---
_id: '2358'
abstract:
- lang: eng
text: A study was conducted on the one-dimensional (1D) bosons in three-dimensional
(3D) traps. A rigorous analysis was carried out on the parameter regions in which
various types of 1D or 3D behavior occurred in the ground state. The four parameter
regions include density, transverse, longitudinal dimensions and scattering length.
author:
- first_name: Élliott
full_name: Lieb, Élliott H
last_name: Lieb
- first_name: Robert
full_name: Robert Seiringer
id: 4AFD0470-F248-11E8-B48F-1D18A9856A87
last_name: Seiringer
orcid: 0000-0002-6781-0521
- first_name: Jakob
full_name: Yngvason, Jakob
last_name: Yngvason
citation:
ama: Lieb É, Seiringer R, Yngvason J. One-dimensional Bosons in three-dimensional
traps. Physical Review Letters. 2003;91(15):1504011-1504014. doi:10.1103/PhysRevLett.91.150401
apa: Lieb, É., Seiringer, R., & Yngvason, J. (2003). One-dimensional Bosons
in three-dimensional traps. Physical Review Letters. American Physical
Society. https://doi.org/10.1103/PhysRevLett.91.150401
chicago: Lieb, Élliott, Robert Seiringer, and Jakob Yngvason. “One-Dimensional Bosons
in Three-Dimensional Traps.” Physical Review Letters. American Physical
Society, 2003. https://doi.org/10.1103/PhysRevLett.91.150401.
ieee: É. Lieb, R. Seiringer, and J. Yngvason, “One-dimensional Bosons in three-dimensional
traps,” Physical Review Letters, vol. 91, no. 15. American Physical Society,
pp. 1504011–1504014, 2003.
ista: Lieb É, Seiringer R, Yngvason J. 2003. One-dimensional Bosons in three-dimensional
traps. Physical Review Letters. 91(15), 1504011–1504014.
mla: Lieb, Élliott, et al. “One-Dimensional Bosons in Three-Dimensional Traps.”
Physical Review Letters, vol. 91, no. 15, American Physical Society, 2003,
pp. 1504011–14, doi:10.1103/PhysRevLett.91.150401.
short: É. Lieb, R. Seiringer, J. Yngvason, Physical Review Letters 91 (2003) 1504011–1504014.
date_created: 2018-12-11T11:57:12Z
date_published: 2003-10-10T00:00:00Z
date_updated: 2021-01-12T06:57:00Z
day: '10'
doi: 10.1103/PhysRevLett.91.150401
extern: 1
intvolume: ' 91'
issue: '15'
main_file_link:
- open_access: '1'
url: http://arxiv.org/abs/cond-mat/0304071
month: '10'
oa: 1
page: 1504011 - 1504014
publication: Physical Review Letters
publication_status: published
publisher: American Physical Society
publist_id: '4571'
quality_controlled: 0
status: public
title: One-dimensional Bosons in three-dimensional traps
type: journal_article
volume: 91
year: '2003'
...
---
_id: '2414'
author:
- first_name: Uli
full_name: Uli Wagner
id: 36690CA2-F248-11E8-B48F-1D18A9856A87
last_name: Wagner
orcid: 0000-0002-1494-0568
citation:
ama: Wagner U. On k-Sets and Their Applications. 2003. doi:10.3929/ethz-a-004708408
apa: Wagner, U. (2003). On k-Sets and Their Applications. ETH Zurich. https://doi.org/10.3929/ethz-a-004708408
chicago: Wagner, Uli. “On K-Sets and Their Applications.” ETH Zurich, 2003. https://doi.org/10.3929/ethz-a-004708408.
ieee: U. Wagner, “On k-Sets and Their Applications,” ETH Zurich, 2003.
ista: Wagner U. 2003. On k-Sets and Their Applications. ETH Zurich.
mla: Wagner, Uli. On K-Sets and Their Applications. ETH Zurich, 2003, doi:10.3929/ethz-a-004708408.
short: U. Wagner, On K-Sets and Their Applications, ETH Zurich, 2003.
date_created: 2018-12-11T11:57:31Z
date_published: 2003-01-01T00:00:00Z
date_updated: 2021-01-12T06:57:20Z
day: '01'
doi: 10.3929/ethz-a-004708408
extern: 1
month: '01'
publication_status: published
publisher: ETH Zurich
publist_id: '4511'
quality_controlled: 0
status: public
title: On k-Sets and Their Applications
type: dissertation
year: '2003'
...
---
_id: '2424'
abstract:
- lang: eng
text: We introduce the adaptive neighborhood graph as a data structure for modeling
a smooth manifold M embedded in some (potentially very high-dimensional) Euclidean
space ℝd. We assume that M is known to us only through a finite sample P ⊂ M,
as it is often the case in applications. The adaptive neighborhood graph is a
geometric graph on P. Its complexity is at most min{2O(k)(n, n2}, where n = |P|
and k = dim M, as opposed to the n⌈d/2⌉ complexity of the Delaunay triangulation,
which is often used to model manifolds. We show that we can provably correctly
infer the connectivity of M and the dimension of M from the adaptive neighborhood
graph provided a certain standard sampling condition is fulfilled. The running
time of the dimension detection algorithm is d2O(k7 log k) for each connected
component of M. If the dimension is considered constant, this is a constant-time
operation, and the adaptive neighborhood graph is of linear size. Moreover, the
exponential dependence of the constants is only on the intrinsic dimension k,
not on the ambient dimension d. This is of particular interest if the co-dimension
is high, i.e., if k is much smaller than d, as is the case in many applications.
The adaptive neighborhood graph also allows us to approximate the geodesic distances
between the points in P.
author:
- first_name: Joachim
full_name: Giesen, Joachim
last_name: Giesen
- first_name: Uli
full_name: Uli Wagner
id: 36690CA2-F248-11E8-B48F-1D18A9856A87
last_name: Wagner
orcid: 0000-0002-1494-0568
citation:
ama: 'Giesen J, Wagner U. Shape dimension and intrinsic metric from samples of manifolds
with high co-dimension. In: ACM; 2003:329-337. doi:10.1145/777792.777841'
apa: 'Giesen, J., & Wagner, U. (2003). Shape dimension and intrinsic metric
from samples of manifolds with high co-dimension (pp. 329–337). Presented at the
SoCG: Symposium on Computational Geometry, ACM. https://doi.org/10.1145/777792.777841'
chicago: Giesen, Joachim, and Uli Wagner. “Shape Dimension and Intrinsic Metric
from Samples of Manifolds with High Co-Dimension,” 329–37. ACM, 2003. https://doi.org/10.1145/777792.777841.
ieee: 'J. Giesen and U. Wagner, “Shape dimension and intrinsic metric from samples
of manifolds with high co-dimension,” presented at the SoCG: Symposium on Computational
Geometry, 2003, pp. 329–337.'
ista: 'Giesen J, Wagner U. 2003. Shape dimension and intrinsic metric from samples
of manifolds with high co-dimension. SoCG: Symposium on Computational Geometry,
329–337.'
mla: Giesen, Joachim, and Uli Wagner. Shape Dimension and Intrinsic Metric from
Samples of Manifolds with High Co-Dimension. ACM, 2003, pp. 329–37, doi:10.1145/777792.777841.
short: J. Giesen, U. Wagner, in:, ACM, 2003, pp. 329–337.
conference:
name: 'SoCG: Symposium on Computational Geometry'
date_created: 2018-12-11T11:57:35Z
date_published: 2003-06-01T00:00:00Z
date_updated: 2021-01-12T06:57:24Z
day: '01'
doi: 10.1145/777792.777841
extern: 1
month: '06'
page: 329 - 337
publication_status: published
publisher: ACM
publist_id: '4501'
quality_controlled: 0
status: public
title: Shape dimension and intrinsic metric from samples of manifolds with high co-dimension
type: conference
year: '2003'
...
---
_id: '2423'
abstract:
- lang: eng
text: A finite set N ⊃ Rd is a weak ε-net for an n-point set X ⊃ Rd (with respect
to convex sets) if N intersects every convex set K with |K ∩ X| ≥ εn. We give
an alternative, and arguably simpler, proof of the fact, first shown by Chazelle
et al. [7], that every point set X in Rd admits a weak ε-net of cardinality O(ε-d
polylog(1/ε)). Moreover, for a number of special point sets (e.g., for points
on the moment curve), our method gives substantially better bounds. The construction
yields an algorithm to construct such weak ε-nets in time O(n ln(1/ε)). We also
prove, by a different method, a near-linear upper bound for points uniformly distributed
on the (d - 1)-dimensional sphere.
author:
- first_name: Jiří
full_name: Matoušek, Jiří
last_name: Matoušek
- first_name: Uli
full_name: Uli Wagner
id: 36690CA2-F248-11E8-B48F-1D18A9856A87
last_name: Wagner
orcid: 0000-0002-1494-0568
citation:
ama: 'Matoušek J, Wagner U. New constructions of weak epsilon-nets. In: ACM; 2003:129-135.
doi:10.1145/777792.777813'
apa: 'Matoušek, J., & Wagner, U. (2003). New constructions of weak epsilon-nets
(pp. 129–135). Presented at the SoCG: Symposium on Computational Geometry, ACM.
https://doi.org/10.1145/777792.777813'
chicago: Matoušek, Jiří, and Uli Wagner. “New Constructions of Weak Epsilon-Nets,”
129–35. ACM, 2003. https://doi.org/10.1145/777792.777813.
ieee: 'J. Matoušek and U. Wagner, “New constructions of weak epsilon-nets,” presented
at the SoCG: Symposium on Computational Geometry, 2003, pp. 129–135.'
ista: 'Matoušek J, Wagner U. 2003. New constructions of weak epsilon-nets. SoCG:
Symposium on Computational Geometry, 129–135.'
mla: Matoušek, Jiří, and Uli Wagner. New Constructions of Weak Epsilon-Nets.
ACM, 2003, pp. 129–35, doi:10.1145/777792.777813.
short: J. Matoušek, U. Wagner, in:, ACM, 2003, pp. 129–135.
conference:
name: 'SoCG: Symposium on Computational Geometry'
date_created: 2018-12-11T11:57:34Z
date_published: 2003-06-01T00:00:00Z
date_updated: 2021-01-12T06:57:24Z
day: '01'
doi: 10.1145/777792.777813
extern: 1
month: '06'
page: 129 - 135
publication_status: published
publisher: ACM
publist_id: '4502'
quality_controlled: 0
status: public
title: New constructions of weak epsilon-nets
type: conference
year: '2003'
...
---
_id: '2422'
abstract:
- lang: eng
text: We prove a lower bound of 0.3288(4 n) for the rectilinear crossing number
cr̄(Kn) of a complete graph on n vertices, or in other words, for the minimum
number of convex quadrilaterals in any set of n points in general position in
the Euclidean plane. As we see it, the main contribution of this paper is not
so much the concrete numerical improvement over earlier bounds, as the novel method
of proof, which is not based on bounding cr̄(Kn) for some small n.
author:
- first_name: Uli
full_name: Uli Wagner
id: 36690CA2-F248-11E8-B48F-1D18A9856A87
last_name: Wagner
orcid: 0000-0002-1494-0568
citation:
ama: 'Wagner U. On the rectilinear crossing number of complete graphs. In: SIAM;
2003:583-588.'
apa: 'Wagner, U. (2003). On the rectilinear crossing number of complete graphs (pp.
583–588). Presented at the SODA: Symposium on Discrete Algorithms, SIAM.'
chicago: Wagner, Uli. “On the Rectilinear Crossing Number of Complete Graphs,” 583–88.
SIAM, 2003.
ieee: 'U. Wagner, “On the rectilinear crossing number of complete graphs,” presented
at the SODA: Symposium on Discrete Algorithms, 2003, pp. 583–588.'
ista: 'Wagner U. 2003. On the rectilinear crossing number of complete graphs. SODA:
Symposium on Discrete Algorithms, 583–588.'
mla: Wagner, Uli. On the Rectilinear Crossing Number of Complete Graphs.
SIAM, 2003, pp. 583–88.
short: U. Wagner, in:, SIAM, 2003, pp. 583–588.
conference:
name: 'SODA: Symposium on Discrete Algorithms'
date_created: 2018-12-11T11:57:34Z
date_published: 2003-01-01T00:00:00Z
date_updated: 2021-01-12T06:57:24Z
day: '01'
extern: 1
main_file_link:
- open_access: '0'
url: http://dl.acm.org/citation.cfm?id=644206
month: '01'
page: 583 - 588
publication_status: published
publisher: SIAM
publist_id: '4503'
quality_controlled: 0
status: public
title: On the rectilinear crossing number of complete graphs
type: conference
year: '2003'
...
---
_id: '2623'
abstract:
- lang: eng
text: Patients with Hodgkin's disease can develop paraneoplastic cerebellar ataxia
because of the generation of autoantibodies against mGluR1 (mGluR1-Abs). Yet,
the pathophysiological mechanisms underlying their motor coordination deficits
remain to be elucidated. Here, we show that application of IgG purified from the
patients' serum to cerebellar slices of mice acutely reduces the basal activity
of Purkinje cells, whereas application to the flocculus of mice in vivo evokes
acute disturbances in the performance of their compensatory eye movements. In
addition, the mGluR1-Abs block induction of long-term depression in cultured mouse
Purkinje cells, whereas the cerebellar motor learning behavior of the patients
is affected in that they show impaired adaptation of their saccadic eye movements.
Finally, postmortem analysis of the cerebellum of a paraneoplastic cerebellar
ataxia patient showed that the number of Purkinje cells was significantly reduced
by approximately two thirds compared with three controls. We conclude that autoantibodies
against mGluR1 can cause cerebellar motor coordination deficits caused by a combination
of rapid effects on both acute and plastic responses of Purkinje cells and chronic
degenerative effects.
author:
- first_name: Michiel
full_name: Coesmans, Michiel P
last_name: Coesmans
- first_name: Peter
full_name: Sillevis-Smitt, Peter A
last_name: Sillevis Smitt
- first_name: David
full_name: Linden, David J
last_name: Linden
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Tomoo
full_name: Hirano, Tomoo
last_name: Hirano
- first_name: Yoshinori
full_name: Yamakawa, Yoshinori
last_name: Yamakawa
- first_name: Adriaan
full_name: Van Alphen, Adriaan M
last_name: Van Alphen
- first_name: Chongde
full_name: Luo, Chongde
last_name: Luo
- first_name: Jos
full_name: Van Der Geest, Jos N
last_name: Van Der Geest
- first_name: Johan
full_name: Kros, Johan M
last_name: Kros
- first_name: Carlo
full_name: Gaillard, Carlo A
last_name: Gaillard
- first_name: Maarten
full_name: Frens, Maarten A
last_name: Frens
- first_name: Chris
full_name: De Zeeuw, Chris I
last_name: De Zeeuw
citation:
ama: Coesmans M, Sillevis Smitt P, Linden D, et al. Mechanisms underlying cerebellar
motor deficits due to mGluR1-autoantibodies. Annals of Neurology. 2003;53(3):325-336.
doi:10.1002/ana.10451
apa: Coesmans, M., Sillevis Smitt, P., Linden, D., Shigemoto, R., Hirano, T., Yamakawa,
Y., … De Zeeuw, C. (2003). Mechanisms underlying cerebellar motor deficits due
to mGluR1-autoantibodies. Annals of Neurology. Wiley-Blackwell. https://doi.org/10.1002/ana.10451
chicago: Coesmans, Michiel, Peter Sillevis Smitt, David Linden, Ryuichi Shigemoto,
Tomoo Hirano, Yoshinori Yamakawa, Adriaan Van Alphen, et al. “Mechanisms Underlying
Cerebellar Motor Deficits Due to MGluR1-Autoantibodies.” Annals of Neurology.
Wiley-Blackwell, 2003. https://doi.org/10.1002/ana.10451.
ieee: M. Coesmans et al., “Mechanisms underlying cerebellar motor deficits
due to mGluR1-autoantibodies,” Annals of Neurology, vol. 53, no. 3. Wiley-Blackwell,
pp. 325–336, 2003.
ista: Coesmans M, Sillevis Smitt P, Linden D, Shigemoto R, Hirano T, Yamakawa Y,
Van Alphen A, Luo C, Van Der Geest J, Kros J, Gaillard C, Frens M, De Zeeuw C.
2003. Mechanisms underlying cerebellar motor deficits due to mGluR1-autoantibodies.
Annals of Neurology. 53(3), 325–336.
mla: Coesmans, Michiel, et al. “Mechanisms Underlying Cerebellar Motor Deficits
Due to MGluR1-Autoantibodies.” Annals of Neurology, vol. 53, no. 3, Wiley-Blackwell,
2003, pp. 325–36, doi:10.1002/ana.10451.
short: M. Coesmans, P. Sillevis Smitt, D. Linden, R. Shigemoto, T. Hirano, Y. Yamakawa,
A. Van Alphen, C. Luo, J. Van Der Geest, J. Kros, C. Gaillard, M. Frens, C. De
Zeeuw, Annals of Neurology 53 (2003) 325–336.
date_created: 2018-12-11T11:58:44Z
date_published: 2003-03-01T00:00:00Z
date_updated: 2021-01-12T06:58:39Z
day: '01'
doi: 10.1002/ana.10451
extern: 1
intvolume: ' 53'
issue: '3'
month: '03'
page: 325 - 336
publication: Annals of Neurology
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4274'
quality_controlled: 0
status: public
title: Mechanisms underlying cerebellar motor deficits due to mGluR1-autoantibodies
type: journal_article
volume: 53
year: '2003'
...
---
_id: '2625'
abstract:
- lang: eng
text: Metabotropic glutamate receptor 1 (mGluR1) plays a crucial role in synaptic
plasticity and motor learning in the cerebellum. We have studied activity-dependent
changes in mGluR1 function in mouse cultured Purkinje neurons. Depolarizing stimulation
potentiated Ca2+ and current responses to an mGluR1 agonist for several hours
in the cultured Purkinje neurons. It also blocked internalization of mGluR1 and
increased the number of mGluR1s on the cell membrane. We found that depolarization
simultaneously increased transcription of Homer1a in Purkinje neurons. Homer1a
inhibited internalization and increased cell-surface expression of mGluR1 when
coexpressed in human embryonic kidney (HEK)-293 cells. Depolarization-induced
Homer1a expression in Purkinje neurons was blocked by a mitogen-activated protein
kinase (MAPK) inhibitor. Changes in internalization and mGluR1-mediated Ca2+ response
were also blocked by inhibition of MAPK activity, suggesting that localization
and activity of mGluR1 were regulated in the same signalling pathway as Homer1a
expression. It is thus suggested that depolarization of the Purkinje neuron leads
to the increment in mGluR1 responsiveness through MAPK activity and induction
of Homer1a expression, which increases active mGluR1 on the cell surface by blocking
internalization of mGluR1.
author:
- first_name: Itsunari
full_name: Minami, Itsunari
last_name: Minami
- first_name: Mineko
full_name: Kengaku, Mineko
last_name: Kengaku
- first_name: Sillevis
full_name: Smitt, Sillevis P
last_name: Smitt
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Tomoo
full_name: Hirano, Tomoo
last_name: Hirano
citation:
ama: Minami I, Kengaku M, Smitt S, Shigemoto R, Hirano T. Long-term potentiation
of mGluR1 activity by depolarization-induced Homer1a in mouse cerebellar Purkinje
neurons. European Journal of Neuroscience. 2003;17(5):1023-1032. doi:10.1046/j.1460-9568.2003.02499.x
apa: Minami, I., Kengaku, M., Smitt, S., Shigemoto, R., & Hirano, T. (2003).
Long-term potentiation of mGluR1 activity by depolarization-induced Homer1a in
mouse cerebellar Purkinje neurons. European Journal of Neuroscience. Wiley-Blackwell.
https://doi.org/10.1046/j.1460-9568.2003.02499.x
chicago: Minami, Itsunari, Mineko Kengaku, Sillevis Smitt, Ryuichi Shigemoto, and
Tomoo Hirano. “Long-Term Potentiation of MGluR1 Activity by Depolarization-Induced
Homer1a in Mouse Cerebellar Purkinje Neurons.” European Journal of Neuroscience.
Wiley-Blackwell, 2003. https://doi.org/10.1046/j.1460-9568.2003.02499.x.
ieee: I. Minami, M. Kengaku, S. Smitt, R. Shigemoto, and T. Hirano, “Long-term potentiation
of mGluR1 activity by depolarization-induced Homer1a in mouse cerebellar Purkinje
neurons,” European Journal of Neuroscience, vol. 17, no. 5. Wiley-Blackwell,
pp. 1023–1032, 2003.
ista: Minami I, Kengaku M, Smitt S, Shigemoto R, Hirano T. 2003. Long-term potentiation
of mGluR1 activity by depolarization-induced Homer1a in mouse cerebellar Purkinje
neurons. European Journal of Neuroscience. 17(5), 1023–1032.
mla: Minami, Itsunari, et al. “Long-Term Potentiation of MGluR1 Activity by Depolarization-Induced
Homer1a in Mouse Cerebellar Purkinje Neurons.” European Journal of Neuroscience,
vol. 17, no. 5, Wiley-Blackwell, 2003, pp. 1023–32, doi:10.1046/j.1460-9568.2003.02499.x.
short: I. Minami, M. Kengaku, S. Smitt, R. Shigemoto, T. Hirano, European Journal
of Neuroscience 17 (2003) 1023–1032.
date_created: 2018-12-11T11:58:44Z
date_published: 2003-03-01T00:00:00Z
date_updated: 2021-01-12T06:58:39Z
day: '01'
doi: 10.1046/j.1460-9568.2003.02499.x
extern: 1
intvolume: ' 17'
issue: '5'
month: '03'
page: 1023 - 1032
publication: European Journal of Neuroscience
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4273'
quality_controlled: 0
status: public
title: Long-term potentiation of mGluR1 activity by depolarization-induced Homer1a
in mouse cerebellar Purkinje neurons
type: journal_article
volume: 17
year: '2003'
...
