Z. (2005). On the generic insecurity of the full domain hash (Vol. 3621, pp. 449–466). Presented at the CRYPTO: International Cryptology Conference, Springer. https://doi.org/10.1007/11535218_27' chicago: Dodis, Yevgeniy, Roberto Oliveira, and Krzysztof Z Pietrzak. “On the Generic Insecurity of the Full Domain Hash,” 3621:449–66. Springer, 2005. https://doi.org/10.1007/11535218_27. ieee: 'Y. Dodis, R. Oliveira, and K. Z. Pietrzak, “On the generic insecurity of the full domain hash,” presented at the CRYPTO: International Cryptology Conference, 2005, vol. 3621, pp. 449–466.' ista: 'Dodis Y, Oliveira R, Pietrzak KZ. 2005. On the generic insecurity of the full domain hash. CRYPTO: International Cryptology Conference, LNCS, vol. 3621, 449–466.' mla: Dodis, Yevgeniy, et al. On the Generic Insecurity of the Full Domain Hash. Vol. 3621, Springer, 2005, pp. 449–66, doi:10.1007/11535218_27. short: Y. Dodis, R. Oliveira, K.Z. Pietrzak, in:, Springer, 2005, pp. 449–466. conference: name: 'CRYPTO: International Cryptology Conference' date_created: 2018-12-11T12:02:03Z date_published: 2005-09-12T00:00:00Z date_updated: 2021-01-12T07:41:50Z day: '12' doi: 10.1007/11535218_27 extern: 1 intvolume: ' 3621' month: '09' page: 449 - 466 publication_status: published publisher: Springer publist_id: '3470' quality_controlled: 0 status: public title: On the generic insecurity of the full domain hash type: conference volume: 3621 year: '2005' ... --- _id: '3213' abstract: - lang: eng text: |- We study the question whether the sequential or parallel composition of two functions, each indistinguishable from a random function by non-adaptive distinguishers is secure against adaptive distinguishers. The sequential composition of F and G is the function G(F()), the parallel composition is F G where ⋆ is some group operation. It has been shown that composition indeed gives adaptive security in the information theoretic setting, but unfortunately the proof does not translate into the more interesting computational case. In this work we show that in the computational setting composition does not imply adaptive security: If there is a prime order cyclic group where the decisional Diffie-Hellman assumption holds, then there are functions F and G which are indistinguishable by non-adaptive polynomially time-bounded adversaries, but whose parallel composition can be completely broken (i.e. we recover the key) with only three adaptive queries. We give a similar result for sequential composition. Interestingly, we need a standard assumption from the asymmetric (aka. public-key) world to prove a negative result for symmetric (aka. private-key) systems. acknowledgement: Supported by the Swiss National Science Foundation, project No. 200020-103847/1. alternative_title: - LNCS author: - first_name: Krzysztof Z full_name: Krzysztof Pietrzak id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87 last_name: Pietrzak orcid: 0000-0002-9139-1654 citation: ama: 'Pietrzak KZ. Composition does not imply adaptive security. In: Vol 3621. Springer; 2005:55-65. doi:10.1007/11535218_4' apa: 'Pietrzak, K. Z. (2005). Composition does not imply adaptive security (Vol. 3621, pp. 55–65). Presented at the CRYPTO: International Cryptology Conference, Springer. https://doi.org/10.1007/11535218_4' chicago: Pietrzak, Krzysztof Z. “Composition Does Not Imply Adaptive Security,” 3621:55–65. Springer, 2005. https://doi.org/10.1007/11535218_4. ieee: 'K. Z. Pietrzak, “Composition does not imply adaptive security,” presented at the CRYPTO: International Cryptology Conference, 2005, vol. 3621, pp. 55–65.' ista: 'Pietrzak KZ. 2005. Composition does