---
_id: '2626'
abstract:
- lang: eng
text: The expression pattern of metabotropic glutamate receptor Iα (mGluR1α) was
immunohistochemically investigated in substantia nigra dopaminergic neurons of
the macaque monkey. In normal monkeys, mGluR1α immunoreactivity was weakly observed
in the dorsal tier of the substantia nigra pars compacta (SNc-d) where calbindin-D28k-containing
dopaminergic neurons invulnerable to parkinsonian degeneration are specifically
located. On the other hand, mGluR1α was strongly expressed in the ventral tier
of the substantia nigra pars cornpacta (SNc-v). In monkeys treated with the parkinsonism-inducing
drug, I-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), mGluR1α expression
was decreased in dopaminergic neurons in the SNc-v that were spared its toxic
action. These results suggest that mGluR1α expression may be involved at least
partly in the vulnerability of dopaminergic neurons to parkinsonian insults.
author:
- first_name: Katsuyuki
full_name: Kaneda, Katsuyuki
last_name: Kaneda
- first_name: Michiko
full_name: Imanishi, Michiko
last_name: Imanishi
- first_name: Atsushi
full_name: Nambu, Atsushi
last_name: Nambu
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Masahiko
full_name: Takada, Masahiko
last_name: Takada
citation:
ama: Kaneda K, Imanishi M, Nambu A, Shigemoto R, Takada M. Differential expression
patterns of mGluR1α in monkey nigral dopamine neurons. Neuroreport. 2003;14(7):947-950.
doi:10.1097/01.wnr.0000074344.81633.e4
apa: Kaneda, K., Imanishi, M., Nambu, A., Shigemoto, R., & Takada, M. (2003).
Differential expression patterns of mGluR1α in monkey nigral dopamine neurons.
Neuroreport. Lippincott, Williams & Wilkins. https://doi.org/10.1097/01.wnr.0000074344.81633.e4
chicago: Kaneda, Katsuyuki, Michiko Imanishi, Atsushi Nambu, Ryuichi Shigemoto,
and Masahiko Takada. “Differential Expression Patterns of MGluR1α in Monkey Nigral
Dopamine Neurons.” Neuroreport. Lippincott, Williams & Wilkins, 2003.
https://doi.org/10.1097/01.wnr.0000074344.81633.e4.
ieee: K. Kaneda, M. Imanishi, A. Nambu, R. Shigemoto, and M. Takada, “Differential
expression patterns of mGluR1α in monkey nigral dopamine neurons,” Neuroreport,
vol. 14, no. 7. Lippincott, Williams & Wilkins, pp. 947–950, 2003.
ista: Kaneda K, Imanishi M, Nambu A, Shigemoto R, Takada M. 2003. Differential expression
patterns of mGluR1α in monkey nigral dopamine neurons. Neuroreport. 14(7), 947–950.
mla: Kaneda, Katsuyuki, et al. “Differential Expression Patterns of MGluR1α in Monkey
Nigral Dopamine Neurons.” Neuroreport, vol. 14, no. 7, Lippincott, Williams
& Wilkins, 2003, pp. 947–50, doi:10.1097/01.wnr.0000074344.81633.e4.
short: K. Kaneda, M. Imanishi, A. Nambu, R. Shigemoto, M. Takada, Neuroreport 14
(2003) 947–950.
date_created: 2018-12-11T11:58:45Z
date_published: 2003-05-01T00:00:00Z
date_updated: 2021-01-12T06:58:40Z
day: '01'
doi: 10.1097/01.wnr.0000074344.81633.e4
extern: 1
intvolume: ' 14'
issue: '7'
month: '05'
page: 947 - 950
publication: Neuroreport
publication_status: published
publisher: Lippincott, Williams & Wilkins
publist_id: '4272'
quality_controlled: 0
status: public
title: Differential expression patterns of mGluR1α in monkey nigral dopamine neurons
type: journal_article
volume: 14
year: '2003'
...
---
_id: '2627'
abstract:
- lang: eng
text: Despite its implications for higher order functions of the brain, little is
currently known about the molecular basis of left-right asymmetry of the brain.
Here we report that synaptic distribution of N-methyl-D-aspartate (NMDA) receptor
GluRε2 (NR2B) subunits in the adult mouse hippocampus is asymmetrical between
the left and right and between the apical and basal dendrites of single neurons.
These asymmetrical allocations of ε2 subunits differentiate the properties of
NMDA receptors and synaptic plasticity between the left and right hippocampus.
These results provide a molecular basis for the structural and functional asymmetry
of the mature brain.
author:
- first_name: Ryosuke
full_name: Kawakami, Ryosuke
last_name: Kawakami
- first_name: Yoshiaki
full_name: Shinohara, Yoshiaki
last_name: Shinohara
- first_name: Yuichiro
full_name: Kato, Yuichiro
last_name: Kato
- first_name: Hiroyuki
full_name: Sugiyama, Hiroyuki
last_name: Sugiyama
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Isao
full_name: Ito, Isao
last_name: Ito
citation:
ama: Kawakami R, Shinohara Y, Kato Y, Sugiyama H, Shigemoto R, Ito I. Asymmetrical
allocation of NMDA receptor ε2 subunits in hippocampal circuitry. Science.
2003;300(5621):990-994. doi:10.1126/science.1082609
apa: Kawakami, R., Shinohara, Y., Kato, Y., Sugiyama, H., Shigemoto, R., & Ito,
I. (2003). Asymmetrical allocation of NMDA receptor ε2 subunits in hippocampal
circuitry. Science. American Association for the Advancement of Science.
https://doi.org/10.1126/science.1082609
chicago: Kawakami, Ryosuke, Yoshiaki Shinohara, Yuichiro Kato, Hiroyuki Sugiyama,
Ryuichi Shigemoto, and Isao Ito. “Asymmetrical Allocation of NMDA Receptor Ε2
Subunits in Hippocampal Circuitry.” Science. American Association for the
Advancement of Science, 2003. https://doi.org/10.1126/science.1082609.
ieee: R. Kawakami, Y. Shinohara, Y. Kato, H. Sugiyama, R. Shigemoto, and I. Ito,
“Asymmetrical allocation of NMDA receptor ε2 subunits in hippocampal circuitry,”
Science, vol. 300, no. 5621. American Association for the Advancement of
Science, pp. 990–994, 2003.
ista: Kawakami R, Shinohara Y, Kato Y, Sugiyama H, Shigemoto R, Ito I. 2003. Asymmetrical
allocation of NMDA receptor ε2 subunits in hippocampal circuitry. Science. 300(5621),
990–994.
mla: Kawakami, Ryosuke, et al. “Asymmetrical Allocation of NMDA Receptor Ε2 Subunits
in Hippocampal Circuitry.” Science, vol. 300, no. 5621, American Association
for the Advancement of Science, 2003, pp. 990–94, doi:10.1126/science.1082609.
short: R. Kawakami, Y. Shinohara, Y. Kato, H. Sugiyama, R. Shigemoto, I. Ito, Science
300 (2003) 990–994.
date_created: 2018-12-11T11:58:45Z
date_published: 2003-05-09T00:00:00Z
date_updated: 2021-01-12T06:58:40Z
day: '09'
doi: 10.1126/science.1082609
extern: 1
intvolume: ' 300'
issue: '5621'
month: '05'
page: 990 - 994
publication: Science
publication_status: published
publisher: American Association for the Advancement of Science
publist_id: '4271'
quality_controlled: 0
status: public
title: Asymmetrical allocation of NMDA receptor ε2 subunits in hippocampal circuitry
type: journal_article
volume: 300
year: '2003'
...
---
_id: '2629'
abstract:
- lang: eng
text: The release of neurotransmitters is modulated by presynaptic metabotropic
glutamate receptors (mGluRs), which show a highly selective expression and subcellular
location in glutamatergic terminals in the hippocampus. Using immunocytochemistry,
we investigated whether one of the receptors, mGluR7, whose level of expression
is governed by the postsynaptic target, was present in GABAergic terminals and
whether such terminals targeted particular cells. A total of 165 interneuron dendritic
profiles receiving 466 synapses (82% mGluR7a-positive) were analysed. The presynaptic
active zones of most GAD-(77%) or GABA-positive (94%) synaptic boutons on interneurons
innervated by mGluR7a-enriched glutamatergic terminals (mGluR7a-decorated) were
immunopositive for mGluR7a. GABAergic terminals on pyramidal cells and most other
interneurons in str. oriens were mGluR7a-immunonegative. The mGluR7a-decorated
cells were mostly somatostatin- and mGluR1α-immunopositive neurons in str. oriens
and the alveus. Their GABAergic input mainly originated from VIP-positive terminals,
90% of which expressed high levels of mGluR7a in the presynaptic active zone.
Parvalbumin-positive synaptic terminals were rare on mGluR7a-decorated cells,
but on these neurons 73% of them were mGluR7a-immunopositive. Some type II synapses
innervating interneurons were immunopositive for mGluR7b, as were some type I
synapses. Because not all target cells of VIP-positive neurons are known it has
not been possible to determine whether mGluR7 is expressed in a target-cell-specific
manner in the terminals of single GABAergic cells. The activation of mGluR7 may
decrease GABA release to mGluR7-decorated cells at times of high pyramidal cell
activity, which elevates extracellular glutamate levels. Alternatively, the presynaptic
receptor may be activated by as yet unidentified endogenous ligands released by
the GABAergic terminals or the postsynaptic dendrites.
author:
- first_name: Péter
full_name: Somogyi, Péter
last_name: Somogyi
- first_name: Yannis
full_name: Dalezios, Yannis
last_name: Dalezios
- first_name: Rafael
full_name: Luján, Rafael
last_name: Luján
- first_name: John
full_name: Roberts, John D
last_name: Roberts
- first_name: Masahiko
full_name: Watanabe, Masahiko
last_name: Watanabe
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
citation:
ama: Somogyi P, Dalezios Y, Luján R, Roberts J, Watanabe M, Shigemoto R. High level
of mGluR7 in the presynaptic active zones of select populations of GABAergic terminals
innervating interneurons in the rat hippocampus. European Journal of Neuroscience.
2003;17(12):2503-2520. doi:10.1046/j.1460-9568.2003.02697.x
apa: Somogyi, P., Dalezios, Y., Luján, R., Roberts, J., Watanabe, M., & Shigemoto,
R. (2003). High level of mGluR7 in the presynaptic active zones of select populations
of GABAergic terminals innervating interneurons in the rat hippocampus. European
Journal of Neuroscience. Wiley-Blackwell. https://doi.org/10.1046/j.1460-9568.2003.02697.x
chicago: Somogyi, Péter, Yannis Dalezios, Rafael Luján, John Roberts, Masahiko Watanabe,
and Ryuichi Shigemoto. “High Level of MGluR7 in the Presynaptic Active Zones of
Select Populations of GABAergic Terminals Innervating Interneurons in the Rat
Hippocampus.” European Journal of Neuroscience. Wiley-Blackwell, 2003.
https://doi.org/10.1046/j.1460-9568.2003.02697.x.
ieee: P. Somogyi, Y. Dalezios, R. Luján, J. Roberts, M. Watanabe, and R. Shigemoto,
“High level of mGluR7 in the presynaptic active zones of select populations of
GABAergic terminals innervating interneurons in the rat hippocampus,” European
Journal of Neuroscience, vol. 17, no. 12. Wiley-Blackwell, pp. 2503–2520,
2003.
ista: Somogyi P, Dalezios Y, Luján R, Roberts J, Watanabe M, Shigemoto R. 2003.
High level of mGluR7 in the presynaptic active zones of select populations of
GABAergic terminals innervating interneurons in the rat hippocampus. European
Journal of Neuroscience. 17(12), 2503–2520.
mla: Somogyi, Péter, et al. “High Level of MGluR7 in the Presynaptic Active Zones
of Select Populations of GABAergic Terminals Innervating Interneurons in the Rat
Hippocampus.” European Journal of Neuroscience, vol. 17, no. 12, Wiley-Blackwell,
2003, pp. 2503–20, doi:10.1046/j.1460-9568.2003.02697.x.
short: P. Somogyi, Y. Dalezios, R. Luján, J. Roberts, M. Watanabe, R. Shigemoto,
European Journal of Neuroscience 17 (2003) 2503–2520.
date_created: 2018-12-11T11:58:46Z
date_published: 2003-06-01T00:00:00Z
date_updated: 2021-01-12T06:58:41Z
day: '01'
doi: 10.1046/j.1460-9568.2003.02697.x
extern: 1
intvolume: ' 17'
issue: '12'
month: '06'
page: 2503 - 2520
publication: European Journal of Neuroscience
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4269'
quality_controlled: 0
status: public
title: High level of mGluR7 in the presynaptic active zones of select populations
of GABAergic terminals innervating interneurons in the rat hippocampus
type: journal_article
volume: 17
year: '2003'
...
---
_id: '2628'
abstract:
- lang: eng
text: We aimed to estimate the number of AMPA receptors (AMPARs) bound by the quantal
transmitter packet, their single-channel conductance and their density in the
postsynaptic membrane at cerebellar Purkinje cell synapses. The synaptic and extrasynaptic
AMPARs were examined in Purkinje cells in 2- to 4-day-old rats, when they receive
synaptic inputs solely from climbing fibres (CFs). Evoked CF EPSCs and whole-cell
AMPA currents displayed roughly linear current-voltage relationships, consistent
with the presence of GluR2 subunits in synaptic and extrasynaptic AMPARs. The
mean quantal size, estimated from the miniature EPSCs (MEPSCs), was ∼300 pS. Peak-scaled
non-stationary fluctuation analysis of spontaneous EPSCs and MEPSCs gave a weighted-mean
synaptic channel conductance of ∼5 pS (∼7 pS when corrected for filtering). By
applying non-stationary fluctuation analysis to extrasynaptic currents activated
by brief glutamate pulses (5 mM), we also obtained a small single-channel conductance
estimate for extrasynaptic AMPARs (∼11 pS). This approach allowed us to obtain
a maximum open probability (Po,max) value for the extrasynaptic receptors (Po,max
= 0.72). Directly resolved extrasynaptic channel openings in the continued presence
of glutamate exhibited clear multiple-conductance levels. The mean area of the
postsynaptic density (PSD) of these synapses was 0.074 μm2, measured by reconstructing
electron-microscopic (EM) serial sections. Postembedding immunogold labelling
by anti-GluR2/3 antibody revealed that AMPARs are localised in PSDs. From these
data and by simulating error factors, we estimate that at least 66 AMPARs are
bound by a quantal transmitter packet at CF-Purkinje cell synapses, and the receptors
are packed at a minimum density of ∼900 μm-2 in the postsynaptic membrane.
author:
- first_name: Akiko
full_name: Momiyama, Akiko
last_name: Momiyama
- first_name: Rachel
full_name: Silver, Rachel A
last_name: Silver
- first_name: Michael
full_name: Häusser, Michael A
last_name: Häusser
- first_name: Takuya
full_name: Notomi, Takuya
last_name: Notomi
- first_name: Yue
full_name: Wu, Yue
last_name: Wu
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Stuart
full_name: Cull-Candy, Stuart G
last_name: Cull Candy
citation:
ama: Momiyama A, Silver R, Häusser M, et al. The density of AMPA receptors activated
by a transmitter quantum at the climbing fibre - Purkinje cell synapse in immature
rats. Journal of Physiology. 2003;549(1):75-92. doi:10.1113/jphysiol.2002.033472
apa: Momiyama, A., Silver, R., Häusser, M., Notomi, T., Wu, Y., Shigemoto, R., &
Cull Candy, S. (2003). The density of AMPA receptors activated by a transmitter
quantum at the climbing fibre - Purkinje cell synapse in immature rats. Journal
of Physiology. Wiley-Blackwell. https://doi.org/10.1113/jphysiol.2002.033472
chicago: Momiyama, Akiko, Rachel Silver, Michael Häusser, Takuya Notomi, Yue Wu,
Ryuichi Shigemoto, and Stuart Cull Candy. “The Density of AMPA Receptors Activated
by a Transmitter Quantum at the Climbing Fibre - Purkinje Cell Synapse in Immature
Rats.” Journal of Physiology. Wiley-Blackwell, 2003. https://doi.org/10.1113/jphysiol.2002.033472.
ieee: A. Momiyama et al., “The density of AMPA receptors activated by a transmitter
quantum at the climbing fibre - Purkinje cell synapse in immature rats,” Journal
of Physiology, vol. 549, no. 1. Wiley-Blackwell, pp. 75–92, 2003.
ista: Momiyama A, Silver R, Häusser M, Notomi T, Wu Y, Shigemoto R, Cull Candy S.
2003. The density of AMPA receptors activated by a transmitter quantum at the
climbing fibre - Purkinje cell synapse in immature rats. Journal of Physiology.
549(1), 75–92.
mla: Momiyama, Akiko, et al. “The Density of AMPA Receptors Activated by a Transmitter
Quantum at the Climbing Fibre - Purkinje Cell Synapse in Immature Rats.” Journal
of Physiology, vol. 549, no. 1, Wiley-Blackwell, 2003, pp. 75–92, doi:10.1113/jphysiol.2002.033472.
short: A. Momiyama, R. Silver, M. Häusser, T. Notomi, Y. Wu, R. Shigemoto, S. Cull
Candy, Journal of Physiology 549 (2003) 75–92.
date_created: 2018-12-11T11:58:45Z
date_published: 2003-05-15T00:00:00Z
date_updated: 2021-01-12T06:58:40Z
day: '15'
doi: 10.1113/jphysiol.2002.033472
extern: 1
intvolume: ' 549'
issue: '1'
month: '05'
page: 75 - 92
publication: Journal of Physiology
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4270'
quality_controlled: 0
status: public
title: The density of AMPA receptors activated by a transmitter quantum at the climbing
fibre - Purkinje cell synapse in immature rats
type: journal_article
volume: 549
year: '2003'
...
---
_id: '2631'
abstract:
- lang: eng
text: Cyclic ADP-ribose (cADP-ribose) is a putative second messenger or modulator.
However, the role of cADP-ribose in the downstream signals of the metabotropic
glutamate receptors (mGluRs) is unclear. Here, we show that glutamate stimulates
ADP-ribosyl cyclase activity in rat or mouse crude membranes of retina via group
III mGluRs or in superior cervical ganglion via group I mGluRs. The retina of
mGluR6-deficient mice showed no increase in the ADP-ribosyl cyclase level in response
to glutamate. GTP enhanced the initial rate of basal and glutamate-stimulated
cyclase activity. GTP-γ-S also stimulated basal activity. To determine whether
the coupling mode of mGluRs to ADP-ribosyl cyclase is a feature common to individual
cloned mGluRs, we expressed each mGluR subtype in NG108-15 neuroblastoma x glioma
hybrid cells. The glutamate-induced stimulation of the cyclase occurs preferentially
in NG108-15 cells over-expressing mGluRs1, 3, 5, and 6. Cells expressing mGluR2
or mGluRs4 and 7 exhibit inhibition or no coupling, respectively. Glutamate-induced
activation or inhibition of the cyclase activity was eliminated after pre-treatment
with cholera or pertussis toxin, respectively. Thus, the subtype-specific coupling
of mGluRs to ADP-ribosyl cyclase via G proteins suggests that some glutamate-evoked
neuronal functions are mediated by cADP-ribose.
author:
- first_name: Haruhiro
full_name: Higashida, Haruhiro
last_name: Higashida
- first_name: Jia
full_name: Zhang, Jia-Sheng
last_name: Zhang
- first_name: Sumiko
full_name: Mochida, Sumiko
last_name: Mochida
- first_name: Xiao
full_name: Chen, Xiao-Liang
last_name: Chen
- first_name: Yeonsook
full_name: Shin, Yeonsook
last_name: Shin
- first_name: Mami
full_name: Noda, Mami
last_name: Noda
- first_name: Kazi
full_name: Hossain, Kazi Z
last_name: Hossain
- first_name: Naoto
full_name: Hoshi, Naoto
last_name: Hoshi
- first_name: Minako
full_name: Hashii, Minako
last_name: Hashii
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Shigetada
full_name: Nakanishi, Shigetada
last_name: Nakanishi
- first_name: Yutaka
full_name: Fukuda, Yutaka
last_name: Fukuda
- first_name: Shigeru
full_name: Yokoyama, Shigeru
last_name: Yokoyama
citation:
ama: Higashida H, Zhang J, Mochida S, et al. Subtype-specific coupling with ADP-ribosyl
cyclase of metabotropic glutamate receptors in retina, cervical superior ganglion
and NG108-15 cells. Journal of Neurochemistry. 2003;85(5):1148-1158. doi:10.1046/j.1471-4159.2003.01751.x
apa: Higashida, H., Zhang, J., Mochida, S., Chen, X., Shin, Y., Noda, M., … Yokoyama,
S. (2003). Subtype-specific coupling with ADP-ribosyl cyclase of metabotropic
glutamate receptors in retina, cervical superior ganglion and NG108-15 cells.
Journal of Neurochemistry. Wiley-Blackwell. https://doi.org/10.1046/j.1471-4159.2003.01751.x
chicago: Higashida, Haruhiro, Jia Zhang, Sumiko Mochida, Xiao Chen, Yeonsook Shin,
Mami Noda, Kazi Hossain, et al. “Subtype-Specific Coupling with ADP-Ribosyl Cyclase
of Metabotropic Glutamate Receptors in Retina, Cervical Superior Ganglion and
NG108-15 Cells.” Journal of Neurochemistry. Wiley-Blackwell, 2003. https://doi.org/10.1046/j.1471-4159.2003.01751.x.
ieee: H. Higashida et al., “Subtype-specific coupling with ADP-ribosyl cyclase
of metabotropic glutamate receptors in retina, cervical superior ganglion and
NG108-15 cells,” Journal of Neurochemistry, vol. 85, no. 5. Wiley-Blackwell,
pp. 1148–1158, 2003.
ista: Higashida H, Zhang J, Mochida S, Chen X, Shin Y, Noda M, Hossain K, Hoshi
N, Hashii M, Shigemoto R, Nakanishi S, Fukuda Y, Yokoyama S. 2003. Subtype-specific
coupling with ADP-ribosyl cyclase of metabotropic glutamate receptors in retina,
cervical superior ganglion and NG108-15 cells. Journal of Neurochemistry. 85(5),
1148–1158.
mla: Higashida, Haruhiro, et al. “Subtype-Specific Coupling with ADP-Ribosyl Cyclase
of Metabotropic Glutamate Receptors in Retina, Cervical Superior Ganglion and
NG108-15 Cells.” Journal of Neurochemistry, vol. 85, no. 5, Wiley-Blackwell,
2003, pp. 1148–58, doi:10.1046/j.1471-4159.2003.01751.x.
short: H. Higashida, J. Zhang, S. Mochida, X. Chen, Y. Shin, M. Noda, K. Hossain,
N. Hoshi, M. Hashii, R. Shigemoto, S. Nakanishi, Y. Fukuda, S. Yokoyama, Journal
of Neurochemistry 85 (2003) 1148–1158.
date_created: 2018-12-11T11:58:46Z
date_published: 2003-06-01T00:00:00Z
date_updated: 2021-01-12T06:58:42Z
day: '01'
doi: 10.1046/j.1471-4159.2003.01751.x
extern: 1
intvolume: ' 85'
issue: '5'
month: '06'
page: 1148 - 1158
publication: Journal of Neurochemistry
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4268'
quality_controlled: 0
status: public
title: Subtype-specific coupling with ADP-ribosyl cyclase of metabotropic glutamate
receptors in retina, cervical superior ganglion and NG108-15 cells
type: journal_article
volume: 85
year: '2003'
...
---
_id: '2633'
abstract:
- lang: eng
text: The modulation of calcium channels by metabotropic glutamate receptors (mGluRs)
is a key event in the fine-tuning of neurotransmitter release. Here we report
that, in cerebrocortical nerve terminals of adult rats, the inhibition of glutamate
release is mediated by mGluR7. In this preparation, the major component of glutamate
release is supported by P/Q-type Ca2+ channels (72.7%). However, mGluR7 selectively
reduced the release component that is associated with N-type Ca2+ channels (29.9%).
Inhibition of P/Q channels by mGluR7 is not masked by the higher efficiency of
these channels in driving glutamate release when compared with N-type channels.
Thus, activation of mGluR7 failed to reduce the release associated with P/Q channels
when the extracellular calcium concentration, ([Ca2+]o), was reduced from 1.3
to 0.5 mM. Through Ca2+ imaging, we show that Ca2+ channels are distributed in
a heterogeneous manner in individual nerve terminals. Indeed, in this preparation,
nerve terminals were observed that contain N-type (31.1%; conotoxin GVIA-sensitive)
or P/Q-type (64.3%; agatoxin IVA-sensitive) channels or that were insensitive
to these two toxins (4.6%). Interestingly, the great majority of the responses
to L-AP4 (95.4%) were observed in nerve terminals containing N-type channels.
This specific co-localization of mGluR7 and N-type Ca2+-channels could explain
the failure of the receptor to inhibit the P/Q channel-associated release component
and also reveal the existence of specific targeting mechanisms to localize the
two proteins in the same nerve terminal subset.
author:
- first_name: Carmelo
full_name: Millán, Carmelo
last_name: Millán
- first_name: Enrique
full_name: Castro, Enrique G
last_name: Castro
- first_name: Magdalena
full_name: Torres, Magdalena
last_name: Torres
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: José
full_name: Sánchez-Prieto, José
last_name: Sánchez Prieto
citation:
ama: Millán C, Castro E, Torres M, Shigemoto R, Sánchez Prieto J. Co-expression
of metabotropic glutamate receptor 7 and N-type Ca2+ channels in single cerebrocortical
nerve terminals of adult rats. Journal of Biological Chemistry. 2003;278(26):23955-23962.
doi:10.1074/jbc.M211471200
apa: Millán, C., Castro, E., Torres, M., Shigemoto, R., & Sánchez Prieto, J.
(2003). Co-expression of metabotropic glutamate receptor 7 and N-type Ca2+ channels
in single cerebrocortical nerve terminals of adult rats. Journal of Biological
Chemistry. American Society for Biochemistry and Molecular Biology. https://doi.org/10.1074/jbc.M211471200
chicago: Millán, Carmelo, Enrique Castro, Magdalena Torres, Ryuichi Shigemoto, and
José Sánchez Prieto. “Co-Expression of Metabotropic Glutamate Receptor 7 and N-Type
Ca2+ Channels in Single Cerebrocortical Nerve Terminals of Adult Rats.” Journal
of Biological Chemistry. American Society for Biochemistry and Molecular Biology,
2003. https://doi.org/10.1074/jbc.M211471200.
ieee: C. Millán, E. Castro, M. Torres, R. Shigemoto, and J. Sánchez Prieto, “Co-expression
of metabotropic glutamate receptor 7 and N-type Ca2+ channels in single cerebrocortical
nerve terminals of adult rats,” Journal of Biological Chemistry, vol. 278,
no. 26. American Society for Biochemistry and Molecular Biology, pp. 23955–23962,
2003.
ista: Millán C, Castro E, Torres M, Shigemoto R, Sánchez Prieto J. 2003. Co-expression
of metabotropic glutamate receptor 7 and N-type Ca2+ channels in single cerebrocortical
nerve terminals of adult rats. Journal of Biological Chemistry. 278(26), 23955–23962.
mla: Millán, Carmelo, et al. “Co-Expression of Metabotropic Glutamate Receptor 7
and N-Type Ca2+ Channels in Single Cerebrocortical Nerve Terminals of Adult Rats.”
Journal of Biological Chemistry, vol. 278, no. 26, American Society for
Biochemistry and Molecular Biology, 2003, pp. 23955–62, doi:10.1074/jbc.M211471200.
short: C. Millán, E. Castro, M. Torres, R. Shigemoto, J. Sánchez Prieto, Journal
of Biological Chemistry 278 (2003) 23955–23962.
date_created: 2018-12-11T11:58:47Z
date_published: 2003-07-27T00:00:00Z
date_updated: 2021-01-12T06:58:42Z
day: '27'
doi: 10.1074/jbc.M211471200
extern: 1
intvolume: ' 278'
issue: '26'
month: '07'
page: 23955 - 23962
publication: Journal of Biological Chemistry
publication_status: published
publisher: American Society for Biochemistry and Molecular Biology
publist_id: '4265'
quality_controlled: 0
status: public
title: Co-expression of metabotropic glutamate receptor 7 and N-type Ca2+ channels
in single cerebrocortical nerve terminals of adult rats
type: journal_article
volume: 278
year: '2003'
...
---
_id: '2632'
abstract:
- lang: eng
text: In many brain regions, hyperpolarization-activated cationic currents (Ih)
are involved in the generation of rhythmic activities, but the role of Ih in olfactory
oscillations remains unclear. Knowledge of the cellular and subcellular distributions
of hyperpolarization-activated and cyclic nucleotide-gated channel (HCN) subunits
is necessary for understanding the role of Ih in olfactory network activities.
Using light microscopic immunocytochemistry, we demonstrate strong HCN1 labelling
of the glomerular layer and moderate staining of granule cell, internal and external
plexiform layers of the rat main olfactory bulb. In the glomerular layer, among
many unlabelled neurons, two distinct subpopulations of juxtaglomerular cells
are labelled. Approximately 10% of the juxtaglomerular cells strongly express
HCN1. These small diameter cells are immunoreactive for GABA and comprise a subpopulation
of periglomerular cells. An additional subset of juxtaglomerular cells (≈ 1%)
expresses low levels of HCN1. They are large in diameter, GABA immunonegative
but immunopositive for vesicular glutamate transporter 2, characterizing them
as external tufted cells. Quantitative immunogold localization revealed that the
somatic plasma membranes of periglomerular cells contain approximately four times
more HCN1 labelling than those of external tufted cells. Unlike in cortical pyramidal
cells, immunogold density for HCN1 does not significantly differ in somatic and
dendritic plasma membranes of external tufted cells, indicating that post-synaptic
potentials arriving at proximal and distal dendrites are modulated by the same
density of I h. Our results demonstrate a cell type-dependent expression of HCN1
in the olfactory bulb and predict a differential contribution of distinct juxtaglomerular
cell types to network oscillations.
author:
- first_name: Noémi
full_name: Holderith, Noémi B
last_name: Holderith
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Zoltán
full_name: Nusser, Zoltán
last_name: Nusser
citation:
ama: Holderith N, Shigemoto R, Nusser Z. Cell type-dependent expression of HCN1
in the main olfactory bulb. European Journal of Neuroscience. 2003;18(2):344-354.
doi:10.1046/j.1460-9568.2003.02756.x
apa: Holderith, N., Shigemoto, R., & Nusser, Z. (2003). Cell type-dependent
expression of HCN1 in the main olfactory bulb. European Journal of Neuroscience.
Wiley-Blackwell. https://doi.org/10.1046/j.1460-9568.2003.02756.x
chicago: Holderith, Noémi, Ryuichi Shigemoto, and Zoltán Nusser. “Cell Type-Dependent
Expression of HCN1 in the Main Olfactory Bulb.” European Journal of Neuroscience.
Wiley-Blackwell, 2003. https://doi.org/10.1046/j.1460-9568.2003.02756.x.
ieee: N. Holderith, R. Shigemoto, and Z. Nusser, “Cell type-dependent expression
of HCN1 in the main olfactory bulb,” European Journal of Neuroscience,
vol. 18, no. 2. Wiley-Blackwell, pp. 344–354, 2003.
ista: Holderith N, Shigemoto R, Nusser Z. 2003. Cell type-dependent expression of
HCN1 in the main olfactory bulb. European Journal of Neuroscience. 18(2), 344–354.
mla: Holderith, Noémi, et al. “Cell Type-Dependent Expression of HCN1 in the Main
Olfactory Bulb.” European Journal of Neuroscience, vol. 18, no. 2, Wiley-Blackwell,
2003, pp. 344–54, doi:10.1046/j.1460-9568.2003.02756.x.
short: N. Holderith, R. Shigemoto, Z. Nusser, European Journal of Neuroscience 18
(2003) 344–354.
date_created: 2018-12-11T11:58:47Z
date_published: 2003-07-01T00:00:00Z
date_updated: 2021-01-12T06:58:42Z
day: '01'
doi: 10.1046/j.1460-9568.2003.02756.x
extern: 1
intvolume: ' 18'
issue: '2'
month: '07'
page: 344 - 354
publication: European Journal of Neuroscience
publication_status: published
publisher: Wiley-Blackwell
publist_id: '4266'
quality_controlled: 0
status: public
title: Cell type-dependent expression of HCN1 in the main olfactory bulb
type: journal_article
volume: 18
year: '2003'
...
---
_id: '2635'
abstract:
- lang: eng
text: Metabotropic GABAB receptors mediate slow inhibitory effects presynaptically
and postsynaptically. Using preembedding immunohistochemical methods combined
with quantitative analysis of GABAB receptor subunit immunoreactivity, this study
provides a detailed description of the cellular and subcellular localization of
GABAB1a/b and GABA B2 in the rat hippocampus. At the light microscopic level,
an overlapping distribution of GABAB1a/b and GABAB2 was revealed in the dendritic
layers of the hippocampus. In addition, expression of the GABAB1a/b subunit was
found in somata of CA1 pyramidal cells and of a subset of GABAergic interneurons.
At the electron microscopic level, immunoreactivity for both subunits was observed
on presynaptic and, more abundantly, on postsynaptic elements. Presynaptically,
subunits were mainly detected in the extrasynaptic membrane and occasionally over
the presynaptic membrane specialization of putative glutamatergic and, to a lesser
extent, GABAergic axon terminals. Postsynaptically, the majority of GABAB receptor
subunits were localized to the extrasynaptic plasma membrane of spines and dendritic
shafts of principal cells and shafts of interneuron dendrites. Quantitative analysis
revealed enrichment of GABAB1a/b around putative glutamatergic synapses on spines
and an even distribution on dendritic shafts of pyramidal cells contacted by GABAergic
boutons. The association of GABAB receptors with glutamatergic synapses at both
presynaptic and postsynaptic sides indicates their intimate involvement in the
modulation of glutamatergic neurotransmission. The dominant extrasynaptic localization
of GABAB receptor subunits suggests that their activation is dependent on spillover
of GABA requiring simultaneous activity of populations of GABAergic cells as it
occurs during population oscillations or epileptic seizures.
author:
- first_name: Ákos
full_name: Kulik, Ákos
last_name: Kulik
- first_name: Imre
full_name: Vida, Imre
last_name: Vida
- first_name: Rafael
full_name: Luján, Rafael
last_name: Luján
- first_name: Carola
full_name: Haas, Carola A
last_name: Haas
- first_name: Guillermina
full_name: López-Bendito, Guillermina
last_name: López Bendito
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Michael
full_name: Frotscher, Michael
last_name: Frotscher
citation:
ama: Kulik Á, Vida I, Luján R, et al. Subcellular Localization of Metabotropic GABAB
Receptor Subunits GABAB1a/b and GABAB2 in the Rat Hippocampus. Journal of Neuroscience.
2003;23(35):11026-11035.
apa: Kulik, Á., Vida, I., Luján, R., Haas, C., López Bendito, G., Shigemoto, R.,
& Frotscher, M. (2003). Subcellular Localization of Metabotropic GABAB Receptor
Subunits GABAB1a/b and GABAB2 in the Rat Hippocampus. Journal of Neuroscience.
Society for Neuroscience.
chicago: Kulik, Ákos, Imre Vida, Rafael Luján, Carola Haas, Guillermina López Bendito,
Ryuichi Shigemoto, and Michael Frotscher. “Subcellular Localization of Metabotropic
GABAB Receptor Subunits GABAB1a/b and GABAB2 in the Rat Hippocampus.” Journal
of Neuroscience. Society for Neuroscience, 2003.
ieee: Á. Kulik et al., “Subcellular Localization of Metabotropic GABAB Receptor
Subunits GABAB1a/b and GABAB2 in the Rat Hippocampus,” Journal of Neuroscience,
vol. 23, no. 35. Society for Neuroscience, pp. 11026–11035, 2003.
ista: Kulik Á, Vida I, Luján R, Haas C, López Bendito G, Shigemoto R, Frotscher
M. 2003. Subcellular Localization of Metabotropic GABAB Receptor Subunits GABAB1a/b
and GABAB2 in the Rat Hippocampus. Journal of Neuroscience. 23(35), 11026–11035.
mla: Kulik, Ákos, et al. “Subcellular Localization of Metabotropic GABAB Receptor
Subunits GABAB1a/b and GABAB2 in the Rat Hippocampus.” Journal of Neuroscience,
vol. 23, no. 35, Society for Neuroscience, 2003, pp. 11026–35.
short: Á. Kulik, I. Vida, R. Luján, C. Haas, G. López Bendito, R. Shigemoto, M.
Frotscher, Journal of Neuroscience 23 (2003) 11026–11035.
date_created: 2018-12-11T11:58:47Z
date_published: 2003-12-03T00:00:00Z
date_updated: 2021-01-12T06:58:43Z
day: '03'
extern: 1
intvolume: ' 23'
issue: '35'
month: '12'
page: 11026 - 11035
publication: Journal of Neuroscience
publication_status: published
publisher: Society for Neuroscience
publist_id: '4263'
quality_controlled: 0
status: public
title: Subcellular Localization of Metabotropic GABAB Receptor Subunits GABAB1a/b
and GABAB2 in the Rat Hippocampus
type: journal_article
volume: 23
year: '2003'
...
---
_id: '2634'
abstract:
- lang: eng
text: To better understand the role of neurotransmitter receptors in neuronal differentiation
and maturation a detailed knowledge of their identity, location and function in
the plasma membrane of specific neuronal populations during development is required.
Combining pre-embedding immunocytochemistry with cell tracking in embryonic brain
slice cultures we show that virtually all neurons (∼98%) migrating through the
lower intermediate zone (LIZ) on their way from the medial ganglionic eminence
to the cerebral cortex, express GABA BR1. Blockade of GABABRs with a specific
antagonist, CGP52432, resulted in a concentration-dependent accumulation of these
tangentially migrating neurons in the ventricular/subventricular zones (VZ/SVZ)
of the cortex and fewer cells were observed in the cortical plate/marginal zone
(CP/MZ) and LIZ. Moreover, they had significantly shorter leading processes compared
with similar migrating cells in control slices. Electrophysiological recording
in LIZ and CP cells revealed no direct effect of either CGP52432 or the GABABR
agonist, baclofen, on resting membrane properties suggesting that the effect of
CGP52432 on migration might be mediated through a metabotropic action or the regulation
of release of factors controlling migration. These results suggest that GABABRs
have an important modulatory role in the migration of cortical interneurons.
author:
- first_name: Guillermina
full_name: López-Bendito, Guillermina
last_name: López Bendito
- first_name: Rafael
full_name: Luján, Rafael
last_name: Luján
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Paul
full_name: Ganter, Paul
last_name: Ganter
- first_name: Ole
full_name: Paulsen, Ole
last_name: Paulsen
- first_name: Zoltán
full_name: Molnár, Zoltán
last_name: Molnár
citation:
ama: López Bendito G, Luján R, Shigemoto R, Ganter P, Paulsen O, Molnár Z. Blockade
of GABAB receptors alters the tangential migration of cortical neurons. Cerebral
Cortex. 2003;13(9):932-942. doi:10.1093/cercor/13.9.932
apa: López Bendito, G., Luján, R., Shigemoto, R., Ganter, P., Paulsen, O., &
Molnár, Z. (2003). Blockade of GABAB receptors alters the tangential migration
of cortical neurons. Cerebral Cortex. Oxford University Press. https://doi.org/10.1093/cercor/13.9.932
chicago: López Bendito, Guillermina, Rafael Luján, Ryuichi Shigemoto, Paul Ganter,
Ole Paulsen, and Zoltán Molnár. “Blockade of GABAB Receptors Alters the Tangential
Migration of Cortical Neurons.” Cerebral Cortex. Oxford University Press,
2003. https://doi.org/10.1093/cercor/13.9.932.
ieee: G. López Bendito, R. Luján, R. Shigemoto, P. Ganter, O. Paulsen, and Z. Molnár,
“Blockade of GABAB receptors alters the tangential migration of cortical neurons,”
Cerebral Cortex, vol. 13, no. 9. Oxford University Press, pp. 932–942,
2003.
ista: López Bendito G, Luján R, Shigemoto R, Ganter P, Paulsen O, Molnár Z. 2003.
Blockade of GABAB receptors alters the tangential migration of cortical neurons.
Cerebral Cortex. 13(9), 932–942.
mla: López Bendito, Guillermina, et al. “Blockade of GABAB Receptors Alters the
Tangential Migration of Cortical Neurons.” Cerebral Cortex, vol. 13, no.
9, Oxford University Press, 2003, pp. 932–42, doi:10.1093/cercor/13.9.932.
short: G. López Bendito, R. Luján, R. Shigemoto, P. Ganter, O. Paulsen, Z. Molnár,
Cerebral Cortex 13 (2003) 932–942.
date_created: 2018-12-11T11:58:47Z
date_published: 2003-09-01T00:00:00Z
date_updated: 2021-01-12T06:58:43Z
day: '01'
doi: 10.1093/cercor/13.9.932
extern: 1
intvolume: ' 13'
issue: '9'
month: '09'
page: 932 - 942
publication: Cerebral Cortex
publication_status: published
publisher: Oxford University Press
publist_id: '4264'
quality_controlled: 0
status: public
title: Blockade of GABAB receptors alters the tangential migration of cortical neurons
type: journal_article
volume: 13
year: '2003'
...
---
_id: '2630'
abstract:
- lang: eng
text: Taste-metabotropic glutamate receptor 4 (taste-mGluR4) and the heteromers
of T1R1 and T1R3 are candidate receptors involved in the sense of umami (monosodium
glutamate) taste. Although the expression of group III mGluRs (taste-mGluR4) has
been demonstrated in taste tissues, no mention has been made of the expression
of group I mGluRs (mGluR1 and mGluR5) in taste tissues. We examined the expression
of mGluR1 and mGluR5 in rat gustatory tissues by using reverse transcription-polymerase
chain reaction (RT-PCR), in situ hybridization, immunohistochemistry and immunoelectron
microscopy. RT-PCR assay showed that mGluR1α and mGluR1β mRNAs were expressed
in circumvallate papillae, but mGluR5 mRNA was not expressed. The positive signals
of mGluR1 mRNA were detected only in circumvallate taste buds by in situ hybridization
analysis. In cryosections of fungiform, foliate and circumvallate papillae, the
antibody against mGluRla gave intense labeling on the taste hairs in all taste
pores examined. In the developing taste buds, the positive signals of mGluR1α
in taste hairs gradually increased with the increase in number of taste bud cells.
These results show that, in addition to taste-mGluR4 and the heteromer of T1R1
and T1R3, mGluR1α may function as a receptor for glutamate (umami) taste sensation.
author:
- first_name: Takashi
full_name: Toyono, Takashi
last_name: Toyono
- first_name: Yuji
full_name: Seta, Yuji
last_name: Seta
- first_name: Shinji
full_name: Kataoka, Shinji
last_name: Kataoka
- first_name: Shintaro
full_name: Kawano, Shintaro
last_name: Kawano
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Kuniaki
full_name: Toyoshima, Kuniaki
last_name: Toyoshima
citation:
ama: Toyono T, Seta Y, Kataoka S, Kawano S, Shigemoto R, Toyoshima K. Expression
of metabotropic glutamate receptor group I in rat gustatory papillae. Cell
and Tissue Research. 2003;313(1):29-35. doi:10.1007/s00441-003-0740-2
apa: Toyono, T., Seta, Y., Kataoka, S., Kawano, S., Shigemoto, R., & Toyoshima,
K. (2003). Expression of metabotropic glutamate receptor group I in rat gustatory
papillae. Cell and Tissue Research. Springer. https://doi.org/10.1007/s00441-003-0740-2
chicago: Toyono, Takashi, Yuji Seta, Shinji Kataoka, Shintaro Kawano, Ryuichi Shigemoto,
and Kuniaki Toyoshima. “Expression of Metabotropic Glutamate Receptor Group I
in Rat Gustatory Papillae.” Cell and Tissue Research. Springer, 2003. https://doi.org/10.1007/s00441-003-0740-2.
ieee: T. Toyono, Y. Seta, S. Kataoka, S. Kawano, R. Shigemoto, and K. Toyoshima,
“Expression of metabotropic glutamate receptor group I in rat gustatory papillae,”
Cell and Tissue Research, vol. 313, no. 1. Springer, pp. 29–35, 2003.
ista: Toyono T, Seta Y, Kataoka S, Kawano S, Shigemoto R, Toyoshima K. 2003. Expression
of metabotropic glutamate receptor group I in rat gustatory papillae. Cell and
Tissue Research. 313(1), 29–35.
mla: Toyono, Takashi, et al. “Expression of Metabotropic Glutamate Receptor Group
I in Rat Gustatory Papillae.” Cell and Tissue Research, vol. 313, no. 1,
Springer, 2003, pp. 29–35, doi:10.1007/s00441-003-0740-2.
short: T. Toyono, Y. Seta, S. Kataoka, S. Kawano, R. Shigemoto, K. Toyoshima, Cell
and Tissue Research 313 (2003) 29–35.
date_created: 2018-12-11T11:58:46Z
date_published: 2003-07-01T00:00:00Z
date_updated: 2021-01-12T06:58:41Z
day: '01'
doi: 10.1007/s00441-003-0740-2
extern: 1
intvolume: ' 313'
issue: '1'
month: '07'
page: 29 - 35
publication: Cell and Tissue Research
publication_status: published
publisher: Springer
publist_id: '4267'
quality_controlled: 0
status: public
title: Expression of metabotropic glutamate receptor group I in rat gustatory papillae
type: journal_article
volume: 313
year: '2003'
...