not imply adaptive security. CRYPTO: International Cryptology Conference, LNCS, vol. 3621, 55–65.' mla: Pietrzak, Krzysztof Z. Composition Does Not Imply Adaptive Security. Vol. 3621, Springer, 2005, pp. 55–65, doi:10.1007/11535218_4. short: K.Z. Pietrzak, in:, Springer, 2005, pp. 55–65. conference: name: 'CRYPTO: International Cryptology Conference' date_created: 2018-12-11T12:02:03Z date_published: 2005-09-12T00:00:00Z date_updated: 2021-01-12T07:41:50Z day: '12' doi: 10.1007/11535218_4 extern: 1 intvolume: ' 3621' month: '09' page: 55 - 65 publication_status: published publisher: Springer publist_id: '3468' quality_controlled: 0 status: public title: Composition does not imply adaptive security type: conference volume: 3621 year: '2005' ... --- _id: '3211' abstract: - lang: eng text: We present an improved bound on the advantage of any q-query adversary at distinguishing between the CBC MAC over a random n-bit permutation and a random function outputting n bits. The result assumes that no message queried is a prefix of any other, as is the case when all messages to be MACed have the same length. We go on to give an improved analysis of the encrypted CBC MAC, where there is no restriction on queried messages. Letting m be the block length of the longest query, our bounds are about mq2/2n for the basic CBC MAC and mo(1)q2/2n for the encrypted CBC MAC, improving prior bounds of m2q2/2n. The new bounds translate into improved guarantees on the probability of forging these MACs. acknowledgement: Pietrzak was supported by the Swiss National Science Foundation, project No. 200020-103847/1. alternative_title: - LNCS author: - first_name: Mihir full_name: Bellare, Mihir last_name: Bellare - first_name: Krzysztof Z full_name: Krzysztof Pietrzak id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87 last_name: Pietrzak orcid: 0000-0002-9139-1654 - first_name: Phillip full_name: Rogaway, Phillip last_name: Rogaway citation: ama: 'Bellare M, Pietrzak KZ, Rogaway P. Improved security analyses for CBC MACs. In: Vol 3621. Springer; 2005:527-545. doi:10.1007/11535218_32' apa: 'Bellare, M., Pietrzak, K. Z., & Rogaway, P. (2005). Improved security analyses for CBC MACs (Vol. 3621, pp. 527–545). Presented at the CRYPTO: International Cryptology Conference, Springer. https://doi.org/10.1007/11535218_32' chicago: Bellare, Mihir, Krzysztof Z Pietrzak, and Phillip Rogaway. “Improved Security Analyses for CBC MACs,” 3621:527–45. Springer, 2005. https://doi.org/10.1007/11535218_32. ieee: 'M. Bellare, K. Z. Pietrzak, and P. Rogaway, “Improved security analyses for CBC MACs,” presented at the CRYPTO: International Cryptology Conference, 2005, vol. 3621, pp. 527–545.' ista: 'Bellare M, Pietrzak KZ, Rogaway P. 2005. Improved security analyses for CBC MACs. CRYPTO: International Cryptology Conference, LNCS, vol. 3621, 527–545.' mla: Bellare, Mihir, et al. Improved Security Analyses for CBC MACs. Vol. 3621, Springer, 2005, pp. 527–45, doi:10.1007/11535218_32. short: M. Bellare, K.Z. Pietrzak, P. Rogaway, in:, Springer, 2005, pp. 527–545. conference: name: 'CRYPTO: International Cryptology Conference' date_created: 2018-12-11T12:02:02Z date_published: 2005-09-12T00:00:00Z date_updated: 2021-01-12T07:41:50Z day: '12' doi: 10.1007/11535218_32 extern: 1 intvolume: ' 3621' month: '09' page: 527 - 545 publication_status: published publisher: Springer publist_id: '3469' quality_controlled: 0 status: public title: Improved security analyses for CBC MACs type: conference volume: 3621 year: '2005' ... --- _id: '3426' abstract: - lang: eng text: We discuss the formation of graded morphogen profiles in a cell layer by