---
_id: '2637'
abstract:
- lang: eng
text: While the cholinergic depletion in Alzheimer's disease (AD) has been known
for some time, a definitive involvement of other neurotransmitter systems has
been somewhat more elusive. Our study demonstrates a clear involvement of both
glutamatergic and, to a lesser extent, GABAergic neurons in an early onset transgenic
mouse model of AD-like amyloid pathology. Immunohistochemical staining and subsequent
quantification has revealed a statistically significant increased density of glutamatergic
and GABAergic presynaptic boutons in both the plaque free and plaque adjacent
cortical neuropile areas of transgenic mice as compared to non-transgenic controls.
Furthermore, amyloid plaque size was shown to have a statistically significant
effect on the relative area occupied by dystrophic glutamatergic neurites in the
peri-plaque neuropile. These findings support our hypothesis that the amyloid
pathology progresses in a time and neurotransmitter specific manner, first in
the cholinergic system which appears to be most vulnerable, followed by the glutamatergic
presynaptic boutons and finally the somewhat more resilient GABAergic terminals.
author:
- first_name: Karen
full_name: Bell, Karen F
last_name: Bell
- first_name: G J
full_name: De Kort, G J
last_name: De Kort
- first_name: S
full_name: Steggerda, S
last_name: Steggerda
- first_name: Ryuichi
full_name: Ryuichi Shigemoto
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Alfredo
full_name: Ribeiro-da-Silva, Alfredo
last_name: Ribeiro Da Silva
- first_name: Augusto
full_name: Cuello, Augusto C
last_name: Cuello
citation:
ama: Bell K, De Kort GJ, Steggerda S, Shigemoto R, Ribeiro Da Silva A, Cuello A.
Structural involvement of the glutamatergic presynaptic boutons in a transgenic
mouse model expressing early onset amyloid pathology. Neuroscience Letters.
2003;353(2):143-147. doi:10.1016/j.neulet.2003.09.027
apa: Bell, K., De Kort, G. J., Steggerda, S., Shigemoto, R., Ribeiro Da Silva, A.,
& Cuello, A. (2003). Structural involvement of the glutamatergic presynaptic
boutons in a transgenic mouse model expressing early onset amyloid pathology.
Neuroscience Letters. Elsevier. https://doi.org/10.1016/j.neulet.2003.09.027
chicago: Bell, Karen, G J De Kort, S Steggerda, Ryuichi Shigemoto, Alfredo Ribeiro
Da Silva, and Augusto Cuello. “Structural Involvement of the Glutamatergic Presynaptic
Boutons in a Transgenic Mouse Model Expressing Early Onset Amyloid Pathology.”
Neuroscience Letters. Elsevier, 2003. https://doi.org/10.1016/j.neulet.2003.09.027.
ieee: K. Bell, G. J. De Kort, S. Steggerda, R. Shigemoto, A. Ribeiro Da Silva, and
A. Cuello, “Structural involvement of the glutamatergic presynaptic boutons in
a transgenic mouse model expressing early onset amyloid pathology,” Neuroscience
Letters, vol. 353, no. 2. Elsevier, pp. 143–147, 2003.
ista: Bell K, De Kort GJ, Steggerda S, Shigemoto R, Ribeiro Da Silva A, Cuello A.
2003. Structural involvement of the glutamatergic presynaptic boutons in a transgenic
mouse model expressing early onset amyloid pathology. Neuroscience Letters. 353(2),
143–147.
mla: Bell, Karen, et al. “Structural Involvement of the Glutamatergic Presynaptic
Boutons in a Transgenic Mouse Model Expressing Early Onset Amyloid Pathology.”
Neuroscience Letters, vol. 353, no. 2, Elsevier, 2003, pp. 143–47, doi:10.1016/j.neulet.2003.09.027.
short: K. Bell, G.J. De Kort, S. Steggerda, R. Shigemoto, A. Ribeiro Da Silva, A.
Cuello, Neuroscience Letters 353 (2003) 143–147.
date_created: 2018-12-11T11:58:48Z
date_published: 2003-12-19T00:00:00Z
date_updated: 2021-01-12T06:58:44Z
day: '19'
doi: 10.1016/j.neulet.2003.09.027
extern: 1
intvolume: ' 353'
issue: '2'
month: '12'
page: 143 - 147
publication: Neuroscience Letters
publication_status: published
publisher: Elsevier
publist_id: '4262'
quality_controlled: 0
status: public
title: Structural involvement of the glutamatergic presynaptic boutons in a transgenic
mouse model expressing early onset amyloid pathology
type: journal_article
volume: 353
year: '2003'
...
---
_id: '2784'
abstract:
- lang: eng
text: We report the results of an experimental study of magnetohydrodynamic damping
of sidewall convection in a rectangular enclosure filled with gallium. In particular
we investigate the suppression of convection when a steady magnetic field is applied
separately in each of the three principal directions of the flow. The strongest
damping of the steady flow is found for a vertical magnetic field, which is in
agreement with theory. However, we observe that the application of a field transverse
to the flow provides greater damping than a longitudinal one, which seems to contradict
available theory. We provide a possible resolution of this apparent dichotomy
in terms of the length scale of the experiment.
author:
- first_name: Björn
full_name: Björn Hof
id: 3A374330-F248-11E8-B48F-1D18A9856A87
last_name: Hof
orcid: 0000-0003-2057-2754
- first_name: Anne
full_name: Juel, Anne
last_name: Juel
- first_name: Tom
full_name: Mullin, Tom P
last_name: Mullin
citation:
ama: Hof B, Juel A, Mullin T. Magnetohydrodynamic damping of convective flows in
molten gallium. Journal of Fluid Mechanics. 2003;482:163-179. doi:10.1017/S0022112003004014
apa: Hof, B., Juel, A., & Mullin, T. (2003). Magnetohydrodynamic damping of
convective flows in molten gallium. Journal of Fluid Mechanics. Cambridge
University Press. https://doi.org/10.1017/S0022112003004014
chicago: Hof, Björn, Anne Juel, and Tom Mullin. “Magnetohydrodynamic Damping of
Convective Flows in Molten Gallium.” Journal of Fluid Mechanics. Cambridge
University Press, 2003. https://doi.org/10.1017/S0022112003004014.
ieee: B. Hof, A. Juel, and T. Mullin, “Magnetohydrodynamic damping of convective
flows in molten gallium,” Journal of Fluid Mechanics, vol. 482. Cambridge
University Press, pp. 163–179, 2003.
ista: Hof B, Juel A, Mullin T. 2003. Magnetohydrodynamic damping of convective flows
in molten gallium. Journal of Fluid Mechanics. 482, 163–179.
mla: Hof, Björn, et al. “Magnetohydrodynamic Damping of Convective Flows in Molten
Gallium.” Journal of Fluid Mechanics, vol. 482, Cambridge University Press,
2003, pp. 163–79, doi:10.1017/S0022112003004014.
short: B. Hof, A. Juel, T. Mullin, Journal of Fluid Mechanics 482 (2003) 163–179.
date_created: 2018-12-11T11:59:35Z
date_published: 2003-05-13T00:00:00Z
date_updated: 2021-01-12T06:59:42Z
day: '13'
doi: 10.1017/S0022112003004014
extern: 1
intvolume: ' 482'
month: '05'
page: 163 - 179
publication: Journal of Fluid Mechanics
publication_status: published
publisher: Cambridge University Press
publist_id: '4105'
quality_controlled: 0
status: public
title: Magnetohydrodynamic damping of convective flows in molten gallium
type: journal_article
volume: 482
year: '2003'
...
---
_id: '2785'
abstract:
- lang: eng
text: Experimental evidence for the scaling of the finite amplitude of perturbation
theory required to promote transition in Poiseuille flow was found. The exponent
is -1 and was uncovered using considerable care in the design and execution of
the experiment. Interestingly, this exponent was also found in experiments on
transition in boundary layers.
author:
- first_name: Björn
full_name: Björn Hof
id: 3A374330-F248-11E8-B48F-1D18A9856A87
last_name: Hof
orcid: 0000-0003-2057-2754
- first_name: Anne
full_name: Juel, Anne
last_name: Juel
- first_name: Tom
full_name: Mullin, Tom P
last_name: Mullin
citation:
ama: Hof B, Juel A, Mullin T. Scaling of the turbulence transition threshold in
a pipe. Physical Review Letters. 2003;91(24):244502/1-244502/4. doi:10.1103/PhysRevLett.91.244502
apa: Hof, B., Juel, A., & Mullin, T. (2003). Scaling of the turbulence transition
threshold in a pipe. Physical Review Letters. American Physical Society.
https://doi.org/10.1103/PhysRevLett.91.244502
chicago: Hof, Björn, Anne Juel, and Tom Mullin. “Scaling of the Turbulence Transition
Threshold in a Pipe.” Physical Review Letters. American Physical Society,
2003. https://doi.org/10.1103/PhysRevLett.91.244502.
ieee: B. Hof, A. Juel, and T. Mullin, “Scaling of the turbulence transition threshold
in a pipe,” Physical Review Letters, vol. 91, no. 24. American Physical
Society, p. 244502/1-244502/4, 2003.
ista: Hof B, Juel A, Mullin T. 2003. Scaling of the turbulence transition threshold
in a pipe. Physical Review Letters. 91(24), 244502/1-244502/4.
mla: Hof, Björn, et al. “Scaling of the Turbulence Transition Threshold in a Pipe.”
Physical Review Letters, vol. 91, no. 24, American Physical Society, 2003,
p. 244502/1-244502/4, doi:10.1103/PhysRevLett.91.244502.
short: B. Hof, A. Juel, T. Mullin, Physical Review Letters 91 (2003) 244502/1-244502/4.
date_created: 2018-12-11T11:59:35Z
date_published: 2003-12-12T00:00:00Z
date_updated: 2021-01-12T06:59:42Z
day: '12'
doi: 10.1103/PhysRevLett.91.244502
extern: 1
intvolume: ' 91'
issue: '24'
month: '12'
page: 244502/1 - 244502/4
publication: Physical Review Letters
publication_status: published
publisher: American Physical Society
publist_id: '4104'
quality_controlled: 0
status: public
title: Scaling of the turbulence transition threshold in a pipe
type: journal_article
volume: 91
year: '2003'
...
---
_id: '2990'
abstract:
- lang: eng
text: Plant growth is marked by its adaptability to continuous changes in environment.
A regulated, differential distribution of auxin underlies many adaptation processes
including organogenesis, meristem patterning and tropisms. In executing its multiple
roles, auxin displays some characteristics of both a hormone and a morphogen.
Studies on auxin transport, as well as tracing the intracellular movement of its
molecular components, have suggested a possible scenario to explain how growth
plasticity is conferred at the cellular and molecular level. The plant perceives
stimuli and changes the subcellular position of auxin-transport components accordingly.
These changes modulate auxin fluxes, and the newly established auxin distribution
triggers the corresponding developmental response.
author:
- first_name: Jirí
full_name: Friml, Jirí
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
citation:
ama: Friml J. Auxin transport - Shaping the plant. Current Opinion in Plant Biology.
2003;6(1):7-12. doi:10.1016/S1369526602000031
apa: Friml, J. (2003). Auxin transport - Shaping the plant. Current Opinion in
Plant Biology. Elsevier. https://doi.org/10.1016/S1369526602000031
chicago: Friml, Jiří. “Auxin Transport - Shaping the Plant.” Current Opinion
in Plant Biology. Elsevier, 2003. https://doi.org/10.1016/S1369526602000031.
ieee: J. Friml, “Auxin transport - Shaping the plant,” Current Opinion in Plant
Biology, vol. 6, no. 1. Elsevier, pp. 7–12, 2003.
ista: Friml J. 2003. Auxin transport - Shaping the plant. Current Opinion in Plant
Biology. 6(1), 7–12.
mla: Friml, Jiří. “Auxin Transport - Shaping the Plant.” Current Opinion in Plant
Biology, vol. 6, no. 1, Elsevier, 2003, pp. 7–12, doi:10.1016/S1369526602000031.
short: J. Friml, Current Opinion in Plant Biology 6 (2003) 7–12.
date_created: 2018-12-11T12:00:43Z
date_published: 2003-02-01T00:00:00Z
date_updated: 2021-01-12T07:40:17Z
day: '01'
doi: 10.1016/S1369526602000031
extern: '1'
intvolume: ' 6'
issue: '1'
language:
- iso: eng
month: '02'
oa_version: None
page: 7 - 12
publication: Current Opinion in Plant Biology
publication_status: published
publisher: Elsevier
publist_id: '3711'
quality_controlled: '1'
status: public
title: Auxin transport - Shaping the plant
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 6
year: '2003'
...
---
_id: '2992'
abstract:
- lang: eng
text: Plants have many polarized cell types, but relatively little is known about
the mechanisms that establish polarity. The orc mutant was identified originally
by defects in root patterning, and positional cloning revealed that the affected
gene encodes STEROL METHYLTRANSFERASE1, which is required for the appropriate
synthesis and composition of major membrane sterols. smt1orc mutants displayed
several conspicuous cell polarity defects. Columella root cap cells revealed perturbed
polar positioning of different organelles, and in the smt1orc root epidermis,
polar initiation of root hairs was more randomized. Polar auxin transport and
expression of the auxin reporter DR5-β-glucuronidase were aberrant in smt1orc.
Patterning defects in smt1orc resembled those observed in mutants of the PIN gene
family of putative auxin efflux transporters. Consistently, the membrane localization
of the PIN1 and PIN3 proteins was disturbed in smt1orc, whereas polar positioning
of the influx carrier AUX1 appeared normal. Our results suggest that balanced
sterol composition is a major requirement for cell polarity and auxin efflux in
Arabidopsis.
author:
- first_name: Viola
full_name: Willemsen, Viola
last_name: Willemsen
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
- first_name: Markus
full_name: Grebe, Markus
last_name: Grebe
- first_name: Albert
full_name: Van Den Toorn, Albert
last_name: Van Den Toorn
- first_name: Klaus
full_name: Palme, Klaus
last_name: Palme
- first_name: Ben
full_name: Scheres, Ben
last_name: Scheres
citation:
ama: Willemsen V, Friml J, Grebe M, Van Den Toorn A, Palme K, Scheres B. Cell polarity
and PIN protein positioning in Arabidopsis require STEROL METHYLTRANSFERASE1 function.
Plant Cell. 2003;15(3):612-625. doi:10.1105/tpc.008433
apa: Willemsen, V., Friml, J., Grebe, M., Van Den Toorn, A., Palme, K., & Scheres,
B. (2003). Cell polarity and PIN protein positioning in Arabidopsis require STEROL
METHYLTRANSFERASE1 function. Plant Cell. American Society of Plant Biologists.
https://doi.org/10.1105/tpc.008433
chicago: Willemsen, Viola, Jiří Friml, Markus Grebe, Albert Van Den Toorn, Klaus
Palme, and Ben Scheres. “Cell Polarity and PIN Protein Positioning in Arabidopsis
Require STEROL METHYLTRANSFERASE1 Function.” Plant Cell. American Society
of Plant Biologists, 2003. https://doi.org/10.1105/tpc.008433.
ieee: V. Willemsen, J. Friml, M. Grebe, A. Van Den Toorn, K. Palme, and B. Scheres,
“Cell polarity and PIN protein positioning in Arabidopsis require STEROL METHYLTRANSFERASE1
function,” Plant Cell, vol. 15, no. 3. American Society of Plant Biologists,
pp. 612–625, 2003.
ista: Willemsen V, Friml J, Grebe M, Van Den Toorn A, Palme K, Scheres B. 2003.
Cell polarity and PIN protein positioning in Arabidopsis require STEROL METHYLTRANSFERASE1
function. Plant Cell. 15(3), 612–625.
mla: Willemsen, Viola, et al. “Cell Polarity and PIN Protein Positioning in Arabidopsis
Require STEROL METHYLTRANSFERASE1 Function.” Plant Cell, vol. 15, no. 3,
American Society of Plant Biologists, 2003, pp. 612–25, doi:10.1105/tpc.008433.
short: V. Willemsen, J. Friml, M. Grebe, A. Van Den Toorn, K. Palme, B. Scheres,
Plant Cell 15 (2003) 612–625.
date_created: 2018-12-11T12:00:44Z
date_published: 2003-03-01T00:00:00Z
date_updated: 2021-01-12T07:40:18Z
day: '01'
doi: 10.1105/tpc.008433
extern: 1
intvolume: ' 15'
issue: '3'
month: '03'
page: 612 - 625
publication: Plant Cell
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '3710'
quality_controlled: 0
status: public
title: Cell polarity and PIN protein positioning in Arabidopsis require STEROL METHYLTRANSFERASE1
function
type: journal_article
volume: 15
year: '2003'
...
---
_id: '2996'
abstract:
- lang: eng
text: |
Plants, compared to animals, exhibit an amazing adaptability and plasticity in their development. This is largely dependent on the ability of plants to form new organs, such as lateral roots, leaves, and flowers during postembryonic development. Organ primordia develop from founder cell populations into organs by coordinated cell division and differentiation. Here, we show that organ formation in Arabidopsis involves dynamic gradients of the signaling molecule auxin with maxima at the primordia tips. These gradients are mediated by cellular efflux requiring asymmetrically localized PIN proteins, which represent a functionally redundant network for auxin distribution in both aerial and underground organs. PIN1 polar localization undergoes a dynamic rearrangement, which correlates with establishment of auxin gradients and primordium development. Our results suggest that PIN-dependent, local auxin gradients represent a common module for formation of all plant organs, regardless of their mature morphology or developmental origin.
author:
- first_name: Eva
full_name: Eva Benková
id: 38F4F166-F248-11E8-B48F-1D18A9856A87
last_name: Benková
orcid: 0000-0002-8510-9739
- first_name: Marta
full_name: Michniewicz, Marta
last_name: Michniewicz
- first_name: Michael
full_name: Sauer, Michael
last_name: Sauer
- first_name: Thomas
full_name: Teichmann, Thomas
last_name: Teichmann
- first_name: Daniela
full_name: Seifertová, Daniela
last_name: Seifertová
- first_name: Gerd
full_name: Jürgens, Gerd
last_name: Jürgens
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
citation:
ama: Benková E, Michniewicz M, Sauer M, et al. Local, efflux-dependent auxin gradients
as a common module for plant organ formation. Cell. 2003;115(5):591-602.
doi:10.1016/S0092-8674(03)00924-3
apa: Benková, E., Michniewicz, M., Sauer, M., Teichmann, T., Seifertová, D., Jürgens,
G., & Friml, J. (2003). Local, efflux-dependent auxin gradients as a common
module for plant organ formation. Cell. Cell Press. https://doi.org/10.1016/S0092-8674(03)00924-3
chicago: Benková, Eva, Marta Michniewicz, Michael Sauer, Thomas Teichmann, Daniela
Seifertová, Gerd Jürgens, and Jiří Friml. “Local, Efflux-Dependent Auxin Gradients
as a Common Module for Plant Organ Formation.” Cell. Cell Press, 2003.
https://doi.org/10.1016/S0092-8674(03)00924-3.
ieee: E. Benková et al., “Local, efflux-dependent auxin gradients as a common
module for plant organ formation,” Cell, vol. 115, no. 5. Cell Press, pp.
591–602, 2003.
ista: Benková E, Michniewicz M, Sauer M, Teichmann T, Seifertová D, Jürgens G, Friml
J. 2003. Local, efflux-dependent auxin gradients as a common module for plant
organ formation. Cell. 115(5), 591–602.
mla: Benková, Eva, et al. “Local, Efflux-Dependent Auxin Gradients as a Common Module
for Plant Organ Formation.” Cell, vol. 115, no. 5, Cell Press, 2003, pp.
591–602, doi:10.1016/S0092-8674(03)00924-3.
short: E. Benková, M. Michniewicz, M. Sauer, T. Teichmann, D. Seifertová, G. Jürgens,
J. Friml, Cell 115 (2003) 591–602.
date_created: 2018-12-11T12:00:46Z
date_published: 2003-11-26T00:00:00Z
date_updated: 2021-01-12T07:40:19Z
day: '26'
doi: 10.1016/S0092-8674(03)00924-3
extern: 1
intvolume: ' 115'
issue: '5'
month: '11'
page: 591 - 602
publication: Cell
publication_status: published
publisher: Cell Press
publist_id: '3706'
quality_controlled: 0
status: public
title: Local, efflux-dependent auxin gradients as a common module for plant organ
formation
type: journal_article
volume: 115
year: '2003'
...
---
_id: '2995'
abstract:
- lang: eng
text: |
Axis formation occurs in plants, as in animals, during early embryogenesis. However, the underlying mechanism is not known. Here we show that the first manifestation of the apical-basal axis in plants, the asymmetric division of the zygote, produces a basal cell that transports and an apical cell that responds to the signalling molecule auxin. This apical-basal auxin activity gradient triggers the specification of apical embryo structures and is actively maintained by a novel component of auxin efflux, PIN7, which is located apically in the basal cell. Later, the developmentally regulated reversal of PIN7 and onset of PIN1 polar localization reorganize the auxin gradient for specification of the basal root pole. An analysis of pin quadruple mutants identifies PIN-dependent transport as an essential part of the mechanism for embryo axis formation. Our results indicate how the establishment of cell polarity, polar auxin efflux and local auxin response result in apical-basal axis formation of the embryo, and thus determine the axiality of the adult plant.
author:
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
- first_name: Anne
full_name: Vieten, Anne
last_name: Vieten
- first_name: Michael
full_name: Sauer, Michael
last_name: Sauer
- first_name: Dolf
full_name: Weijers, Dolf
last_name: Weijers
- first_name: Heinz
full_name: Schwarz, Heinz
last_name: Schwarz
- first_name: Thorsten
full_name: Hamann, Thorsten
last_name: Hamann
- first_name: Remko
full_name: Offringa, Remko
last_name: Offringa
- first_name: Gerd
full_name: Jürgens, Gerd
last_name: Jürgens
citation:
ama: Friml J, Vieten A, Sauer M, et al. Efflux dependent auxin gradients establish
the apical basal axis of Arabidopsis. Nature. 2003;426(6963):147-153. doi:10.1038/nature02085
apa: Friml, J., Vieten, A., Sauer, M., Weijers, D., Schwarz, H., Hamann, T., … Jürgens,
G. (2003). Efflux dependent auxin gradients establish the apical basal axis of
Arabidopsis. Nature. Nature Publishing Group. https://doi.org/10.1038/nature02085
chicago: Friml, Jiří, Anne Vieten, Michael Sauer, Dolf Weijers, Heinz Schwarz, Thorsten
Hamann, Remko Offringa, and Gerd Jürgens. “Efflux Dependent Auxin Gradients Establish
the Apical Basal Axis of Arabidopsis.” Nature. Nature Publishing Group,
2003. https://doi.org/10.1038/nature02085.
ieee: J. Friml et al., “Efflux dependent auxin gradients establish the apical
basal axis of Arabidopsis,” Nature, vol. 426, no. 6963. Nature Publishing
Group, pp. 147–153, 2003.
ista: Friml J, Vieten A, Sauer M, Weijers D, Schwarz H, Hamann T, Offringa R, Jürgens
G. 2003. Efflux dependent auxin gradients establish the apical basal axis of Arabidopsis.
Nature. 426(6963), 147–153.
mla: Friml, Jiří, et al. “Efflux Dependent Auxin Gradients Establish the Apical
Basal Axis of Arabidopsis.” Nature, vol. 426, no. 6963, Nature Publishing
Group, 2003, pp. 147–53, doi:10.1038/nature02085.
short: J. Friml, A. Vieten, M. Sauer, D. Weijers, H. Schwarz, T. Hamann, R. Offringa,
G. Jürgens, Nature 426 (2003) 147–153.
date_created: 2018-12-11T12:00:45Z
date_published: 2003-11-13T00:00:00Z
date_updated: 2021-01-12T07:40:19Z
day: '13'
doi: 10.1038/nature02085
extern: 1
intvolume: ' 426'
issue: '6963'
month: '11'
page: 147 - 153
publication: Nature
publication_status: published
publisher: Nature Publishing Group
publist_id: '3708'
quality_controlled: 0
status: public
title: Efflux dependent auxin gradients establish the apical basal axis of Arabidopsis
type: journal_article
volume: 426
year: '2003'
...
---
_id: '2994'
abstract:
- lang: eng
text: The regular arrangement of leaves around a plant's stem, called phyllotaxis,
has for centuries attracted the attention of philosophers, mathematicians and
natural scientists; however, to date, studies of phyllotaxis have been largely
theoretical. Leaves and flowers are formed from the shoot apical meristem, triggered
by the plant hormone auxin. Auxin is transported through plant tissues by specific
cellular influx and efflux carrier proteins. Here we show that proteins involved
in auxin transport regulate phyllotaxis. Our data indicate that auxin is transported
upwards into the meristem through the epidermis and the outermost meristem cell
layer. Existing leaf primordia act as sinks, redistributing auxin and creating
its heterogeneous distribution in the meristem. Auxin accumulation occurs only
at certain minimal distances from existing primordia, defining the position of
future primordia. This model for phyllotaxis accounts for its reiterative nature,
as well as its regularity and stability.
author:
- first_name: Didier
full_name: Reinhardt, Didier
last_name: Reinhardt
- first_name: Eva
full_name: Pesce, Eva-Rachele
last_name: Pesce
- first_name: Pia
full_name: Stieger, Pia
last_name: Stieger
- first_name: Therese
full_name: Mandel, Therese
last_name: Mandel
- first_name: Kurt
full_name: Baltensperger, Kurt
last_name: Baltensperger
- first_name: Malcolm
full_name: Bennett, Malcolm
last_name: Bennett
- first_name: Jan
full_name: Traas, Jan
last_name: Traas
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
- first_name: Cris
full_name: Kuhlemeier, Cris
last_name: Kuhlemeier
citation:
ama: Reinhardt D, Pesce E, Stieger P, et al. Regulation of phyllotaxis by polar
auxin transport. Nature. 2003;426(6964):255-260. doi:10.1038/nature02081
apa: Reinhardt, D., Pesce, E., Stieger, P., Mandel, T., Baltensperger, K., Bennett,
M., … Kuhlemeier, C. (2003). Regulation of phyllotaxis by polar auxin transport.
Nature. Nature Publishing Group. https://doi.org/10.1038/nature02081
chicago: Reinhardt, Didier, Eva Pesce, Pia Stieger, Therese Mandel, Kurt Baltensperger,
Malcolm Bennett, Jan Traas, Jiří Friml, and Cris Kuhlemeier. “Regulation of Phyllotaxis
by Polar Auxin Transport.” Nature. Nature Publishing Group, 2003. https://doi.org/10.1038/nature02081.
ieee: D. Reinhardt et al., “Regulation of phyllotaxis by polar auxin transport,”
Nature, vol. 426, no. 6964. Nature Publishing Group, pp. 255–260, 2003.
ista: Reinhardt D, Pesce E, Stieger P, Mandel T, Baltensperger K, Bennett M, Traas
J, Friml J, Kuhlemeier C. 2003. Regulation of phyllotaxis by polar auxin transport.
Nature. 426(6964), 255–260.
mla: Reinhardt, Didier, et al. “Regulation of Phyllotaxis by Polar Auxin Transport.”
Nature, vol. 426, no. 6964, Nature Publishing Group, 2003, pp. 255–60,
doi:10.1038/nature02081.
short: D. Reinhardt, E. Pesce, P. Stieger, T. Mandel, K. Baltensperger, M. Bennett,
J. Traas, J. Friml, C. Kuhlemeier, Nature 426 (2003) 255–260.
date_created: 2018-12-11T12:00:45Z
date_published: 2003-11-20T00:00:00Z
date_updated: 2021-01-12T07:40:18Z
day: '20'
doi: 10.1038/nature02081
extern: 1
intvolume: ' 426'
issue: '6964'
month: '11'
page: 255 - 260
publication: Nature
publication_status: published
publisher: Nature Publishing Group
publist_id: '3707'
quality_controlled: 0
status: public
title: Regulation of phyllotaxis by polar auxin transport
type: journal_article
volume: 426
year: '2003'
...
---
_id: '2993'
abstract:
- lang: eng
text: Plant biology is currently experiencing a growing demand for easy and reliable
mRNA and protein localisation techniques. Here, we present novel whole mount in
situ hybridisation and immunolocalisation protocols, suitable to localise mRNAs
and proteins in Arabidopsis seedlings. We demonstrate that these methods can be
used in different organs of Arabidopsis seedlings as well as in other plant species.
In order to achieve better reproducibility and higher throughput, we modified
these protocols for automation to be performed by a liquid handling robot. In
addition, we show that other procedures such as reporter enzyme assays and tissue
clearing can be similarly automated. We present examples of application of our
protocols including mRNA localisation and proteins and epitope tag (co)localisations
which demonstrate that these methods provide reliable and versatile tools for
expression, localisation and anatomical studies in plants.
author:
- first_name: Jirí
full_name: Jirí Friml
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
- first_name: Eva
full_name: Eva Benková
id: 38F4F166-F248-11E8-B48F-1D18A9856A87
last_name: Benková
orcid: 0000-0002-8510-9739
- first_name: Ulrike
full_name: Mayer, Ulrike
last_name: Mayer
- first_name: Klaus
full_name: Palme, Klaus
last_name: Palme
- first_name: Gerhard
full_name: Muster, Gerhard
last_name: Muster
citation:
ama: Friml J, Benková E, Mayer U, Palme K, Muster G. Automated whole mount localisation
techniques for plant seedlings. Plant Journal. 2003;34(1):115-124. doi:10.1046/j.1365-313X.2003.01705.x
apa: Friml, J., Benková, E., Mayer, U., Palme, K., & Muster, G. (2003). Automated
whole mount localisation techniques for plant seedlings. Plant Journal.
Wiley-Blackwell. https://doi.org/10.1046/j.1365-313X.2003.01705.x
chicago: Friml, Jiří, Eva Benková, Ulrike Mayer, Klaus Palme, and Gerhard Muster.
“Automated Whole Mount Localisation Techniques for Plant Seedlings.” Plant
Journal. Wiley-Blackwell, 2003. https://doi.org/10.1046/j.1365-313X.2003.01705.x.
ieee: J. Friml, E. Benková, U. Mayer, K. Palme, and G. Muster, “Automated whole
mount localisation techniques for plant seedlings,” Plant Journal, vol.
34, no. 1. Wiley-Blackwell, pp. 115–124, 2003.
ista: Friml J, Benková E, Mayer U, Palme K, Muster G. 2003. Automated whole mount
localisation techniques for plant seedlings. Plant Journal. 34(1), 115–124.
mla: Friml, Jiří, et al. “Automated Whole Mount Localisation Techniques for Plant
Seedlings.” Plant Journal, vol. 34, no. 1, Wiley-Blackwell, 2003, pp. 115–24,
doi:10.1046/j.1365-313X.2003.01705.x.
short: J. Friml, E. Benková, U. Mayer, K. Palme, G. Muster, Plant Journal 34 (2003)
115–124.
date_created: 2018-12-11T12:00:44Z
date_published: 2003-04-01T00:00:00Z
date_updated: 2021-01-12T07:40:18Z
day: '01'
doi: 10.1046/j.1365-313X.2003.01705.x
extern: 1
intvolume: ' 34'
issue: '1'
month: '04'
page: 115 - 124
publication: Plant Journal
publication_status: published
publisher: Wiley-Blackwell
publist_id: '3709'
quality_controlled: 0
status: public
title: Automated whole mount localisation techniques for plant seedlings
type: journal_article
volume: 34
year: '2003'
...
---
_id: '3151'
abstract:
- lang: eng
text: Biosynthesis of most peptide hormones and neuropeptides requires proteolytic
excision of the active peptide from inactive proprotein precursors, an activity
carried out by subtilisin-like proprotein convertases (SPCs) in constitutive or
regulated secretory pathways. The Drosophila amontillado (amon) gene encodes a
homolog of the mammalian PC2 protein, an SPC that functions in the regulated secretory
pathway in neuroendocrine tissues. We have identified amon mutants by isolating
ethylmethanesulfonate (EMS)-induced lethal and visible mutations that define two
complementation groups in the amon interval at 97D1 of the third chromosome. DNA
sequencing identified the amon complementation group and the DNA sequence change
for each of the nine amon alleles isolated. amon mutants display partial embryonic
lethality, are defective in larval growth, and arrest during the first to second
instar larval molt. Mutant larvae can be rescued by heat-shock-induced expression
of the amon protein. Rescued larvae arrest at the subsequent larval molt, suggesting
that amon is also required for the second to third instar larval molt. Our data
indicate that the amon proprotein convertase is required during embryogenesis
and larval development in Drosophila and support the hypothesis that AMON acts
to proteolytically process peptide hormones that regulate hatching, larval growth,
and larval ecdysis.
author:
- first_name: Lowell
full_name: Rayburn, Lowell Y
last_name: Rayburn
- first_name: Holly
full_name: Gooding, Holly C
last_name: Gooding
- first_name: Semil
full_name: Choksi, Semil P
last_name: Choksi
- first_name: Dhea
full_name: Maloney, Dhea
last_name: Maloney
- first_name: Ambrose
full_name: Kidd, Ambrose R
last_name: Kidd
- first_name: Daria E
full_name: Daria Siekhaus
id: 3D224B9E-F248-11E8-B48F-1D18A9856A87
last_name: Siekhaus
orcid: 0000-0001-8323-8353
- first_name: Michael
full_name: Bender, Michael
last_name: Bender
citation:
ama: Rayburn L, Gooding H, Choksi S, et al. Amontillado, the Drosophila homolog
of the prohormone processing protease PC2, is required during embryogenesis and
early larval development. Genetics. 2003;163(1):227-237.
apa: Rayburn, L., Gooding, H., Choksi, S., Maloney, D., Kidd, A., Siekhaus, D. E.,
& Bender, M. (2003). Amontillado, the Drosophila homolog of the prohormone
processing protease PC2, is required during embryogenesis and early larval development.
Genetics. Genetics Society of America.
chicago: Rayburn, Lowell, Holly Gooding, Semil Choksi, Dhea Maloney, Ambrose Kidd,
Daria E Siekhaus, and Michael Bender. “Amontillado, the Drosophila Homolog of
the Prohormone Processing Protease PC2, Is Required during Embryogenesis and Early
Larval Development.” Genetics. Genetics Society of America, 2003.
ieee: L. Rayburn et al., “Amontillado, the Drosophila homolog of the prohormone
processing protease PC2, is required during embryogenesis and early larval development,”
Genetics, vol. 163, no. 1. Genetics Society of America, pp. 227–237, 2003.
ista: Rayburn L, Gooding H, Choksi S, Maloney D, Kidd A, Siekhaus DE, Bender M.
2003. Amontillado, the Drosophila homolog of the prohormone processing protease
PC2, is required during embryogenesis and early larval development. Genetics.
163(1), 227–237.
mla: Rayburn, Lowell, et al. “Amontillado, the Drosophila Homolog of the Prohormone
Processing Protease PC2, Is Required during Embryogenesis and Early Larval Development.”
Genetics, vol. 163, no. 1, Genetics Society of America, 2003, pp. 227–37.
short: L. Rayburn, H. Gooding, S. Choksi, D. Maloney, A. Kidd, D.E. Siekhaus, M.
Bender, Genetics 163 (2003) 227–237.
date_created: 2018-12-11T12:01:41Z
date_published: 2003-01-01T00:00:00Z
date_updated: 2021-01-12T07:41:25Z
day: '01'
extern: 1
intvolume: ' 163'
issue: '1'
month: '01'
page: 227 - 237
publication: Genetics
publication_status: published
publisher: Genetics Society of America
publist_id: '3545'
quality_controlled: 0
status: public
title: Amontillado, the Drosophila homolog of the prohormone processing protease PC2,
is required during embryogenesis and early larval development
type: journal_article
volume: 163
year: '2003'
...
---
_id: '3150'
abstract:
- lang: eng
text: Tripartite G-protein-coupled receptors (GPCRs) represent one of the largest
groups of signal transducers, transmitting signals from hormones, neuropeptides,
odorants, food and light. Ligand-bound receptors catalyse GDP/GTP exchange on
the G-protein α-subunit (Gα), leading to α-GTP separation from the βγ subunits
and pathway activation. Activating mutations in the receptors or G proteins underlie
many human diseases, including some cancers, dwarfism and premature puberty. Regulators
of G-protein signalling (RGS proteins) are known to modulate the level and duration
of ligand-induced signalling by accelerating the intrinsic GTPase activity of
the Gα subunit, and thus reformation of the inactive GDP-bound Gα. Here we find
that even in the absence of receptor, mutation of the RGS family member Sst2 (refs
6-9) permits spontaneous activation of the G-protein-coupled mating pathway in
Saccharomyces cerevisiae at levels normally seen only in the presence of ligand.
Our work demonstrates the occurence of spontaneous tripartite G-protein signalling
in vivo and identifies a requirement for RGS proteins in preventing such receptor-independent
activation.
author:
- first_name: Daria E
full_name: Daria Siekhaus
id: 3D224B9E-F248-11E8-B48F-1D18A9856A87
last_name: Siekhaus
orcid: 0000-0001-8323-8353
- first_name: David
full_name: Drubin, David G
last_name: Drubin
citation:
ama: Siekhaus DE, Drubin D. Spontaneous receptor-independent heterotrimeric G-protein
signalling in an RGS mutant. Nature Cell Biology. 2003;5(3):231-235. doi:10.1038/ncb941
apa: Siekhaus, D. E., & Drubin, D. (2003). Spontaneous receptor-independent
heterotrimeric G-protein signalling in an RGS mutant. Nature Cell Biology.
Nature Publishing Group. https://doi.org/10.1038/ncb941
chicago: Siekhaus, Daria E, and David Drubin. “Spontaneous Receptor-Independent
Heterotrimeric G-Protein Signalling in an RGS Mutant.” Nature Cell Biology.
Nature Publishing Group, 2003. https://doi.org/10.1038/ncb941.
ieee: D. E. Siekhaus and D. Drubin, “Spontaneous receptor-independent heterotrimeric
G-protein signalling in an RGS mutant,” Nature Cell Biology, vol. 5, no.
3. Nature Publishing Group, pp. 231–235, 2003.
ista: Siekhaus DE, Drubin D. 2003. Spontaneous receptor-independent heterotrimeric
G-protein signalling in an RGS mutant. Nature Cell Biology. 5(3), 231–235.
mla: Siekhaus, Daria E., and David Drubin. “Spontaneous Receptor-Independent Heterotrimeric
G-Protein Signalling in an RGS Mutant.” Nature Cell Biology, vol. 5, no.
3, Nature Publishing Group, 2003, pp. 231–35, doi:10.1038/ncb941.
short: D.E. Siekhaus, D. Drubin, Nature Cell Biology 5 (2003) 231–235.
date_created: 2018-12-11T12:01:41Z
date_published: 2003-03-01T00:00:00Z
date_updated: 2021-01-12T07:41:24Z
day: '01'
doi: 10.1038/ncb941
extern: 1
intvolume: ' 5'
issue: '3'
month: '03'
page: 231 - 235
publication: Nature Cell Biology
publication_status: published
publisher: Nature Publishing Group
publist_id: '3544'
quality_controlled: 0
status: public
title: Spontaneous receptor-independent heterotrimeric G-protein signalling in an
RGS mutant
type: journal_article
volume: 5
year: '2003'
...
---
_id: '3209'
abstract:
- lang: eng
text: We show that the fixed alphabet shortest common supersequence (SCS) and the
fixed alphabet longest common subsequence (LCS) problems parameterized in the
number of strings are W[1]-hard. Unless W[1]=FPT, this rules out the existence
of algorithms with time complexity of O(f(k)nα) for those problems. Here n is
the size of the problem instance, α is constant, k is the number of strings and
f is any function of k. The fixed alphabet version of the LCS problem is of particular
interest considering the importance of sequence comparison (e.g. multiple sequence
alignment) in the fixed length alphabet world of DNA and protein sequences.
author:
- first_name: Krzysztof Z
full_name: Krzysztof Pietrzak
id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87
last_name: Pietrzak
orcid: 0000-0002-9139-1654
citation:
ama: Pietrzak KZ. On the parameterized complexity of the fixed alphabet shortest
common supersequence and longest common subsequence problems. Journal of Computer
and System Sciences. 2003;67(4):757-771. doi:10.1016/S0022-0000(03)00078-3
apa: Pietrzak, K. Z. (2003). On the parameterized complexity of the fixed alphabet
shortest common supersequence and longest common subsequence problems. Journal
of Computer and System Sciences. Elsevier. https://doi.org/10.1016/S0022-0000(03)00078-3
chicago: Pietrzak, Krzysztof Z. “On the Parameterized Complexity of the Fixed Alphabet
Shortest Common Supersequence and Longest Common Subsequence Problems.” Journal
of Computer and System Sciences. Elsevier, 2003. https://doi.org/10.1016/S0022-0000(03)00078-3.
ieee: K. Z. Pietrzak, “On the parameterized complexity of the fixed alphabet shortest
common supersequence and longest common subsequence problems,” Journal of Computer
and System Sciences, vol. 67, no. 4. Elsevier, pp. 757–771, 2003.
ista: Pietrzak KZ. 2003. On the parameterized complexity of the fixed alphabet shortest
common supersequence and longest common subsequence problems. Journal of Computer
and System Sciences. 67(4), 757–771.
mla: Pietrzak, Krzysztof Z. “On the Parameterized Complexity of the Fixed Alphabet
Shortest Common Supersequence and Longest Common Subsequence Problems.” Journal
of Computer and System Sciences, vol. 67, no. 4, Elsevier, 2003, pp. 757–71,
doi:10.1016/S0022-0000(03)00078-3.
short: K.Z. Pietrzak, Journal of Computer and System Sciences 67 (2003) 757–771.
date_created: 2018-12-11T12:02:01Z
date_published: 2003-12-01T00:00:00Z
date_updated: 2021-01-12T07:41:49Z
day: '01'
doi: 10.1016/S0022-0000(03)00078-3
extern: 1
intvolume: ' 67'
issue: '4'
month: '12'
page: 757 - 771
publication: Journal of Computer and System Sciences
publication_status: published
publisher: Elsevier
publist_id: '3472'
quality_controlled: 0
status: public
title: On the parameterized complexity of the fixed alphabet shortest common supersequence
and longest common subsequence problems
type: journal_article
volume: 67
year: '2003'
...
---
_id: '3210'
abstract:
- lang: eng
text: 'Luby and Rackoff showed how to construct a (super-)pseudo-random permutation
{0,1}2n→ {0,1}2n from some number r of pseudo-random functions {0,1}n → {0,1}n.
Their construction, motivated by DES, consists of a cascade of r Feistel permutations.