nonlinear transport phenomena, important for patterning developing organisms. We focus on a process termed transcytosis, where morphogen transport results from the binding of ligands to receptors on the cell surface, incorporation into the cell, and subsequent externalization. Starting from a microscopic model, we derive effective transport equations. We show that, in contrast to morphogen transport by extracellular diffusion, transcytosis leads to robust ligand profiles which are insensitive to the rate of ligand production. article_processing_charge: No author: - first_name: Mark Tobias full_name: Bollenbach, Mark Tobias id: 3E6DB97A-F248-11E8-B48F-1D18A9856A87 last_name: Bollenbach orcid: 0000-0003-4398-476X - first_name: Karsten full_name: Kruse, Karsten last_name: Kruse - first_name: Periklis full_name: Pantazis, Periklis last_name: Pantazis - first_name: Marcos full_name: González Gaitán, Marcos last_name: González Gaitán - first_name: Frank full_name: Jülicher, Frank last_name: Jülicher citation: ama: Bollenbach MT, Kruse K, Pantazis P, González Gaitán M, Jülicher F. Robust formation of morphogen gradients. Physical Review Letters. 2005;94(1). doi:10.1103/PhysRevLett.94.018103 apa: Bollenbach, M. T., Kruse, K., Pantazis, P., González Gaitán, M., & Jülicher, F. (2005). Robust formation of morphogen gradients. Physical Review Letters. American Physical Society. https://doi.org/10.1103/PhysRevLett.94.018103 chicago: Bollenbach, Mark Tobias, Karsten Kruse, Periklis Pantazis, Marcos González Gaitán, and Frank Jülicher. “Robust Formation of Morphogen Gradients.” Physical Review Letters. American Physical Society, 2005. https://doi.org/10.1103/PhysRevLett.94.018103. ieee: M. T. Bollenbach, K. Kruse, P. Pantazis, M. González Gaitán, and F. Jülicher, “Robust formation of morphogen gradients,” Physical Review Letters, vol. 94, no. 1. American Physical Society, 2005. ista: Bollenbach MT, Kruse K, Pantazis P, González Gaitán M, Jülicher F. 2005. Robust formation of morphogen gradients. Physical Review Letters. 94(1). mla: Bollenbach, Mark Tobias, et al. “Robust Formation of Morphogen Gradients.” Physical Review Letters, vol. 94, no. 1, American Physical Society, 2005, doi:10.1103/PhysRevLett.94.018103. short: M.T. Bollenbach, K. Kruse, P. Pantazis, M. González Gaitán, F. Jülicher, Physical Review Letters 94 (2005). date_created: 2018-12-11T12:03:16Z date_published: 2005-01-01T00:00:00Z date_updated: 2021-01-12T07:43:23Z day: '01' doi: 10.1103/PhysRevLett.94.018103 extern: '1' external_id: arxiv: - q-bio/0412014 intvolume: ' 94' issue: '1' language: - iso: eng main_file_link: - open_access: '1' url: http://arxiv.org/abs/q-bio/0412014 month: '01' oa: 1 oa_version: Preprint publication: Physical Review Letters publication_status: published publisher: American Physical Society publist_id: '2975' status: public title: Robust formation of morphogen gradients type: journal_article user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87 volume: 94 year: '2005' ... --- _id: '3443' abstract: - lang: eng text: In the hippocampal CA1 area, a relatively homogenous population of pyramidal cells is accompanied by a diversity of GABAergic interneurons. Previously, we found that parvalbumin-expressing basket, axo-axonic, bistratified, and oriens-lacunosum moleculare cells, innervating different domains of pyramidal cells, have distinct firing patterns during network oscillations in vivo. A second family of interneurons, expressing cholecystokinin but not parvalbumin, is known to target the same domains of pyramidal cells as do the parvalbumin cells. To test the temporal activity of these independent