A Feistel permutation 1for a pseudo-random function f is defined as (L, R) → (R,L
⊕ f (R)), where L and R are the left and right part of the input and ⊕ denotes
bitwise XOR or, in this paper, any other group operation on {0,1}n. The only non-trivial
step of the security proof consists of proving that the cascade of r Feistel permutations
with independent uniform random functions {0,1}n → {0,1}n, denoted Ψ2nr is indistinguishable
from a uniform random permutation {0,1}2n → {0,1}2n by any computationally unbounded
adaptive distinguisher making at most O(2cn) combined chosen plaintext/ciphertext
queries for any c < α, where a is a security parameter. Luby and Rackoff proved
α = 1/2 for r = 4. A natural problem, proposed by Pieprzyk is to improve on α
for larger r. The best known result, α = 3/4 for r = 6, is due to Patarin. In
this paper we prove a = 1 -O(1/r), i.e., the trivial upper bound α = 1 can be
approached. The proof uses some new techniques that can be of independent interest. '
alternative_title:
- LNCS
author:
- first_name: Ueli
full_name: Maurer, Ueli M
last_name: Maurer
- first_name: Krzysztof Z
full_name: Krzysztof Pietrzak
id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87
last_name: Pietrzak
orcid: 0000-0002-9139-1654
citation:
ama: 'Maurer U, Pietrzak KZ. The security of many round Luby Rackoff pseudo random
permutations. In: Vol 2656. Springer; 2003:544-561. doi:10.1007/3-540-39200-9_34'
apa: 'Maurer, U., & Pietrzak, K. Z. (2003). The security of many round Luby
Rackoff pseudo random permutations (Vol. 2656, pp. 544–561). Presented at the
EUROCRYPT: Theory and Applications of Cryptographic Techniques, Springer. https://doi.org/10.1007/3-540-39200-9_34'
chicago: Maurer, Ueli, and Krzysztof Z Pietrzak. “The Security of Many Round Luby
Rackoff Pseudo Random Permutations,” 2656:544–61. Springer, 2003. https://doi.org/10.1007/3-540-39200-9_34.
ieee: 'U. Maurer and K. Z. Pietrzak, “The security of many round Luby Rackoff pseudo
random permutations,” presented at the EUROCRYPT: Theory and Applications of Cryptographic
Techniques, 2003, vol. 2656, pp. 544–561.'
ista: 'Maurer U, Pietrzak KZ. 2003. The security of many round Luby Rackoff pseudo
random permutations. EUROCRYPT: Theory and Applications of Cryptographic Techniques,
LNCS, vol. 2656, 544–561.'
mla: Maurer, Ueli, and Krzysztof Z. Pietrzak. The Security of Many Round Luby
Rackoff Pseudo Random Permutations. Vol. 2656, Springer, 2003, pp. 544–61,
doi:10.1007/3-540-39200-9_34.
short: U. Maurer, K.Z. Pietrzak, in:, Springer, 2003, pp. 544–561.
conference:
name: 'EUROCRYPT: Theory and Applications of Cryptographic Techniques'
date_created: 2018-12-11T12:02:02Z
date_published: 2003-06-04T00:00:00Z
date_updated: 2021-01-12T07:41:49Z
day: '04'
doi: 10.1007/3-540-39200-9_34
extern: 1
intvolume: ' 2656'
month: '06'
page: 544 - 561
publication_status: published
publisher: Springer
publist_id: '3473'
quality_controlled: 0
status: public
title: The security of many round Luby Rackoff pseudo random permutations
type: conference
volume: 2656
year: '2003'
...
---
_id: '3425'
alternative_title:
- Nato Science Series II
article_processing_charge: No
author:
- first_name: Mark Tobias
full_name: Bollenbach, Mark Tobias
id: 3E6DB97A-F248-11E8-B48F-1D18A9856A87
last_name: Bollenbach
orcid: 0000-0003-4398-476X
- first_name: T.
full_name: Strother, T.
last_name: Strother
- first_name: Wolfgang
full_name: Bauer, Wolfgang
last_name: Bauer
citation:
ama: 'Bollenbach MT, Strother T, Bauer W. 3D supernova collapse calculations. In:
Vol 166. Springer; 2003:277-288. doi:10.1007/978-1-4020-2705-5_21'
apa: Bollenbach, M. T., Strother, T., & Bauer, W. (2003). 3D supernova collapse
calculations (Vol. 166, pp. 277–288). Presented at the NATO ASI on Structure and
Dynamics of Elementary Matter, Springer. https://doi.org/10.1007/978-1-4020-2705-5_21
chicago: Bollenbach, Mark Tobias, T. Strother, and Wolfgang Bauer. “3D Supernova
Collapse Calculations,” 166:277–88. Springer, 2003. https://doi.org/10.1007/978-1-4020-2705-5_21.
ieee: M. T. Bollenbach, T. Strother, and W. Bauer, “3D supernova collapse calculations,”
presented at the NATO ASI on Structure and Dynamics of Elementary Matter, 2003,
vol. 166, pp. 277–288.
ista: Bollenbach MT, Strother T, Bauer W. 2003. 3D supernova collapse calculations.
NATO ASI on Structure and Dynamics of Elementary Matter, Nato Science Series II,
vol. 166, 277–288.
mla: Bollenbach, Mark Tobias, et al. 3D Supernova Collapse Calculations.
Vol. 166, Springer, 2003, pp. 277–88, doi:10.1007/978-1-4020-2705-5_21.
short: M.T. Bollenbach, T. Strother, W. Bauer, in:, Springer, 2003, pp. 277–288.
conference:
name: NATO ASI on Structure and Dynamics of Elementary Matter
date_created: 2018-12-11T12:03:16Z
date_published: 2003-01-01T00:00:00Z
date_updated: 2021-01-12T07:43:23Z
day: '01'
doi: 10.1007/978-1-4020-2705-5_21
extern: '1'
intvolume: ' 166'
language:
- iso: eng
month: '01'
oa_version: None
page: 277 - 288
publication_status: published
publisher: Springer
publist_id: '2976'
status: public
title: 3D supernova collapse calculations
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 166
year: '2003'
...
---
_id: '3458'
author:
- first_name: Peter M
full_name: Peter Jonas
id: 353C1B58-F248-11E8-B48F-1D18A9856A87
last_name: Jonas
orcid: 0000-0001-5001-4804
- first_name: Klaus
full_name: Unsicker, Klaus
last_name: Unsicker
citation:
ama: 'Jonas PM, Unsicker K. Molekulare und zelluläre Grundlagen des Nervensystems.
In: Schmidt R, ed. Lehrbuch Vorklinik. Vol B. Deutscher Ärzte Verlag; 2003:3-26.'
apa: Jonas, P. M., & Unsicker, K. (2003). Molekulare und zelluläre Grundlagen
des Nervensystems. In R. Schmidt (Ed.), Lehrbuch Vorklinik (Vol. B, pp.
3–26). Deutscher Ärzte Verlag.
chicago: Jonas, Peter M, and Klaus Unsicker. “Molekulare Und Zelluläre Grundlagen
Des Nervensystems.” In Lehrbuch Vorklinik, edited by R. Schmidt, B:3–26.
Deutscher Ärzte Verlag, 2003.
ieee: P. M. Jonas and K. Unsicker, “Molekulare und zelluläre Grundlagen des Nervensystems.,”
in Lehrbuch Vorklinik, vol. B, R. Schmidt, Ed. Deutscher Ärzte Verlag,
2003, pp. 3–26.
ista: 'Jonas PM, Unsicker K. 2003.Molekulare und zelluläre Grundlagen des Nervensystems.
In: Lehrbuch Vorklinik. vol. B, 3–26.'
mla: Jonas, Peter M., and Klaus Unsicker. “Molekulare Und Zelluläre Grundlagen Des
Nervensystems.” Lehrbuch Vorklinik, edited by R. Schmidt, vol. B, Deutscher
Ärzte Verlag, 2003, pp. 3–26.
short: P.M. Jonas, K. Unsicker, in:, R. Schmidt (Ed.), Lehrbuch Vorklinik, Deutscher
Ärzte Verlag, 2003, pp. 3–26.
date_created: 2018-12-11T12:03:26Z
date_published: 2003-01-01T00:00:00Z
date_updated: 2021-01-12T07:43:35Z
day: '01'
editor:
- first_name: R.
full_name: Schmidt, R. F.
last_name: Schmidt
extern: 1
month: '01'
page: 3 - 26
publication: Lehrbuch Vorklinik
publication_status: published
publisher: Deutscher Ärzte Verlag
publist_id: '2929'
quality_controlled: 0
status: public
title: Molekulare und zelluläre Grundlagen des Nervensystems.
type: book_chapter
volume: B
year: '2003'
...
---
_id: '3536'
abstract:
- lang: eng
text: 'Genetic engineering of the mouse brain allows investigators to address novel
hypotheses in vivo. Because of the paucity of information on the network patterns
of the mouse hippocampus, we investigated the electrical patterns in the behaving
animal using multisite silicon probes and wire tetrodes. Theta (6-9 Hz) and gamma
(40-100 Hz) oscillations were present during exploration and rapid eye movement
sleep. Gamma power and theta power were comodulated and gamma power varied as
a function of the theta cycle. Pyramidal cells and putative interneurons were
phase-locked to theta oscillations. During immobility, consummatory behaviors
and slow-wave sleep, sharp waves were present in cornu ammonis region CA1 of the
hippocampus stratum radiatum associated with 140-200-Hz “ripples” in the pyramidal
cell layer and population burst of CA1 neurons. In the hilus, large-amplitude
“dentate spikes” occurred in association with increased discharge of hilar neurons.
The amplitude of field patterns was larger in the mouse than in the rat, likely
reflecting the higher neuron density in a smaller brain. We suggest that the main
hippocampal network patterns are mediated by similar pathways and mechanisms in
mouse and rat. '
author:
- first_name: György
full_name: Buzsáki, György
last_name: Buzsáki
- first_name: Derek
full_name: Buhl, Derek L
last_name: Buhl
- first_name: Kenneth
full_name: Harris, Kenneth D
last_name: Harris
- first_name: Jozsef L
full_name: Jozsef Csicsvari
id: 3FA14672-F248-11E8-B48F-1D18A9856A87
last_name: Csicsvari
orcid: 0000-0002-5193-4036
- first_name: Boldizsár
full_name: Czéh, Boldizsár
last_name: Czéh
- first_name: Alexei
full_name: Morozov, Alexei
last_name: Morozov
citation:
ama: Buzsáki G, Buhl D, Harris K, Csicsvari JL, Czéh B, Morozov A. Hippocampal network
patterns of activity in the mouse. Neuroscience. 2003;116(1):201-211. doi:10.1016/S0306-4522(02)00669-3
apa: Buzsáki, G., Buhl, D., Harris, K., Csicsvari, J. L., Czéh, B., & Morozov,
A. (2003). Hippocampal network patterns of activity in the mouse. Neuroscience.
Elsevier. https://doi.org/10.1016/S0306-4522(02)00669-3
chicago: Buzsáki, György, Derek Buhl, Kenneth Harris, Jozsef L Csicsvari, Boldizsár
Czéh, and Alexei Morozov. “Hippocampal Network Patterns of Activity in the Mouse.”
Neuroscience. Elsevier, 2003. https://doi.org/10.1016/S0306-4522(02)00669-3.
ieee: G. Buzsáki, D. Buhl, K. Harris, J. L. Csicsvari, B. Czéh, and A. Morozov,
“Hippocampal network patterns of activity in the mouse,” Neuroscience,
vol. 116, no. 1. Elsevier, pp. 201–211, 2003.
ista: Buzsáki G, Buhl D, Harris K, Csicsvari JL, Czéh B, Morozov A. 2003. Hippocampal
network patterns of activity in the mouse. Neuroscience. 116(1), 201–211.
mla: Buzsáki, György, et al. “Hippocampal Network Patterns of Activity in the Mouse.”
Neuroscience, vol. 116, no. 1, Elsevier, 2003, pp. 201–11, doi:10.1016/S0306-4522(02)00669-3.
short: G. Buzsáki, D. Buhl, K. Harris, J.L. Csicsvari, B. Czéh, A. Morozov, Neuroscience
116 (2003) 201–211.
date_created: 2018-12-11T12:03:50Z
date_published: 2003-01-15T00:00:00Z
date_updated: 2021-01-12T07:44:09Z
day: '15'
doi: 10.1016/S0306-4522(02)00669-3
extern: 1
intvolume: ' 116'
issue: '1'
month: '01'
page: 201 - 211
publication: Neuroscience
publication_status: published
publisher: Elsevier
publist_id: '2849'
quality_controlled: 0
status: public
title: Hippocampal network patterns of activity in the mouse
type: journal_article
volume: 116
year: '2003'
...
---
_id: '3556'
abstract:
- lang: eng
text: We define the Morse-Smale complex of a Morse function over a 3-manifold as
the overlay of the descending and as- cending manifolds of all critical points.
In the generic case, its 3-dimensional cells are shaped like crystals and are
sepa- rated by quadrangular faces. In this paper, we give a combi- natorial algorithm
for constructing such complexes for piece- wise linear data.
author:
- first_name: Herbert
full_name: Herbert Edelsbrunner
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: John
full_name: Harer, John
last_name: Harer
- first_name: Vijay
full_name: Natarajan, Vijay
last_name: Natarajan
- first_name: Valerio
full_name: Pascucci, Valerio
last_name: Pascucci
citation:
ama: 'Edelsbrunner H, Harer J, Natarajan V, Pascucci V. Morse-Smale complexes for
piecewise linear 3-manifolds. In: ACM; 2003:361-370. doi:10.1145/777792.777846'
apa: 'Edelsbrunner, H., Harer, J., Natarajan, V., & Pascucci, V. (2003). Morse-Smale
complexes for piecewise linear 3-manifolds (pp. 361–370). Presented at the SCG:
Symposium on Computational Geometry, ACM. https://doi.org/10.1145/777792.777846'
chicago: Edelsbrunner, Herbert, John Harer, Vijay Natarajan, and Valerio Pascucci.
“Morse-Smale Complexes for Piecewise Linear 3-Manifolds,” 361–70. ACM, 2003. https://doi.org/10.1145/777792.777846.
ieee: 'H. Edelsbrunner, J. Harer, V. Natarajan, and V. Pascucci, “Morse-Smale complexes
for piecewise linear 3-manifolds,” presented at the SCG: Symposium on Computational
Geometry, 2003, pp. 361–370.'
ista: 'Edelsbrunner H, Harer J, Natarajan V, Pascucci V. 2003. Morse-Smale complexes
for piecewise linear 3-manifolds. SCG: Symposium on Computational Geometry, 361–370.'
mla: Edelsbrunner, Herbert, et al. Morse-Smale Complexes for Piecewise Linear
3-Manifolds. ACM, 2003, pp. 361–70, doi:10.1145/777792.777846.
short: H. Edelsbrunner, J. Harer, V. Natarajan, V. Pascucci, in:, ACM, 2003, pp.
361–370.
conference:
name: 'SCG: Symposium on Computational Geometry'
date_created: 2018-12-11T12:03:57Z
date_published: 2003-06-01T00:00:00Z
date_updated: 2021-01-12T07:44:17Z
day: '01'
doi: 10.1145/777792.777846
extern: 1
main_file_link:
- open_access: '0'
url: http://citeseerx.ist.psu.edu/viewdoc/summary?doi=10.1.1.14.9592
month: '06'
page: 361 - 370
publication_status: published
publisher: ACM
publist_id: '2829'
quality_controlled: 0
status: public
title: Morse-Smale complexes for piecewise linear 3-manifolds
type: conference
year: '2003'
...
---
_id: '3573'
abstract:
- lang: eng
text: Given a finite point set in R, the surface reconstruction problem asks for
a surface that passes through many but not necessarily all points. We describe
an unambigu- ous definition of such a surface in geometric and topological terms,
and sketch a fast algorithm for constructing it. Our solution overcomes past limitations
to special point distributions and heuristic design decisions.
author:
- first_name: Herbert
full_name: Herbert Edelsbrunner
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
citation:
ama: 'Edelsbrunner H. Surface reconstruction by wrapping finite sets in space. In:
Discrete & Computational Geometry. Springer; 2003:379-404. doi:10.1007/978-3-642-55566-4_17'
apa: Edelsbrunner, H. (2003). Surface reconstruction by wrapping finite sets in
space. In Discrete & Computational Geometry (pp. 379–404). Springer.
https://doi.org/10.1007/978-3-642-55566-4_17
chicago: Edelsbrunner, Herbert. “Surface Reconstruction by Wrapping Finite Sets
in Space.” In Discrete & Computational Geometry, 379–404. Springer,
2003. https://doi.org/10.1007/978-3-642-55566-4_17.
ieee: H. Edelsbrunner, “Surface reconstruction by wrapping finite sets in space,”
in Discrete & Computational Geometry, Springer, 2003, pp. 379–404.
ista: 'Edelsbrunner H. 2003.Surface reconstruction by wrapping finite sets in space.
In: Discrete & Computational Geometry. , 379–404.'
mla: Edelsbrunner, Herbert. “Surface Reconstruction by Wrapping Finite Sets in Space.”
Discrete & Computational Geometry, Springer, 2003, pp. 379–404, doi:10.1007/978-3-642-55566-4_17.
short: H. Edelsbrunner, in:, Discrete & Computational Geometry, Springer, 2003,
pp. 379–404.
date_created: 2018-12-11T12:04:02Z
date_published: 2003-06-23T00:00:00Z
date_updated: 2021-01-12T07:44:24Z
day: '23'
doi: 10.1007/978-3-642-55566-4_17
extern: 1
main_file_link:
- open_access: '0'
url: http://citeseerx.ist.psu.edu/viewdoc/summary?doi=10.1.1.129.3633
month: '06'
page: 379 - 404
publication: Discrete & Computational Geometry
publication_status: published
publisher: Springer
publist_id: '2812'
quality_controlled: 0
status: public
title: Surface reconstruction by wrapping finite sets in space
type: book_chapter
year: '2003'
...
---
_id: '3584'
abstract:
- lang: eng
text: We develop fast algorithms for computing the linking number of a simplicial
complex within a filtration.We give experimental results in applying our work
toward the detection of non-trivial tangling in biomolecules, modeled as alpha
complexes.
author:
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: Afra
full_name: Zomorodian, Afra
last_name: Zomorodian
citation:
ama: Edelsbrunner H, Zomorodian A. Computing linking numbers of a filtration. Homology,
Homotopy and Applications. 2003;5(2):19-37.
apa: Edelsbrunner, H., & Zomorodian, A. (2003). Computing linking numbers of
a filtration. Homology, Homotopy and Applications. International Press.
chicago: Edelsbrunner, Herbert, and Afra Zomorodian. “Computing Linking Numbers
of a Filtration.” Homology, Homotopy and Applications. International Press,
2003.
ieee: H. Edelsbrunner and A. Zomorodian, “Computing linking numbers of a filtration,”
Homology, Homotopy and Applications, vol. 5, no. 2. International Press,
pp. 19–37, 2003.
ista: Edelsbrunner H, Zomorodian A. 2003. Computing linking numbers of a filtration.
Homology, Homotopy and Applications. 5(2), 19–37.
mla: Edelsbrunner, Herbert, and Afra Zomorodian. “Computing Linking Numbers of a
Filtration.” Homology, Homotopy and Applications, vol. 5, no. 2, International
Press, 2003, pp. 19–37.
short: H. Edelsbrunner, A. Zomorodian, Homology, Homotopy and Applications 5 (2003)
19–37.
date_created: 2018-12-11T12:04:05Z
date_published: 2003-04-22T00:00:00Z
date_updated: 2021-01-12T07:44:28Z
day: '22'
extern: '1'
intvolume: ' 5'
issue: '2'
language:
- iso: eng
main_file_link:
- url: http://projecteuclid.org/euclid.hha/1088453320
month: '04'
oa_version: None
page: 19 - 37
publication: Homology, Homotopy and Applications
publication_status: published
publisher: International Press
publist_id: '2801'
quality_controlled: '1'
status: public
title: Computing linking numbers of a filtration
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 5
year: '2003'
...
---
_id: '3620'
abstract:
- lang: eng
text: 'Stable hybrid zones in which ecologically divergent taxa give rise to a range
of recombinants are natural laboratories in which the genetic basis of adaptation
and reproductive isolation can be unraveled. One such hybrid zone is formed by
the fire-bellied toads Bombina bombina and B. variegata (Anura: Discoglossidae).
Adaptations to permanent and ephemeral breeding habitats, respectively, have shaped
numerous phenotypic differences between the taxa. All of these are, in principle,
candidates for a genetic dissection via QTL mapping. We present here a linkage
map of 28 codominant and 10 dominant markers in the Bombina genome. In an F2 cross,
markers that were mainly microsatellites, SSCPs or allozymes were mapped to 20
linkage groups. Among the 40 isolated CA microsatellites, we noted a preponderance
of compound and frequently interleaved CA-TA repeats as well as a striking polarity
at the 5′ end of the repeats.'
author:
- first_name: Beate
full_name: Nürnberger, Beate
last_name: Nürnberger
- first_name: Sebastian
full_name: Hofman, Sebastian
last_name: Hofman
- first_name: Bqruni
full_name: Förg-Brey, Bqruni
last_name: Förg Brey
- first_name: Gabriele
full_name: Praetzel, Gabriele
last_name: Praetzel
- first_name: Alan
full_name: Maclean, Alan W
last_name: Maclean
- first_name: Jacek
full_name: Szymura, Jacek M
last_name: Szymura
- first_name: Catherine
full_name: Abbott, Catherine M
last_name: Abbott
- first_name: Nicholas H
full_name: Nicholas Barton
id: 4880FE40-F248-11E8-B48F-1D18A9856A87
last_name: Barton
orcid: 0000-0002-8548-5240
citation:
ama: 'Nürnberger B, Hofman S, Förg Brey B, et al. A linkage map for the hybridising
toads Bombina bombina and B. variegata (Anura: Discoglossidae). Heredity.
2003;91(2):136-142. doi:10.1038/sj.hdy.6800291'
apa: 'Nürnberger, B., Hofman, S., Förg Brey, B., Praetzel, G., Maclean, A., Szymura,
J., … Barton, N. H. (2003). A linkage map for the hybridising toads Bombina bombina
and B. variegata (Anura: Discoglossidae). Heredity. Nature Publishing Group.
https://doi.org/10.1038/sj.hdy.6800291'
chicago: 'Nürnberger, Beate, Sebastian Hofman, Bqruni Förg Brey, Gabriele Praetzel,
Alan Maclean, Jacek Szymura, Catherine Abbott, and Nicholas H Barton. “A Linkage
Map for the Hybridising Toads Bombina Bombina and B. Variegata (Anura: Discoglossidae).”