and parallel GABAergic inputs, we recorded the precise spike timing of identified cholecystokinin interneurons during hippocampal network oscillations in anesthetized rats and determined their molecular expression profiles and synaptic targets. The cells were cannabinoid receptor type 1 immunopositive. Contrary to the stereotyped firing of parvalbumin interneurons, cholecystokinin-expressing basket and dendrite-innervating cells discharge, on average, with 1.7 ± 2.0 Hz during high-frequency ripple oscillations in an episode-dependent manner. During theta oscillations, cholecystokinin- expressing interneurons fire with 8.8 ± 3.3 Hz at a characteristic time on the ascending phase of theta waves (155 ± 81°), when place cells start firing in freely moving animals. The firing patterns of some interneurons recorded in drug-free behaving rats were similar to cholecystokinin cells in anesthetized animals. Our results demonstrate that cholecystokinin- and parvalbumin-expressing interneurons make different contributions to network oscillations and play distinct roles in different brain states. We suggest that the specific spike timing of cholecystokinin interneurons and their sensitivity to endocannabinoids might contribute to differentiate subgroups of pyramidal cells forming neuronal assemblies, whereas parvalbumin interneurons contribute to synchronizing the entire network. Copyright © 2005 Society for Neuroscience. author: - first_name: Thomas full_name: Klausberger,Thomas last_name: Klausberger - first_name: Laszlo full_name: Marton,Laszlo F last_name: Marton - first_name: Joseph full_name: Joseph O'Neill id: 426376DC-F248-11E8-B48F-1D18A9856A87 last_name: O'Neill - first_name: Jojanneke full_name: Huck, Jojanneke H last_name: Huck - first_name: Yannis full_name: Dalezios, Yannis last_name: Dalezios - first_name: Pablo full_name: Fuentealba,Pablo last_name: Fuentealba - first_name: Wai full_name: Suen, Wai Yee last_name: Suen - first_name: Edit full_name: Papp, Edit Cs last_name: Papp - first_name: Takeshi full_name: Kaneko, Takeshi last_name: Kaneko - first_name: Masahiko full_name: Watanabe, Masahiko last_name: Watanabe - first_name: Jozsef L full_name: Jozsef Csicsvari id: 3FA14672-F248-11E8-B48F-1D18A9856A87 last_name: Csicsvari orcid: 0000-0002-5193-4036 - first_name: Péter full_name: Somogyi, Péter last_name: Somogyi citation: ama: Klausberger T, Marton L, O’Neill J, et al. Complementary roles of cholecystokinin- and parvalbumin-expressing GABAergic neurons in hippocampal network oscillations. Journal of Neuroscience. 2005;25(42):9782-9793. doi:10.1523/JNEUROSCI.3269-05.2005 apa: Klausberger, T., Marton, L., O’Neill, J., Huck, J., Dalezios, Y., Fuentealba, P., … Somogyi, P. (2005). Complementary roles of cholecystokinin- and parvalbumin-expressing GABAergic neurons in hippocampal network oscillations. Journal of Neuroscience. Society for Neuroscience. https://doi.org/10.1523/JNEUROSCI.3269-05.2005 chicago: Klausberger, Thomas, Laszlo Marton, Joseph O’Neill, Jojanneke Huck, Yannis Dalezios, Pablo Fuentealba, Wai Suen, et al. “Complementary Roles of Cholecystokinin- and Parvalbumin-Expressing GABAergic Neurons in Hippocampal Network Oscillations.” Journal of Neuroscience. Society for Neuroscience, 2005. https://doi.org/10.1523/JNEUROSCI.3269-05.2005. ieee: T. Klausberger et al., “Complementary roles of cholecystokinin- and parvalbumin-expressing GABAergic neurons in hippocampal network oscillations,” Journal of Neuroscience, vol. 25, no. 42. Society for Neuroscience, pp. 9782–9793, 2005. ista: Klausberger T, Marton L, O’Neill J, Huck J, Dalezios Y, Fuentealba P, Suen W, Papp E, Kaneko T, Watanabe M, Csicsvari JL, Somogyi P. 2005. Complementary roles of cholecystokinin- and parvalbumin-expressing GABAergic neurons in hippocampal network oscillations. Journal of Neuroscience. 25(42), 9782–9793. mla: Klausberger, Thomas, et al. “Complementary Roles of Cholecystokinin- and Parvalbumin-Expressing GABAergic Neurons in Hippocampal Network Oscillations.” Journal of Neuroscience, vol. 25, no. 42, Society for Neuroscience, 2005, pp. 9782–93, doi:10.1523/JNEUROSCI.3269-05.2005. short: T. Klausberger, L. Marton, J. O’Neill, J. Huck, Y. Dalezios, P. Fuentealba, W. Suen, E. Papp, T. Kaneko, M. Watanabe, J.L. Csicsvari, P. Somogyi, Journal of Neuroscience 25 (2005) 9782–9793. date_created: 2018-12-11T12:03:21Z date_published: 2005-10-19T00:00:00Z date_updated: 2021-01-12T07:43:30Z day: '19' doi: 10.1523/JNEUROSCI.3269-05.2005 extern: 1 intvolume: ' 25' issue: '42' month: '10' page: 9782 - 9793 publication: Journal of Neuroscience publication_status: published publisher: Society for Neuroscience publist_id: '2944' quality_controlled: 0 status: public title: Complementary roles of cholecystokinin- and parvalbumin-expressing GABAergic neurons in hippocampal network oscillations type: journal_article volume: 25 year: '2005' ... --- _id: '3557' abstract: - lang: eng text: A challenging problem in computer-aided geometric design is the decomposition of a surface into four-sided regions that are then represented by NURBS patches. There are various approaches published in the literature and implemented as commercially available software, but all fall short in either automation or quality of the result. At Raindrop Geomagic, we have recently taken a fresh approach based on concepts from Morse theory. This by itself is not a new idea, but we have some novel ingredients that make this work, one being a rational notion of hierarchy that guides the construction of a simplified decomposition sensitive to only the major critical points. author: - first_name: Herbert full_name: Herbert Edelsbrunner id: 3FB178DA-F248-11E8-B48F-1D18A9856A87 last_name: Edelsbrunner orcid: 0000-0002-9823-6833 citation: ama: 'Edelsbrunner H. Surface tiling with differential topology. In: ACM; 2005:9-11. doi:http://dx.doi.org/10.2312/SGP/SGP05/009-011' apa: 'Edelsbrunner, H. (2005). Surface tiling with differential topology (pp. 9–11). Presented at the SGP: Eurographics Symposium on Geometry processing, ACM. http://dx.doi.org/10.2312/SGP/SGP05/009-011' chicago: Edelsbrunner, Herbert. “Surface Tiling with Differential Topology,” 9–11. ACM, 2005. http://dx.doi.org/10.2312/SGP/SGP05/009-011. ieee: 'H. Edelsbrunner, “Surface tiling with differential topology,” presented at the SGP: Eurographics Symposium on Geometry processing, 2005, pp. 9–11.' ista: 'Edelsbrunner H. 2005. Surface tiling with differential topology. SGP: Eurographics Symposium on Geometry processing, 9–11.' mla: Edelsbrunner, Herbert. Surface Tiling with Differential Topology. ACM, 2005, pp. 9–11, doi:http://dx.doi.org/10.2312/SGP/SGP05/009-011. short: H. Edelsbrunner, in:, ACM, 2005, pp. 9–11. conference: name: 'SGP: Eurographics Symposium on Geometry processing' date_created: 2018-12-11T12:03:57Z date_published: 2005-07-01T00:00:00Z date_updated: 2021-01-12T07:44:17Z day: '01' doi: http://dx.doi.org/10.2312/SGP/SGP05/009-011 extern: 1 main_file_link: - open_access: '0' url: http://www.cs.duke.edu/~edels/Papers/2005-P-03-SurfaceTiling.pdf month: '07' page: 9 - 11 publication_status: published publisher: ACM publist_id: '2828' quality_controlled: 0 status: public title: Surface tiling with differential topology type: conference year: '2005' ...