Heredity. Nature Publishing Group, 2003. https://doi.org/10.1038/sj.hdy.6800291.'
ieee: 'B. Nürnberger et al., “A linkage map for the hybridising toads Bombina
bombina and B. variegata (Anura: Discoglossidae),” Heredity, vol. 91, no.
2. Nature Publishing Group, pp. 136–142, 2003.'
ista: 'Nürnberger B, Hofman S, Förg Brey B, Praetzel G, Maclean A, Szymura J, Abbott
C, Barton NH. 2003. A linkage map for the hybridising toads Bombina bombina and
B. variegata (Anura: Discoglossidae). Heredity. 91(2), 136–142.'
mla: 'Nürnberger, Beate, et al. “A Linkage Map for the Hybridising Toads Bombina
Bombina and B. Variegata (Anura: Discoglossidae).” Heredity, vol. 91, no.
2, Nature Publishing Group, 2003, pp. 136–42, doi:10.1038/sj.hdy.6800291.'
short: B. Nürnberger, S. Hofman, B. Förg Brey, G. Praetzel, A. Maclean, J. Szymura,
C. Abbott, N.H. Barton, Heredity 91 (2003) 136–142.
date_created: 2018-12-11T12:04:17Z
date_published: 2003-08-01T00:00:00Z
date_updated: 2021-01-12T07:44:43Z
day: '01'
doi: 10.1038/sj.hdy.6800291
extern: 1
intvolume: ' 91'
issue: '2'
month: '08'
page: 136 - 142
publication: Heredity
publication_status: published
publisher: Nature Publishing Group
publist_id: '2763'
quality_controlled: 0
status: public
title: 'A linkage map for the hybridising toads Bombina bombina and B. variegata (Anura:
Discoglossidae)'
type: journal_article
volume: 91
year: '2003'
...
---
_id: '3619'
abstract:
- lang: eng
text: What is the chance that some part of a stretch of genome will survive? In
a population of constant size, and with no selection, the probability of survival
of some part of a stretch of map length y<1 approaches View the MathML source
for View the MathML source. Thus, the whole genome is certain to be lost, but
the rate of loss is extremely slow. This solution extends to give the whole distribution
of surviving block sizes as a function of time. We show that the expected number
of blocks at time t is 1+yt and give expressions for the moments of the number
of blocks and the total amount of genome that survives for a given time. The solution
is based on a branching process and assumes complete interference between crossovers,
so that each descendant carries only a single block of ancestral material. We
consider cases where most individuals carry multiple blocks, either because there
are multiple crossovers in a long genetic map, or because enough time has passed
that most individuals in the population are related to each other. For species
such as ours, which have a long genetic map, the genome of any individual which
leaves descendants (∼80% of the population for a Poisson offspring number with
mean two) is likely to persist for an extremely long time, in the form of a few
short blocks of genome.
author:
- first_name: Stuart
full_name: Baird, Stuart J
last_name: Baird
- first_name: Nicholas H
full_name: Nicholas Barton
id: 4880FE40-F248-11E8-B48F-1D18A9856A87
last_name: Barton
orcid: 0000-0002-8548-5240
- first_name: Alison
full_name: Etheridge, Alison M
last_name: Etheridge
citation:
ama: Baird S, Barton NH, Etheridge A. The distribution of surviving blocks of an
ancestral genome. Theoretical Population Biology. 2003;64(4):451-471. doi:10.1016/S0040-5809(03)00098-4
apa: Baird, S., Barton, N. H., & Etheridge, A. (2003). The distribution of surviving
blocks of an ancestral genome. Theoretical Population Biology. Academic
Press. https://doi.org/10.1016/S0040-5809(03)00098-4
chicago: Baird, Stuart, Nicholas H Barton, and Alison Etheridge. “The Distribution
of Surviving Blocks of an Ancestral Genome.” Theoretical Population Biology.
Academic Press, 2003. https://doi.org/10.1016/S0040-5809(03)00098-4.
ieee: S. Baird, N. H. Barton, and A. Etheridge, “The distribution of surviving blocks
of an ancestral genome,” Theoretical Population Biology, vol. 64, no. 4.
Academic Press, pp. 451–471, 2003.
ista: Baird S, Barton NH, Etheridge A. 2003. The distribution of surviving blocks
of an ancestral genome. Theoretical Population Biology. 64(4), 451–471.
mla: Baird, Stuart, et al. “The Distribution of Surviving Blocks of an Ancestral
Genome.” Theoretical Population Biology, vol. 64, no. 4, Academic Press,
2003, pp. 451–71, doi:10.1016/S0040-5809(03)00098-4.
short: S. Baird, N.H. Barton, A. Etheridge, Theoretical Population Biology 64 (2003)
451–471.
date_created: 2018-12-11T12:04:17Z
date_published: 2003-12-01T00:00:00Z
date_updated: 2021-01-12T07:44:42Z
day: '01'
doi: 10.1016/S0040-5809(03)00098-4
extern: 1
intvolume: ' 64'
issue: '4'
month: '12'
page: 451 - 471
publication: Theoretical Population Biology
publication_status: published
publisher: Academic Press
publist_id: '2764'
quality_controlled: 0
status: public
title: The distribution of surviving blocks of an ancestral genome
type: journal_article
volume: 64
year: '2003'
...
---
_id: '3618'
abstract:
- lang: eng
text: There are several analyses in evolutionary ecology which assume that a family
of offspring has come from only two parents. Here, we present a simple test for
detecting when a batch involves two or more subfamilies. It is based on the fact
that the mixing of families generates associations amongst unlinked marker loci.
We also present simulations illustrating the power of our method for varying numbers
of loci, alleles per locus and genotyped individuals.
author:
- first_name: Timothy
full_name: Vines, Timothy H
last_name: Vines
- first_name: Nicholas H
full_name: Nicholas Barton
id: 4880FE40-F248-11E8-B48F-1D18A9856A87
last_name: Barton
orcid: 0000-0002-8548-5240
citation:
ama: Vines T, Barton NH. A new approach to detecting mixed families. Molecular
Ecology. 2003;12(7):1999-2002. doi:10.1046/j.1365-294X.2003.01867.x
apa: Vines, T., & Barton, N. H. (2003). A new approach to detecting mixed families.
Molecular Ecology. Wiley-Blackwell. https://doi.org/10.1046/j.1365-294X.2003.01867.x
chicago: Vines, Timothy, and Nicholas H Barton. “A New Approach to Detecting Mixed
Families.” Molecular Ecology. Wiley-Blackwell, 2003. https://doi.org/10.1046/j.1365-294X.2003.01867.x.
ieee: T. Vines and N. H. Barton, “A new approach to detecting mixed families,” Molecular
Ecology, vol. 12, no. 7. Wiley-Blackwell, pp. 1999–2002, 2003.
ista: Vines T, Barton NH. 2003. A new approach to detecting mixed families. Molecular
Ecology. 12(7), 1999–2002.
mla: Vines, Timothy, and Nicholas H. Barton. “A New Approach to Detecting Mixed
Families.” Molecular Ecology, vol. 12, no. 7, Wiley-Blackwell, 2003, pp.
1999–2002, doi:10.1046/j.1365-294X.2003.01867.x.
short: T. Vines, N.H. Barton, Molecular Ecology 12 (2003) 1999–2002.
date_created: 2018-12-11T12:04:16Z
date_published: 2003-07-01T00:00:00Z
date_updated: 2021-01-12T07:44:42Z
day: '01'
doi: 10.1046/j.1365-294X.2003.01867.x
extern: 1
intvolume: ' 12'
issue: '7'
month: '07'
page: 1999 - 2002
publication: Molecular Ecology
publication_status: published
publisher: Wiley-Blackwell
publist_id: '2765'
quality_controlled: 0
status: public
title: A new approach to detecting mixed families
type: journal_article
volume: 12
year: '2003'
...
---
_id: '3752'
abstract:
- lang: eng
text: We use the lac operon in Escherichia coli as a prototype system to illustrate
the current state, applicability, and limitations of modeling the dynamics of
cellular networks. We integrate three different levels of description (molecular,
cellular, and that of cell population) into a single model, which seems to capture
many experimental aspects of the system.
author:
- first_name: Jose
full_name: Vilar,Jose M
last_name: Vilar
- first_name: Calin C
full_name: Calin Guet
id: 47F8433E-F248-11E8-B48F-1D18A9856A87
last_name: Guet
orcid: 0000-0001-6220-2052
- first_name: Stanislas
full_name: Leibler, Stanislas
last_name: Leibler
citation:
ama: 'Vilar J, Guet CC, Leibler S. Modeling network dynamics: the lac operon, a
case study. Journal of Cell Biology. 2003;161(3):471-476. doi:10.1083/jcb.200301125'
apa: 'Vilar, J., Guet, C. C., & Leibler, S. (2003). Modeling network dynamics:
the lac operon, a case study. Journal of Cell Biology. Rockefeller University
Press. https://doi.org/10.1083/jcb.200301125'
chicago: 'Vilar, Jose, Calin C Guet, and Stanislas Leibler. “Modeling Network Dynamics:
The Lac Operon, a Case Study.” Journal of Cell Biology. Rockefeller University
Press, 2003. https://doi.org/10.1083/jcb.200301125.'
ieee: 'J. Vilar, C. C. Guet, and S. Leibler, “Modeling network dynamics: the lac
operon, a case study,” Journal of Cell Biology, vol. 161, no. 3. Rockefeller
University Press, pp. 471–476, 2003.'
ista: 'Vilar J, Guet CC, Leibler S. 2003. Modeling network dynamics: the lac operon,
a case study. Journal of Cell Biology. 161(3), 471–476.'
mla: 'Vilar, Jose, et al. “Modeling Network Dynamics: The Lac Operon, a Case Study.”
Journal of Cell Biology, vol. 161, no. 3, Rockefeller University Press,
2003, pp. 471–76, doi:10.1083/jcb.200301125.'
short: J. Vilar, C.C. Guet, S. Leibler, Journal of Cell Biology 161 (2003) 471–476.
date_created: 2018-12-11T12:04:58Z
date_published: 2003-01-12T00:00:00Z
date_updated: 2021-01-12T07:51:57Z
day: '12'
doi: 10.1083/jcb.200301125
extern: 1
intvolume: ' 161'
issue: '3'
main_file_link:
- open_access: '1'
url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2172934/?tool=pubmed
month: '01'
oa: 1
page: 471 - 476
publication: Journal of Cell Biology
publication_status: published
publisher: Rockefeller University Press
publist_id: '2475'
quality_controlled: 0
status: public
title: 'Modeling network dynamics: the lac operon, a case study'
type: journal_article
volume: 161
year: '2003'
...
---
_id: '3797'
article_processing_charge: No
author:
- first_name: Wolfgang
full_name: Bauer, Wolfgang
last_name: Bauer
- first_name: Marco
full_name: Kleine Berkenbusch, Marco
last_name: Kleine Berkenbusch
- first_name: Mark Tobias
full_name: Bollenbach, Mark Tobias
id: 3E6DB97A-F248-11E8-B48F-1D18A9856A87
last_name: Bollenbach
orcid: 0000-0003-4398-476X
citation:
ama: 'Bauer W, Kleine Berkenbusch M, Bollenbach MT. Breaking atomic nuclei into
little pieces: evidence for a phase transition. Revista Mexicana De Fisica.
2003;49(4):1-6.'
apa: 'Bauer, W., Kleine Berkenbusch, M., & Bollenbach, M. T. (2003). Breaking
atomic nuclei into little pieces: evidence for a phase transition. Revista
Mexicana De Fisica. Sociedad Mexicana de Física.'
chicago: 'Bauer, Wolfgang, Marco Kleine Berkenbusch, and Mark Tobias Bollenbach.
“Breaking Atomic Nuclei into Little Pieces: Evidence for a Phase Transition.”
Revista Mexicana De Fisica. Sociedad Mexicana de Física, 2003.'
ieee: 'W. Bauer, M. Kleine Berkenbusch, and M. T. Bollenbach, “Breaking atomic nuclei
into little pieces: evidence for a phase transition,” Revista Mexicana De Fisica,
vol. 49, no. 4. Sociedad Mexicana de Física, pp. 1–6, 2003.'
ista: 'Bauer W, Kleine Berkenbusch M, Bollenbach MT. 2003. Breaking atomic nuclei
into little pieces: evidence for a phase transition. Revista Mexicana De Fisica.
49(4), 1–6.'
mla: 'Bauer, Wolfgang, et al. “Breaking Atomic Nuclei into Little Pieces: Evidence
for a Phase Transition.” Revista Mexicana De Fisica, vol. 49, no. 4, Sociedad
Mexicana de Física, 2003, pp. 1–6.'
short: W. Bauer, M. Kleine Berkenbusch, M.T. Bollenbach, Revista Mexicana De Fisica
49 (2003) 1–6.
date_created: 2018-12-11T12:05:13Z
date_published: 2003-01-01T00:00:00Z
date_updated: 2021-01-12T07:52:16Z
day: '01'
extern: '1'
intvolume: ' 49'
issue: '4'
language:
- iso: eng
month: '01'
oa_version: None
page: 1 - 6
publication: Revista Mexicana De Fisica
publication_status: published
publisher: Sociedad Mexicana de Física
publist_id: '2413'
status: public
title: 'Breaking atomic nuclei into little pieces: evidence for a phase transition'
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 49
year: '2003'
...
---
_id: '3897'
abstract:
- lang: eng
text: Many verification, planning, and control problems can be modeled as games
played on state-transition graphs by one or two players whose conflicting goals
are to form a path in the graph. The focus here is on simple stochastic parity
games, that is, two-player games with turn-based probabilistic transitions and
omega-regular objectives formalized as parity (Rabin chain) winning conditions.
An efficient translation from simple stochastic parity games to nonstochastic
parity games is given. As many algorithms are known for solving the latter, the
translation yields efficient algorithms for computing the states of a simple stochastic
parity game from which a player can win with probability 1. An important special
case of simple stochastic parity games are the Markov decision processes with
Buchi objectives. For this special case a first provably subquadratic algorithm
is given for computing the states from which the single player has a strategy
to achieve a Buchi objective with probability 1. For game graphs with m edges
the algorithm works in time O(mrootm). Interestingly, a similar technique sheds
light on the question of the computational complexity of solving simple Buchi
games and yields the first provably subquadratic algorithm, with a running time
of O(n(2)/log n) for game graphs with n vertices and O(n) edges.
acknowledgement: This research was supported in part by the DARPA grant F33615-C-98-3614,
the ONR grant N00014-02-1-0671, the NSF grants CCR-9988172 and CCR-0225610, and
the Polish KBN grant 7-T11C-027-20.
alternative_title:
- LNCS
author:
- first_name: Krishnendu
full_name: Krishnendu Chatterjee
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
- first_name: Marcin
full_name: Jurdziński, Marcin
last_name: Jurdziński
- first_name: Thomas A
full_name: Thomas Henzinger
id: 40876CD8-F248-11E8-B48F-1D18A9856A87
last_name: Henzinger
orcid: 0000−0002−2985−7724
citation:
ama: 'Chatterjee K, Jurdziński M, Henzinger TA. Simple stochastic parity games.
In: Vol 2803. Springer; 2003:100-113. doi:10.1007/978-3-540-45220-1_11'
apa: 'Chatterjee, K., Jurdziński, M., & Henzinger, T. A. (2003). Simple stochastic
parity games (Vol. 2803, pp. 100–113). Presented at the CSL: Computer Science
Logic, Springer. https://doi.org/10.1007/978-3-540-45220-1_11'
chicago: Chatterjee, Krishnendu, Marcin Jurdziński, and Thomas A Henzinger. “Simple
Stochastic Parity Games,” 2803:100–113. Springer, 2003. https://doi.org/10.1007/978-3-540-45220-1_11.
ieee: 'K. Chatterjee, M. Jurdziński, and T. A. Henzinger, “Simple stochastic parity
games,” presented at the CSL: Computer Science Logic, 2003, vol. 2803, pp. 100–113.'
ista: 'Chatterjee K, Jurdziński M, Henzinger TA. 2003. Simple stochastic parity
games. CSL: Computer Science Logic, LNCS, vol. 2803, 100–113.'
mla: Chatterjee, Krishnendu, et al. Simple Stochastic Parity Games. Vol.
2803, Springer, 2003, pp. 100–13, doi:10.1007/978-3-540-45220-1_11.
short: K. Chatterjee, M. Jurdziński, T.A. Henzinger, in:, Springer, 2003, pp. 100–113.
conference:
name: 'CSL: Computer Science Logic'
date_created: 2018-12-11T12:05:46Z
date_published: 2003-08-18T00:00:00Z
date_updated: 2021-01-12T07:53:02Z
day: '18'
doi: 10.1007/978-3-540-45220-1_11
extern: 1
intvolume: ' 2803'
month: '08'
page: 100 - 113
publication_status: published
publisher: Springer
publist_id: '2259'
quality_controlled: 0
status: public
title: Simple stochastic parity games
type: conference
volume: 2803
year: '2003'
...
---
_id: '3898'
abstract:
- lang: eng
text: We study the problem of determining stack boundedness and the exact maximum
stack size for three classes of interrupt-driven programs. Interrupt-driven programs
axe used in many real-time applications that require responsive interrupt handling.
In order to ensure responsiveness, programmers often enable interrupt processing
in the body of lower-priority interrupt handlers. In such programs a programming
error can allow interrupt handlers to be interrupted in cyclic fashion to lead
to an unbounded stack, causing the system to crash. For a restricted class of
interrupt-driven programs, we show that there is a polynomial-time procedure to
check stack boundedness, while determining the exact maximum stack size is PSPACE-complete.
For a larger class of programs, the two problems are both PSPACE-complete, and
for the largest class of programs we consider, the two problems are PSPACE-hard
and can be solved in exponential time.
acknowledgement: Jens Palsberg, Di Ma, and Tian Zhao were supported by the NSF ITR
award 0112628. Thomas A. Henzinger, Krishnendu Chatterjee, and Rupak Majumdar were
supported by the AFOSR grant F49620-00-1-0327, the DARPA grants F33615-C-98-3614
and F33615-00-C-1693, the MARCO grant 98-DT-660, and the NSF grants CCR-0208875
and CCR-0085949.
alternative_title:
- LNCS
author:
- first_name: Krishnendu
full_name: Krishnendu Chatterjee
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
- first_name: Di
full_name: Ma, Di
last_name: Ma
- first_name: Ritankar
full_name: Majumdar, Ritankar S
last_name: Majumdar
- first_name: Tian
full_name: Zhao, Tian
last_name: Zhao
- first_name: Thomas A
full_name: Thomas Henzinger
id: 40876CD8-F248-11E8-B48F-1D18A9856A87
last_name: Henzinger
orcid: 0000−0002−2985−7724
- first_name: Jens
full_name: Palsberg, Jens
last_name: Palsberg
citation:
ama: 'Chatterjee K, Ma D, Majumdar R, Zhao T, Henzinger TA, Palsberg J. Stack size
analysis for interrupt-driven programs. In: Vol 2694. Springer; 2003:109-126.
doi:10.1007/3-540-44898-5_7'
apa: 'Chatterjee, K., Ma, D., Majumdar, R., Zhao, T., Henzinger, T. A., & Palsberg,
J. (2003). Stack size analysis for interrupt-driven programs (Vol. 2694, pp. 109–126).
Presented at the SAS: Static Analysis Symposium, Springer. https://doi.org/10.1007/3-540-44898-5_7'
chicago: Chatterjee, Krishnendu, Di Ma, Ritankar Majumdar, Tian Zhao, Thomas A Henzinger,
and Jens Palsberg. “Stack Size Analysis for Interrupt-Driven Programs,” 2694:109–26.
Springer, 2003. https://doi.org/10.1007/3-540-44898-5_7.
ieee: 'K. Chatterjee, D. Ma, R. Majumdar, T. Zhao, T. A. Henzinger, and J. Palsberg,
“Stack size analysis for interrupt-driven programs,” presented at the SAS: Static
Analysis Symposium, 2003, vol. 2694, pp. 109–126.'
ista: 'Chatterjee K, Ma D, Majumdar R, Zhao T, Henzinger TA, Palsberg J. 2003. Stack
size analysis for interrupt-driven programs. SAS: Static Analysis Symposium, LNCS,
vol. 2694, 109–126.'
mla: Chatterjee, Krishnendu, et al. Stack Size Analysis for Interrupt-Driven
Programs. Vol. 2694, Springer, 2003, pp. 109–26, doi:10.1007/3-540-44898-5_7.
short: K. Chatterjee, D. Ma, R. Majumdar, T. Zhao, T.A. Henzinger, J. Palsberg,
in:, Springer, 2003, pp. 109–126.
conference:
name: 'SAS: Static Analysis Symposium'
date_created: 2018-12-11T12:05:46Z
date_published: 2003-05-28T00:00:00Z
date_updated: 2021-01-12T07:53:02Z
day: '28'
doi: 10.1007/3-540-44898-5_7
extern: 1
intvolume: ' 2694'
month: '05'
page: 109 - 126
publication_status: published
publisher: Springer
publist_id: '2260'
quality_controlled: 0
status: public
title: Stack size analysis for interrupt-driven programs
type: conference
volume: 2694
year: '2003'
...
---
_id: '3993'
abstract:
- lang: eng
text: We present algorithms for constructing a hierarchy of increasingly coarse
Morse-Smale complexes that decompose a piecewise linear 2-manifold. While these
complexes are defined only in the smooth category, we extend the construction
to the piecewise linearcategory by ensuring structural integrity and simulating
differentiability. We then simplify Morse-Smale complexes by canceling pairs of
critical points in order of increasing persistence.
acknowledgement: Partially supported by ARO under Grant DAAG55-98-1-0177, NSF under
Grants CCR-97-12088, EIA-9972879 and CCR-00-86013.
author:
- first_name: Herbert
full_name: Herbert Edelsbrunner
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: John
full_name: Harer, John
last_name: Harer
- first_name: Afra
full_name: Zomorodian, Afra
last_name: Zomorodian
citation:
ama: Edelsbrunner H, Harer J, Zomorodian A. Hierarchical Morse-Smale complexes for
piecewise linear 2-manifolds. Discrete & Computational Geometry. 2003;30(1):87-107.
doi:10.1007/s00454-003-2926-5
apa: Edelsbrunner, H., Harer, J., & Zomorodian, A. (2003). Hierarchical Morse-Smale
complexes for piecewise linear 2-manifolds. Discrete & Computational Geometry.
Springer. https://doi.org/10.1007/s00454-003-2926-5
chicago: Edelsbrunner, Herbert, John Harer, and Afra Zomorodian. “Hierarchical Morse-Smale
Complexes for Piecewise Linear 2-Manifolds.” Discrete & Computational Geometry.
Springer, 2003. https://doi.org/10.1007/s00454-003-2926-5.
ieee: H. Edelsbrunner, J. Harer, and A. Zomorodian, “Hierarchical Morse-Smale complexes
for piecewise linear 2-manifolds,” Discrete & Computational Geometry,
vol. 30, no. 1. Springer, pp. 87–107, 2003.
ista: Edelsbrunner H, Harer J, Zomorodian A. 2003. Hierarchical Morse-Smale complexes
for piecewise linear 2-manifolds. Discrete & Computational Geometry. 30(1),
87–107.
mla: Edelsbrunner, Herbert, et al. “Hierarchical Morse-Smale Complexes for Piecewise
Linear 2-Manifolds.” Discrete & Computational Geometry, vol. 30, no.
1, Springer, 2003, pp. 87–107, doi:10.1007/s00454-003-2926-5.
short: H. Edelsbrunner, J. Harer, A. Zomorodian, Discrete & Computational Geometry
30 (2003) 87–107.
date_created: 2018-12-11T12:06:19Z
date_published: 2003-07-01T00:00:00Z
date_updated: 2021-01-12T07:53:43Z
day: '01'
doi: 10.1007/s00454-003-2926-5
extern: 1
intvolume: ' 30'
issue: '1'
month: '07'
page: 87 - 107
publication: Discrete & Computational Geometry
publication_status: published
publisher: Springer
publist_id: '2134'
quality_controlled: 0
status: public
title: Hierarchical Morse-Smale complexes for piecewise linear 2-manifolds
type: journal_article
volume: 30
year: '2003'
...
---
_id: '3994'
abstract:
- lang: eng
text: The body defined by a finite collection of disks is a subset of the plane
bounded by a tangent continuous curve, which we call the skin. We give analytic
formulas for the area, the perimeter, the area derivative, and the perimeter derivative
of the body. Given the filtrations of the Delaunay triangulation and the Voronoi
diagram of the disks, all formulas can be evaluated in time proportional to the
number of disks.
acknowledgement: NSF under grant DMS-98-73945, ARO under grant DAAG55-98-1-0177 and
by NSF under grants CCR- 97-12088, EIA-9972879, and CCR-00-86013.
author:
- first_name: Ho
full_name: Cheng, Ho-Lun
last_name: Cheng
- first_name: Herbert
full_name: Herbert Edelsbrunner
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
citation:
ama: 'Cheng H, Edelsbrunner H. Area, perimeter and derivatives of a skin curve.
Computational Geometry: Theory and Applications. 2003;26(2):173-192. doi:10.1016/S0925-7721(02)00124-4'
apa: 'Cheng, H., & Edelsbrunner, H. (2003). Area, perimeter and derivatives
of a skin curve. Computational Geometry: Theory and Applications. Elsevier.
https://doi.org/10.1016/S0925-7721(02)00124-4'
chicago: 'Cheng, Ho, and Herbert Edelsbrunner. “Area, Perimeter and Derivatives
of a Skin Curve.” Computational Geometry: Theory and Applications. Elsevier,
2003. https://doi.org/10.1016/S0925-7721(02)00124-4.'
ieee: 'H. Cheng and H. Edelsbrunner, “Area, perimeter and derivatives of a skin
curve,” Computational Geometry: Theory and Applications, vol. 26, no. 2.
Elsevier, pp. 173–192, 2003.'
ista: 'Cheng H, Edelsbrunner H. 2003. Area, perimeter and derivatives of a skin
curve. Computational Geometry: Theory and Applications. 26(2), 173–192.'
mla: 'Cheng, Ho, and Herbert Edelsbrunner. “Area, Perimeter and Derivatives of a
Skin Curve.” Computational Geometry: Theory and Applications, vol. 26,
no. 2, Elsevier, 2003, pp. 173–92, doi:10.1016/S0925-7721(02)00124-4.'
short: 'H. Cheng, H. Edelsbrunner, Computational Geometry: Theory and Applications
26 (2003) 173–192.'
date_created: 2018-12-11T12:06:20Z
date_published: 2003-10-01T00:00:00Z
date_updated: 2021-01-12T07:53:43Z
day: '01'
doi: 10.1016/S0925-7721(02)00124-4
extern: 1
intvolume: ' 26'
issue: '2'
month: '10'
page: 173 - 192
publication: 'Computational Geometry: Theory and Applications'
publication_status: published
publisher: Elsevier
publist_id: '2135'
quality_controlled: 0
status: public
title: Area, perimeter and derivatives of a skin curve
type: journal_article
volume: 26
year: '2003'
...
---
_id: '3139'
abstract:
- lang: eng
text: Significant advances have been made during the past few years in our understanding
of how the spinal monosynaptic reflex develops. Transcription factors in the Neurogenin,
Runt, ETS, and LIM families control sequential steps of the specification of various
subtypes of dorsal root ganglia sensory neurons. The initiation of muscle spindle
differentiation requires neuregulin 1, derived from Ia afferent sensory neurons,
and signaling through ErbB receptors in intrafusal muscle fibers. Several retrograde
signals from the periphery are important for the establishment of late connectivity
in the reflex circuit. Finally, neurotrophin 3 released from muscle spindles regulates
the strength of sensory-motor connections within the spinal cord postnatally.
author:
- first_name: Hsiao
full_name: Chen, Hsiao Huei
last_name: Chen
- first_name: Simon
full_name: Simon Hippenmeyer
id: 37B36620-F248-11E8-B48F-1D18A9856A87
last_name: Hippenmeyer
orcid: 0000-0003-2279-1061
- first_name: Silvia
full_name: Arber, Silvia
last_name: Arber
- first_name: Eric
full_name: Frank, Eric
last_name: Frank
citation:
ama: Chen H, Hippenmeyer S, Arber S, Frank E. Development of the monosynaptic stretch
reflex circuit. Current Opinion in Neurobiology. 2003;13(1):96-102. doi:10.1016/S0959-4388(03)00006-0
apa: Chen, H., Hippenmeyer, S., Arber, S., & Frank, E. (2003). Development of
the monosynaptic stretch reflex circuit. Current Opinion in Neurobiology.
Elsevier. https://doi.org/10.1016/S0959-4388(03)00006-0
chicago: Chen, Hsiao, Simon Hippenmeyer, Silvia Arber, and Eric Frank. “Development
of the Monosynaptic Stretch Reflex Circuit.” Current Opinion in Neurobiology.
Elsevier, 2003. https://doi.org/10.1016/S0959-4388(03)00006-0.
ieee: H. Chen, S. Hippenmeyer, S. Arber, and E. Frank, “Development of the monosynaptic
stretch reflex circuit,” Current Opinion in Neurobiology, vol. 13, no.
1. Elsevier, pp. 96–102, 2003.
ista: Chen H, Hippenmeyer S, Arber S, Frank E. 2003. Development of the monosynaptic
stretch reflex circuit. Current Opinion in Neurobiology. 13(1), 96–102.
mla: Chen, Hsiao, et al. “Development of the Monosynaptic Stretch Reflex Circuit.”
Current Opinion in Neurobiology, vol. 13, no. 1, Elsevier, 2003, pp. 96–102,
doi:10.1016/S0959-4388(03)00006-0.
short: H. Chen, S. Hippenmeyer, S. Arber, E. Frank, Current Opinion in Neurobiology
13 (2003) 96–102.
date_created: 2018-12-11T12:01:37Z
date_published: 2003-02-01T00:00:00Z
date_updated: 2019-04-26T07:22:24Z
day: '01'
doi: 10.1016/S0959-4388(03)00006-0
extern: 1
intvolume: ' 13'
issue: '1'
month: '02'
page: 96 - 102
publication: Current Opinion in Neurobiology
publication_status: published
publisher: Elsevier
publist_id: '3557'
quality_controlled: 0
status: public
title: Development of the monosynaptic stretch reflex circuit
type: review
volume: 13
year: '2003'
...
---
_id: '3171'
abstract:
- lang: eng
text: 'Reconstructing a 3-D scene from more than one camera is a classical problem
in computer vision. One of the major sources of difficulty is the fact that not
all scene elements are visible from all cameras. In the last few years, two promising
approaches have been developed 11,12 that formulate the scene reconstruction problem
in terms of energy minimization, and minimize the energy using graph cuts. These
energy minimization approaches treat the input images symmetrically, handle visibility
constraints correctly, and allow spatial smoothness to be enforced. However, these
algorithm propose different problem formulations, and handle a limited class of
smoothness terms. One algorithm 11 uses a problem formulation that is restricted
to two-camera stereo, and imposes smoothness between a pair of cameras. The other
algorithm 12 can handle an arbitrary number of cameras, but imposes smoothness
only with respect to a single camera. In this paper we give a more general energy
minimization formulation for the problem, which allows a larger class of spatial
smoothness constraints. We show that our formulation includes both of the previous
approaches as special cases, as well as permitting new energy functions. Experimental
results on real data with ground truth are also included. '
alternative_title:
- LNCS
author:
- first_name: Vladimir
full_name: Vladimir Kolmogorov
id: 3D50B0BA-F248-11E8-B48F-1D18A9856A87
last_name: Kolmogorov
- first_name: Ramin
full_name: Zabih, Ramin
last_name: Zabih
- first_name: Steven
full_name: Gortler, Steven
last_name: Gortler
citation:
ama: 'Kolmogorov V, Zabih R, Gortler S. Generalized multi camera scene reconstruction
using graph cuts. In: Vol 2683. Springer; 2003:501-516. doi:10.1007/978-3-540-45063-4_32'
apa: 'Kolmogorov, V., Zabih, R., & Gortler, S. (2003). Generalized multi camera
scene reconstruction using graph cuts (Vol. 2683, pp. 501–516). Presented at the
EMMCVPR: Energy Minimization Methods in Computer Vision and Pattern Recognition,
Springer. https://doi.org/10.1007/978-3-540-45063-4_32'
chicago: Kolmogorov, Vladimir, Ramin Zabih, and Steven Gortler. “Generalized Multi
Camera Scene Reconstruction Using Graph Cuts,” 2683:501–16. Springer, 2003. https://doi.org/10.1007/978-3-540-45063-4_32.
ieee: 'V. Kolmogorov, R. Zabih, and S. Gortler, “Generalized multi camera scene
reconstruction using graph cuts,” presented at the EMMCVPR: Energy Minimization
Methods in Computer Vision and Pattern Recognition, 2003, vol. 2683, pp. 501–516.'
ista: 'Kolmogorov V, Zabih R, Gortler S. 2003. Generalized multi camera scene reconstruction
using graph cuts. EMMCVPR: Energy Minimization Methods in Computer Vision and
Pattern Recognition, LNCS, vol. 2683, 501–516.'
mla: Kolmogorov, Vladimir, et al. Generalized Multi Camera Scene Reconstruction
Using Graph Cuts. Vol. 2683, Springer, 2003, pp. 501–16, doi:10.1007/978-3-540-45063-4_32.
short: V. Kolmogorov, R. Zabih, S. Gortler, in:, Springer, 2003, pp. 501–516.
conference:
name: 'EMMCVPR: Energy Minimization Methods in Computer Vision and Pattern Recognition'
date_created: 2018-12-11T12:01:48Z
date_published: 2003-06-26T00:00:00Z
date_updated: 2021-01-12T07:41:34Z
day: '26'
doi: 10.1007/978-3-540-45063-4_32
extern: 1
intvolume: ' 2683'
month: '06'
page: 501 - 516
publication_status: published
publisher: Springer
publist_id: '3512'
quality_controlled: 0
status: public
title: Generalized multi camera scene reconstruction using graph cuts
type: conference
volume: 2683
year: '2003'
...
---
_id: '3174'
abstract:
- lang: eng
text: We address visual correspondence problems without assuming that scene points
have similar intensities in different views. This situation is common, usually
due to non-lambertian scenes or to differences between cameras. We use maximization
of mutual information, a powerful technique for registering images that requires
no a priori model of the relationship between scene intensities in different views.
However, it has proven difficult to use mutual information to compute dense visual
correspondence. Comparing fixed-size windows via mutual information suffers from
the well-known problems of fixed windows, namely poor performance at discontinuities
and in low-texture regions. In this paper, we show how to compute visual correspondence
using mutual information without suffering from these problems. Using 'a simple
approximation, mutual information can be incorporated into the standard energy
minimization framework used in early vision. The energy can then be efficiently
minimized using graph cuts, which preserve discontinuities and handle low-texture
regions. The resulting algorithm combines the accurate disparity maps that come
from graph cuts with the tolerance for intensity changes that comes from mutual
information.
author:
- first_name: Junhwan
full_name: Kim, Junhwan
last_name: Kim
- first_name: Vladimir
full_name: Vladimir Kolmogorov
id: 3D50B0BA-F248-11E8-B48F-1D18A9856A87
last_name: Kolmogorov
- first_name: Ramin
full_name: Zabih, Ramin
last_name: Zabih
citation:
ama: 'Kim J, Kolmogorov V, Zabih R. Visual correspondence using energy minimization
and mutual information. In: Vol 2. IEEE; 2003:1033-1040. doi:10.1109/ICCV.2003.1238463'
apa: 'Kim, J., Kolmogorov, V., & Zabih, R. (2003). Visual correspondence using
energy minimization and mutual information (Vol. 2, pp. 1033–1040). Presented
at the ICCV: International Conference on Computer Vision, IEEE. https://doi.org/10.1109/ICCV.2003.1238463'
chicago: Kim, Junhwan, Vladimir Kolmogorov, and Ramin Zabih. “Visual Correspondence
Using Energy Minimization and Mutual Information,” 2:1033–40. IEEE, 2003. https://doi.org/10.1109/ICCV.2003.1238463.
ieee: 'J. Kim, V. Kolmogorov, and R. Zabih, “Visual correspondence using energy
minimization and mutual information,” presented at the ICCV: International Conference
on Computer Vision, 2003, vol. 2, pp. 1033–1040.'
ista: 'Kim J, Kolmogorov V, Zabih R. 2003. Visual correspondence using energy minimization
and mutual information. ICCV: International Conference on Computer Vision vol.
2, 1033–1040.'
mla: Kim, Junhwan, et al. Visual Correspondence Using Energy Minimization and
Mutual Information. Vol. 2, IEEE, 2003, pp. 1033–40, doi:10.1109/ICCV.2003.1238463.
short: J. Kim, V. Kolmogorov, R. Zabih, in:, IEEE, 2003, pp. 1033–1040.
conference:
name: 'ICCV: International Conference on Computer Vision'
date_created: 2018-12-11T12:01:49Z
date_published: 2003-09-30T00:00:00Z
date_updated: 2021-01-12T07:41:35Z
day: '30'
doi: 10.1109/ICCV.2003.1238463
extern: 1
intvolume: ' 2'
month: '09'
page: 1033 - 1040
publication_status: published
publisher: IEEE
publist_id: '3510'
quality_controlled: 0
status: public
title: Visual correspondence using energy minimization and mutual information
type: conference
volume: 2
year: '2003'
...
---
_id: '3170'
abstract:
- lang: eng
text: Geodesic active contours and graph cuts are two standard image segmentation
techniques. We introduce a new segmentation method combining some of their benefits.
Our main intuition is that any cut on a graph embedded in some continuous space
can be interpreted as a contour (in 2D) or a surface (in 3D). We show how to build
a grid graph and set its edge weights so that the cost of cuts is arbitrarily
close to the length (area) of the corresponding contours (surfaces) for any anisotropic
Riemannian metric. There are two interesting consequences of this technical result.
First, graph cut algorithms can be used to find globally minimum geodesic contours
(minimal surfaces in 3D) under arbitrary Riemannian metric for a given set of
boundary conditions. Second, we show how to minimize metrication artifacts in
existing graph-cut based methods in vision. Theoretically speaking, our work provides
an interesting link between several branches of mathematics -differential geometry,
integral geometry, and combinatorial optimization. The main technical problem
is solved using Cauchy-Crofton formula from integral geometry.
author:
- first_name: Yuri
full_name: Boykov, Yuri
last_name: Boykov
- first_name: Vladimir
full_name: Vladimir Kolmogorov
id: 3D50B0BA-F248-11E8-B48F-1D18A9856A87
last_name: Kolmogorov
citation:
ama: 'Boykov Y, Kolmogorov V. Computing geodesics and minimal surfaces via graph
cuts. In: Vol 1. IEEE; 2003:26-33. doi:10.1109/ICCV.2003.1238310'
apa: 'Boykov, Y., & Kolmogorov, V. (2003). Computing geodesics and minimal surfaces
via graph cuts (Vol. 1, pp. 26–33). Presented at the ICCV: International Conference
on Computer Vision, IEEE. https://doi.org/10.1109/ICCV.2003.1238310'
chicago: Boykov, Yuri, and Vladimir Kolmogorov. “Computing Geodesics and Minimal
Surfaces via Graph Cuts,” 1:26–33. IEEE, 2003. https://doi.org/10.1109/ICCV.2003.1238310.
ieee: 'Y. Boykov and V. Kolmogorov, “Computing geodesics and minimal surfaces via
graph cuts,” presented at the ICCV: International Conference on Computer Vision,
2003, vol. 1, pp. 26–33.'
ista: 'Boykov Y, Kolmogorov V. 2003. Computing geodesics and minimal surfaces via
graph cuts. ICCV: International Conference on Computer Vision vol. 1, 26–33.'
mla: Boykov, Yuri, and Vladimir Kolmogorov. Computing Geodesics and Minimal Surfaces
via Graph Cuts. Vol. 1, IEEE, 2003, pp. 26–33, doi:10.1109/ICCV.2003.1238310.
short: Y. Boykov, V. Kolmogorov, in:, IEEE, 2003, pp. 26–33.
conference:
name: 'ICCV: International Conference on Computer Vision'
date_created: 2018-12-11T12:01:48Z
date_published: 2003-09-30T00:00:00Z
date_updated: 2021-01-12T07:41:33Z
day: '30'
doi: 10.1109/ICCV.2003.1238310
extern: 1
intvolume: ' 1'
month: '09'
page: 26 - 33
publication_status: published
publisher: IEEE
publist_id: '3511'
quality_controlled: 0
status: public
title: Computing geodesics and minimal surfaces via graph cuts
type: conference
volume: 1
year: '2003'
...
---
_id: '3526'
abstract:
- lang: eng
text: Neurons can produce action potentials with high temporal precision(1). A fundamental
issue is whether, and how, this capability is used in information processing.
According to the `cell assembly' hypothesis, transient synchrony of anatomically
distributed groups of neurons underlies processing of both external sensory input
and internal cognitive mechanisms(2-4). Accordingly, neuron populations should
be arranged into groups whose synchrony exceeds that predicted by common modulation
by sensory input. Here we find that the spike times of hippocampal pyramidal cells
can be predicted more accurately by using the spike times of simultaneously recorded
neurons in addition to the animals location in space. This improvement remained
when the spatial prediction was refined with a spatially dependent theta phase
modulation(5-8). The time window in which spike times are best predicted from
simultaneous peer activity is 10-30 ms, suggesting that cell assemblies are synchronized
at this timescale. Because this temporal window matches the membrane time constant
of pyramidal neurons(9), the period of the hippocampal gamma oscillation(10) and
the time window for synaptic plasticity(11), we propose that cooperative activity
at this timescale is optimal for information transmission and storage in cortical
circuits.
author:
- first_name: Kenneth
full_name: Harris, Kenneth D
last_name: Harris
- first_name: Jozsef L
full_name: Jozsef Csicsvari
id: 3FA14672-F248-11E8-B48F-1D18A9856A87
last_name: Csicsvari
orcid: 0000-0002-5193-4036
- first_name: Hajima
full_name: Hirase, Hajima
last_name: Hirase
- first_name: George
full_name: Dragoi, George
last_name: Dragoi
- first_name: György
full_name: Buzsáki, György
last_name: Buzsáki
citation:
ama: Harris K, Csicsvari JL, Hirase H, Dragoi G, Buzsáki G. Organization of cell
assemblies in the hippocampus. Nature. 2003;424(6948):552-556. doi:0.1038/nature01834
apa: Harris, K., Csicsvari, J. L., Hirase, H., Dragoi, G., & Buzsáki, G. (2003).
Organization of cell assemblies in the hippocampus. Nature. Nature Publishing
Group. https://doi.org/0.1038/nature01834
chicago: Harris, Kenneth, Jozsef L Csicsvari, Hajima Hirase, George Dragoi, and
György Buzsáki. “Organization of Cell Assemblies in the Hippocampus.” Nature.
Nature Publishing Group, 2003. https://doi.org/0.1038/nature01834.
ieee: K. Harris, J. L. Csicsvari, H. Hirase, G. Dragoi, and G. Buzsáki, “Organization
of cell assemblies in the hippocampus,” Nature, vol. 424, no. 6948. Nature
Publishing Group, pp. 552–556, 2003.
ista: Harris K, Csicsvari JL, Hirase H, Dragoi G, Buzsáki G. 2003. Organization
of cell assemblies in the hippocampus. Nature. 424(6948), 552–556.
mla: Harris, Kenneth, et al. “Organization of Cell Assemblies in the Hippocampus.”
Nature, vol. 424, no. 6948, Nature Publishing Group, 2003, pp. 552–56,
doi:0.1038/nature01834.
short: K. Harris, J.L. Csicsvari, H. Hirase, G. Dragoi, G. Buzsáki, Nature 424 (2003)
552–556.
date_created: 2018-12-11T12:03:47Z
date_published: 2003-07-31T00:00:00Z
date_updated: 2021-01-12T07:44:04Z
day: '31'
doi: 0.1038/nature01834
extern: 1
intvolume: ' 424'
issue: '6948'
month: '07'
page: 552 - 556
publication: Nature
publication_status: published
publisher: Nature Publishing Group
publist_id: '2859'
quality_controlled: 0
status: public
title: Organization of cell assemblies in the hippocampus
type: journal_article
volume: 424
year: '2003'
...